Predictive Factors for Access-Site Pain Chronicity after Percutaneous Coronary Intervention via Radial Artery Access

Objectives. The aim of this study is to assess the prevalence and predictive factors for developing chronic access-site (A-S) pain after percutaneous coronary intervention (PCI) via radial artery access. Methods. Data of selected patients (n = 161) who underwent elective PCI were collected prospecti...

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Main Authors: Liuda Brogiene, Giedre Baksyte, Agne Klimaite, Martynas Paliokas, Andrius Macas
Format: Article
Language:English
Published: Wiley 2020-01-01
Series:Pain Research and Management
Online Access:http://dx.doi.org/10.1155/2020/8887499
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author Liuda Brogiene
Giedre Baksyte
Agne Klimaite
Martynas Paliokas
Andrius Macas
author_facet Liuda Brogiene
Giedre Baksyte
Agne Klimaite
Martynas Paliokas
Andrius Macas
author_sort Liuda Brogiene
collection DOAJ
description Objectives. The aim of this study is to assess the prevalence and predictive factors for developing chronic access-site (A-S) pain after percutaneous coronary intervention (PCI) via radial artery access. Methods. Data of selected patients (n = 161) who underwent elective PCI were collected prospectively and analysed in 2020. Verbal analogue scale was used to evaluate pain intensity after 12, 24, and 48 h and 3 months after PCI. The univariate logistic regression analysis was used. Results. Pain prevalence decreased from 29% straight after PCI and 54% two hours later to 3.7% following 3 months after procedure. The predictors for A-S pain chronicity are diabetes (OR = 5.77 95% CI (1.07–31.08), p=0.041), hematoma (OR = 6.48, 95% CI (1.06–39.66), p=0.043), A-S hand neuropathy (OR = 19.93 95% CI (1.27–312.32), p=0.033), A-S pain immediately after PCI (OR = 14.60 95% CI (1.63–130.27), p=0.016), after 12 h (OR = 17.2 95% CI (1.60–185.27), p=0.019), 24 h (OR = 48 95% CI (4.87–487), p=0.01), and 48 h (OR = 23.46 95% CI (3.81–144.17), p=0.001), and pain intensity immediately after procedure (OR = 3.30 95% CI (1.65–6.60), p=0.001), after 2 h (OR = 2.56 95% CI (1.15–5.73), p=0.022), after 12 h (OR = 3.02 95% CI (1.70–5.39), p<0.001), after 24 h (OR = 3.58 95% CI (1.90–6.74), p<0.001), and after 48 h (OR = 2.89 95% CI (1.72–4.87), p<0.001). Pain control was performed with Ketoprofen and Ibuprofen as most used NSAIDs. 10 mg of Morphine intravenously was the choice from strong opioids if necessary. Conclusions. The prevalence of chronic A-S pain is 3.7%. Main predictive factors for the A-S pain chronicity are diabetes, hematoma, and persistent pain and pain intensity during 48 h period after PCI.
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spelling doaj-art-fac10aecfaa5408699e92e6b36a7d0cf2025-02-03T05:58:27ZengWileyPain Research and Management1203-67651918-15232020-01-01202010.1155/2020/88874998887499Predictive Factors for Access-Site Pain Chronicity after Percutaneous Coronary Intervention via Radial Artery AccessLiuda Brogiene0Giedre Baksyte1Agne Klimaite2Martynas Paliokas3Andrius Macas4Department of Anaesthesiology, Lithuanian University of Health Sciences, Kaunas, LithuaniaDepartment of Cardiology, Lithuanian University of Health Sciences, Kaunas, LithuaniaDepartment of Cardiology, Lithuanian University of Health Sciences, Kaunas, LithuaniaDepartment of Anaesthesiology, Lithuanian University of Health Sciences, Kaunas, LithuaniaDepartment of Anaesthesiology, Lithuanian University of Health Sciences, Kaunas, LithuaniaObjectives. The aim of this study is to assess the prevalence and predictive factors for developing chronic access-site (A-S) pain after percutaneous coronary intervention (PCI) via radial artery access. Methods. Data of selected patients (n = 161) who underwent elective PCI were collected prospectively and analysed in 2020. Verbal analogue scale was used to evaluate pain intensity after 12, 24, and 48 h and 3 months after PCI. The univariate logistic regression analysis was used. Results. Pain prevalence decreased from 29% straight after PCI and 54% two hours later to 3.7% following 3 months after procedure. The predictors for A-S pain chronicity are diabetes (OR = 5.77 95% CI (1.07–31.08), p=0.041), hematoma (OR = 6.48, 95% CI (1.06–39.66), p=0.043), A-S hand neuropathy (OR = 19.93 95% CI (1.27–312.32), p=0.033), A-S pain immediately after PCI (OR = 14.60 95% CI (1.63–130.27), p=0.016), after 12 h (OR = 17.2 95% CI (1.60–185.27), p=0.019), 24 h (OR = 48 95% CI (4.87–487), p=0.01), and 48 h (OR = 23.46 95% CI (3.81–144.17), p=0.001), and pain intensity immediately after procedure (OR = 3.30 95% CI (1.65–6.60), p=0.001), after 2 h (OR = 2.56 95% CI (1.15–5.73), p=0.022), after 12 h (OR = 3.02 95% CI (1.70–5.39), p<0.001), after 24 h (OR = 3.58 95% CI (1.90–6.74), p<0.001), and after 48 h (OR = 2.89 95% CI (1.72–4.87), p<0.001). Pain control was performed with Ketoprofen and Ibuprofen as most used NSAIDs. 10 mg of Morphine intravenously was the choice from strong opioids if necessary. Conclusions. The prevalence of chronic A-S pain is 3.7%. Main predictive factors for the A-S pain chronicity are diabetes, hematoma, and persistent pain and pain intensity during 48 h period after PCI.http://dx.doi.org/10.1155/2020/8887499
spellingShingle Liuda Brogiene
Giedre Baksyte
Agne Klimaite
Martynas Paliokas
Andrius Macas
Predictive Factors for Access-Site Pain Chronicity after Percutaneous Coronary Intervention via Radial Artery Access
Pain Research and Management
title Predictive Factors for Access-Site Pain Chronicity after Percutaneous Coronary Intervention via Radial Artery Access
title_full Predictive Factors for Access-Site Pain Chronicity after Percutaneous Coronary Intervention via Radial Artery Access
title_fullStr Predictive Factors for Access-Site Pain Chronicity after Percutaneous Coronary Intervention via Radial Artery Access
title_full_unstemmed Predictive Factors for Access-Site Pain Chronicity after Percutaneous Coronary Intervention via Radial Artery Access
title_short Predictive Factors for Access-Site Pain Chronicity after Percutaneous Coronary Intervention via Radial Artery Access
title_sort predictive factors for access site pain chronicity after percutaneous coronary intervention via radial artery access
url http://dx.doi.org/10.1155/2020/8887499
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