Glutamatergic CYLD deletion leads to aberrant excitatory activity in the basolateral amygdala: association with enhanced cued fear expression

Glutamatergic CYLD deletion leads to aberrant excitatory activity in the basolateral amygdala: association with enhanced cued fear expression

Neuronal activity, synaptic transmission, and molecular changes in the basolateral amygdala play critical roles in fear memory. Cylindromatosis (CYLD) is a deubiquitinase that negatively regulates the nuclear factor kappa-B pathway. CYLD is well studied in non-neuronal cells, yet under-investigated...

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Bibliographic Details
Main Authors: Huidong Li, Faqin Li, Zhaoyi Chen, Erwen Wu, Xiaoxi Dai, Danni Li, Haojie An, Shiyi Zeng, Chunyan Wang, Li Yang, Cheng Long
Format: Article
Language:English
Published: Wolters Kluwer Medknow Publications 2025-11-01
Series:Neural Regeneration Research
Subjects:
basolateral amygdala
cued fear expression
cylindromatosis
deubiquitinase
glutamate receptor 1
glutamatergic neuron
microglial activation
n-methyl-d-aspartate receptor 1
neuronal activation
synaptic transmission
Online Access:https://journals.lww.com/10.4103/NRR.NRR-D-24-00054
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https://journals.lww.com/10.4103/NRR.NRR-D-24-00054

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