Showing 5,901 - 5,920 results of 14,077 for search '"genetics"', query time: 0.20s Refine Results
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    USE OF SYNTHETIC FORMS IN THE PRESERVATION AND EXPLOITATION OF THE GENE POOL OF WILD COMMON WHEAT RELATIVES by R. O. Davoyan, I. V. Bebyakina, O. R. Davoyan, A. N. Zinchenko, E. R. Davoyan, A. M. Kravchenko, Y. S. Zubanova

    Published 2014-12-01
    “…The synthetic forms provide a unique genetic basis for preservation and use of the gene pool of wild relatives in wheat breeding. …”
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    Iniencephaly and Holoprosencephaly: Report of a Rare Association by Aytekin Tokmak, Hakan Timur, Korkut Dağlar, Özgür Kara

    Published 2014-01-01
    “…The family declined an autopsy and genetic counseling. In this case, genetics and environmental causes, including lower socioeconomic status and lack of folic acid supplementation, may be risk factors for the current disorder. …”
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    Vitamin K Deficiency Embryopathy from Hyperemesis Gravidarum by Andrew S. Lane, Jennifer L. Stallworth, Kacey Y. Eichelberger, Kenneth F. Trofatter

    Published 2015-01-01
    “…At repeated anatomy ultrasound at 21-week gestation, delivery, and postnatal pediatric genetics exam, nasal hypoplasia was consistent with vitamin K deficiency embryopathy from HG. …”
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    Multi-tissue characterization of the constitutive heterochromatin proteome in Drosophila identifies a link between satellite DNA organization and transposon repression. by Ankita Chavan, Lena Skrutl, Federico Uliana, Melanie Pfister, Franziska Brändle, Laszlo Tirian, Delora Baptista, Dominik Handler, David Burke, Anna Sintsova, Pedro Beltrao, Julius Brennecke, Madhav Jagannathan

    Published 2025-01-01
    “…Interestingly, we find that multiple components of the transposon-silencing piRNA pathway are associated with D1 and Prod in embryos. Using genetics, transcriptomics, and small RNA profiling, we show that flies lacking D1 during embryogenesis exhibit transposon expression and gonadal atrophy as adults. …”
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  20. 5920

    The DNA Replication Stress Hypothesis of Alzheimer’s Disease by Yuri B. Yurov, Svetlana G. Vorsanova, Ivan Y. Iourov

    Published 2011-01-01
    “…DNA replication stress can be a key element of the pathogenetic cascade explaining the interplay between ectopic cell cycle events and genetic instabilities in the AD brain. Abnormal cell cycle reentry and somatic genome variations can be used for updating the cell cycle theory introducing replication stress as a missing link between cell genetics and neurobiology of AD.…”
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