RYBP promotes HIV-1 latency through promoting H2AK119ub and decreasing H3K4me3

RYBP promotes HIV-1 latency through promoting H2AK119ub and decreasing H3K4me3

Abstract Background Acquired immunodeficiency syndrome (AIDS) cannot be completely cured, and the main obstacle is the existence of viral reservoirs. However, we currently do not fully understand the molecular mechanisms by which HIV-1 latency is established and maintained. Methods Here, based on en...

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Main Authors: Xinyi Yang, Yuqi Zhu, Xiaying Zhao, Jingna Xun, Xingyu Wang, Yipeng Cheng, Su Xiong, Xingwen Yu, Suixiang Li, Danqing Wang, Zhiliang Hu, Yinzhong Shen, Shibo Jiang, Hongzhou Lu, Gang Wang, Huanzhang Zhu
Format: Article
Language:English
Published: BMC 2025-05-01
Series:Cell Communication and Signaling
Subjects:
RYBP
KDM2B
HIV-1 latency
H2AK119ub
EnChIP
Online Access:https://doi.org/10.1186/s12964-025-02221-z
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https://doi.org/10.1186/s12964-025-02221-z

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