Showing 1 - 5 results of 5 for search '"anterograde amnesia"', query time: 0.03s Refine Results
  1. 1

    An “Engram-Centric” Approach to Transient Global Amnesia (TGA) and Other Acute-Onset Amnesias by Andrew J. Larner

    Published 2025-01-01
    “…The most common of these, TGA, is a rare but well-described condition characterised by a self-limited episode of dense anterograde amnesia with variable retrograde amnesia. Although the clinical phenomenology of TGA is well described, its pathogenesis is not currently understood, thus preventing the development of evidence-based therapeutic recommendations. …”
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  2. 2

    Wernicke-Korsakoff Syndrome following Small Bowel Obstruction by Shoumitro Deb, Richard Law-Min, David Fearnley

    Published 2002-01-01
    “…Within a few months of treatment with a daily oral dose of thiamine 200 mgs supplemented by multivitamins the patient showed subjective evidence of improvement in confusion, confabulation, and anterograde amnesia, although objective tests showed residual deficits in many areas of cognitive functioning, including immediate and delayed recall of verbal and non-verbal materials, planning and switching of attention.…”
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  3. 3

    From memory impairment to two occluders in the heart – a multidisciplinary case report by A. Wiśniewski, K. Filipska, A. Ekkert

    Published 2021-12-01
    “… Transient global amnesia (TGA) is a rare cause of a sudden onset of confusion and anterograde amnesia. It is considered of ischemic, epileptogenic, or venous origin. …”
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  4. 4

    Transient Global Amnesia with Reversible White Matter Lesions: A Variant of Posterior Reversible Encephalopathy Syndrome? by Tomoki Nakamizo, Ippei Tsuzuki, Takashi Koide

    Published 2015-01-01
    “…She had complete anterograde amnesia and slight retrograde amnesia without other neurological findings. …”
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  5. 5

    Neurotoxic Dorsal CA1 Lesions versus 4 VO Ischaemic Lesions: Behavioural Comparisons by J. A. Nunn, J. A. Gray, H. Hodges

    Published 1999-01-01
    “…Anterograde amnesia, a common consequence of transient cerebral ischaemia, has been attributed to cell loss in the hippocampal CA1 subfield. …”
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