Disruption of the biorhythm in gastric epithelial cell triggers inflammation in Helicobacter pylori-associated gastritis by aberrantly regulating NFIL3 via CagA activated ERK-SP1 pathway

Disruption of the biorhythm in gastric epithelial cell triggers inflammation in Helicobacter pylori-associated gastritis by aberrantly regulating NFIL3 via CagA activated ERK-SP1 pathway

Abstract Helicobacter pylori (H. pylori) associated gastritis, marked by chronic gastric inflammation, heightens gastric cancer risk by fostering a malignancy-prone microenvironment. Disruption of the biorhythm contribute to the onset of various gastrointestinal disorders, such as gastric dyspepsia,...

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Bibliographic Details
Main Authors: Yongsheng Teng, Yipin Lv, Wanyan Chen, Fangyuan Mao, Liusheng Peng, He Huang, Haiyan Li, Liwei Shi, Quanming Zou, Yuan Zhuang, Wenqing Tian, Hong Guo
Format: Article
Language:English
Published: BMC 2025-06-01
Series:Cell Communication and Signaling
Subjects:
Helicobacter pylori
NFIL3
Biorhythm
Gastric epithelial cells
Gastritis
Online Access:https://doi.org/10.1186/s12964-025-02302-z
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https://doi.org/10.1186/s12964-025-02302-z

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