Showing 21 - 40 results of 901 for search '"Alzheimers"', query time: 0.08s Refine Results
  1. 21

    CLOCK Genes and Circadian Rhythmicity in Alzheimer Disease by J. Thome, A. N. Coogan, A. G. Woods, C. C. Darie, F. Häßler

    Published 2011-01-01
    “…Disturbed circadian rhythms with sleep problems and disrupted diurnal activity are often seen in patients suffering from Alzheimer disease (AD). Both endogenous CLOCK genes and external Zeitgeber are responsible for the maintenance of circadian rhythmicity in humans. …”
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  2. 22

    Spines, Plasticity, and Cognition in Alzheimer's Model Mice by Tara Spires-Jones, Shira Knafo

    Published 2012-01-01
    “…The pathological hallmarks of Alzheimer's disease (AD)—widespread synaptic and neuronal loss and the pathological accumulation of amyloid-beta peptide (Aβ) in senile plaques, as well as hyperphosphorylated tau in neurofibrillary tangles—have been known for many decades, but the links between AD pathology and dementia and effective therapeutic strategies remain elusive. …”
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  3. 23

    Diagnosis and Treatment of Alzheimer’s Disease: Current Update by Younghee Yim, Won-Jin Moon

    Published 2025-01-01
    Subjects: “…alzheimer’s disease…”
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    Article
  4. 24

    Synaptic Cell Adhesion Molecules in Alzheimer’s Disease by Iryna Leshchyns’ka, Vladimir Sytnyk

    Published 2016-01-01
    “…Alzheimer’s disease (AD) is a neurodegenerative brain disorder associated with the loss of synapses between neurons in the brain. …”
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  5. 25

    Application of Low Intensity Ultrasound in the Treatment of Alzheimer’s Disease by Hong Fei, Xu Fei

    Published 2025-03-01
    Subjects: “…|alzheimer's disease|pathology and pathogenesis|low intensity ultrasound…”
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    Article
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    Consensus on rapid screening for prodromal Alzheimer’s disease in China by Yao Lu, Ying Wang, Jianping Jia, Qihao Guo, Ya Miao, Lin Huang, Xiaochun Chen, Qinjie Li, Fengfeng Pan, Liang Cui, Tianlu Chen, Yatian Li, Jingnan Wu

    Published 2024-02-01
    “…Alzheimer’s disease (AD) is a common cause of dementia, characterised by cerebral amyloid-β deposition, pathological tau and neurodegeneration. …”
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    Alzheimer’s Disease and Cardiovascular Disease: A Particular Association by Giacomo Tini, Riccardo Scagliola, Fiammetta Monacelli, Giovanni La Malfa, Italo Porto, Claudio Brunelli, Gian Marco Rosa

    Published 2020-01-01
    “…During ageing, the prevalence of dementia, and especially of Alzheimer’s disease (AD) and cardiovascular disease (CVD), increases. …”
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    Hepatocyte Growth Factor in Synaptic Plasticity and Alzheimer's Disease by Shiv K. Sharma

    Published 2010-01-01
    “…This review describes the role of HGF in hippocampal neurons, synaptic plasticity, and the memory impairment condition, Alzheimer's disease.…”
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  16. 36

    Neuropsychological Rehabilitation in Mild and Moderate Alzheimer’s Disease Patients by Renata Ávila, Isabel A. M. Carvalho, Cássio M.C. Bottino, Eliane C. Miotto

    Published 2007-01-01
    “…Objective: The purpose of this study was to analyze the effect of a neuropsychological rehabilitation (NR) program on patients with Alzheimer’s disease (AD).…”
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    Exercise Intervention Associated with Cognitive Improvement in Alzheimer’s Disease by Meng Ying Cui, Yang Lin, Ji Yao Sheng, Xuewen Zhang, Ran Ji Cui

    Published 2018-01-01
    “…Alzheimer’s disease (AD) is a progressive neurodegenerative disease with the syndrome of cognitive and functional decline. …”
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  19. 39

    SPECT Findings in Alzheimer’s Disease and Parkinson’s Disease with Dementia by S. E. Starkstein, S. Vázquez, G. Petracca, L. Sabe, M. Merello, R. Leiguarda

    Published 1997-01-01
    “…We examined, with single photon emission tomography (SPECT) and (99mTc)-HMPAO, 18 patients with idiopathic Parkinson's disease and no dementia (PD), 12 patients with PD and dementia, 24 patients with probable Alzheimer's disease (AD), and 14 controls. While the three patient groups showed significantly lower perfusion in frontal inferior and temporal inferior areas as compared to controls, both demented groups showed significantly more severe bilateral hypoperfusion in superior frontal, superior temporal and parietal areas as compared to non-demented PD patients and controls. …”
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