IL-33 Acts to Express Schaffer Collateral/CA1 LTP and Regulate Learning and Memory by Targeting MyD88
Interleukin-33 (IL-33) is recognized to transmit a signal through a heterodimeric receptor complex ST2/interleukin-1 receptor accessory protein (IL-1RAcP) bearing activation of myeloid differentiation factor 88 (MyD88). High-frequency stimulation to the Schaffer collateral induced long-term potentia...
Saved in:
Main Author: | Tomoyuki Nishizaki |
---|---|
Format: | Article |
Language: | English |
Published: |
Wiley
2017-01-01
|
Series: | Neural Plasticity |
Online Access: | http://dx.doi.org/10.1155/2017/2531453 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
In Vivo Plasticity at Hippocampal Schaffer Collateral-CA1 Synapses: Replicability of the LTP Response and Pharmacology in the Long-Evans Rat
by: A. Ahnaou, et al.
Published: (2020-01-01) -
Functional Metaplasticity of Hippocampal Schaffer Collateral-CA1 Synapses Is Reversed in Chronically Epileptic Rats
by: Mirko Rehberg, et al.
Published: (2017-01-01) -
Wedelolactone Mitigates Alcoholic Steatohepatitis via Modulating the TLR4/MyD88/NF-κB Pathway
by: Tao Jiang, et al.
Published: (2024-01-01) -
AICpred: Machine Learning-Based Prediction of Potential Anti-Inflammatory Compounds Targeting TLR4-MyD88 Binding Mechanism
by: Lucindah N. Fry-Nartey, et al.
Published: (2025-01-01) -
Extracellular HMGB1 Induced Glomerular Endothelial Cell Injury via TLR4/MyD88 Signaling Pathway in Lupus Nephritis
by: Tian Yu, et al.
Published: (2021-01-01)