Showing 1,081 - 1,100 results of 4,487 for search '"bone"', query time: 0.07s Refine Results
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    A Patient Specific Biomechanical Analysis of Custom Root Analogue Implant Designs on Alveolar Bone Stress: A Finite Element Study by David Anssari Moin, Bassam Hassan, Daniel Wismeijer

    Published 2016-01-01
    “…The aim of this study was to analyse by means of FEA the influence of 5 custom RAI designs on stress distribution of peri-implant bone and to evaluate the impact on microdisplacement for a specific patient case. …”
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  9. 1089

    Collagen Extraction From Bigeye Tuna (Thunnus obesus) Bone Byproduct: Process Modeling and Technoeconomic Assessment Using SuperPro Designer by Abyan Shiddiiq, Elly Septia Yulianti, Misri Gozan, Siti Fauziyah Rahman

    Published 2025-01-01
    “…This study evaluates the potential of utilizing tuna bone byproducts for acid-soluble collagen (ASC) extraction in Indonesia, targeting the biomedical industry’s growing collagen demand. …”
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    Healing beyond bone: a qualitative study of the core decompression experience of physically active patients with stage II hip avascular necrosis by Mehmet Yiğit Gökmen, Habibe Sevim Gökmen, Mesut Uluöz, Özhan Pazarcı, Alaaddin Levent Özgözen, Ender Ali Uluç, Osman Çiloğlu

    Published 2025-01-01
    “…Abstract Background Avascular necrosis (AVN) of the femoral head is a degenerative condition characterized by ischemic bone death, resulting in pain and impaired mobility. …”
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  12. 1092

    Dexamethasone Coanalgesic Administration in Steroid Naïve and Steroid Non-Naïve Patients for the Prevention of Pain Flares after Palliative Radiotherapy for Bone Metastases by Dimitar G. Tonev, Silvia A. Lalova, Elena P. Petkova-Lungova, Nikolay V. Timenov, Gabriela M. Radeva, Todor G. Kundurzhiev

    Published 2022-01-01
    “…Dexamethasone could be an effective prophylactic agent for the prevention of pain flares after palliative radiotherapy (RT) for uncomplicated bone metastases. To date, there are no data on its prophylactic coanalgesic (opioid-sparing) effect after RT in patients with complicated bone metastases compared to uncomplicated ones, which is the aim of our study. …”
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    Renal Ischemia-Reperfusion Injury in a Diabetic Monkey Model and Therapeutic Testing of Human Bone Marrow-Derived Mesenchymal Stem Cells by Kyo Won Lee, Tae Min Kim, Kyeong Sik Kim, Seunghwan Lee, Junhun Cho, Jae Berm Park, Ghee Young Kwon, Sung Joo Kim

    Published 2018-01-01
    “…We also investigated whether human bone marrow-derived mesenchymal stem cells (hBM-MSCs) infused via the renal artery could ameliorate renal IRI in DM monkeys. …”
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    Triptolide Inhibits Osteoclast Differentiation and Bone Resorption In Vitro via Enhancing the Production of IL-10 and TGF-β1 by Regulatory T Cells by Huihui Xu, Hongyan Zhao, Cheng Lu, Qi Qiu, Gui Wang, Jing Huang, Minghui Guo, Baosheng Guo, Yong Tan, Cheng Xiao

    Published 2016-01-01
    “…Although triptolide has demonstrated that it could suppress bone destruction in collagen-induced mice, its therapeutic mechanism remains unclear. …”
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  17. 1097

    All-Trans Retinoic Acid Improves the Effects of Bone Marrow-Derived Mesenchymal Stem Cells on the Treatment of Ankylosing Spondylitis: An In Vitro Study by Deng Li, Peng Wang, Yuxi Li, Zhongyu Xie, Le Wang, Hongjun Su, Wen Deng, Yanfeng Wu, Huiyong Shen

    Published 2015-01-01
    “…The present study aimed to assess the immunoregulatory effects of ATRA on MSCs in the treatment of ankylosing spondylitis (AS). Bone marrow-derived MSCs from healthy donors were pretreated with ATRA and cocultured with CD3/28-activated peripheral blood mononuclear cells (PBMCs) derived from AS patients. …”
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    Advanced Glycation End Products Enhance Murine Monocyte Proliferation in Bone Marrow and Prime Them into an Inflammatory Phenotype through MAPK Signaling by Xian Jin, Liang Liu, Yaping Zhang, Yin Xiang, Guizhi Yin, Yi Lu, Ludong Shi, Jian Dong, Chengxing Shen

    Published 2018-01-01
    “…The present study tested the hypothesis that advanced glycation end products (AGEs) promote bone marrow monocytes to proliferate and drive them into an inflammatory phenotype. …”
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