Molecular Mechanisms of Treadmill Therapy on Neuromuscular Atrophy Induced via Botulinum Toxin A

Botulinum toxin A (BoNT-A) is a bacterial zinc-dependent endopeptidase that acts specifically on neuromuscular junctions. BoNT-A blocks the release of acetylcholine, thereby decreasing the ability of a spastic muscle to generate forceful contraction, which results in a temporal local weakness and th...

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Bibliographic Details
Main Authors: Sen-Wei Tsai, Hsiao-Ling Chen, Yi-Chun Chang, Chuan-Mu Chen
Format: Article
Language:English
Published: Wiley 2013-01-01
Series:Neural Plasticity
Online Access:http://dx.doi.org/10.1155/2013/593271
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