Epigenetic modifications in follicular cell-derived thyroid cancer: new dimensions in pathogenesis and treatment

The incidence of thyroid cancer has been rising in recent years. While tumorigenesis has traditionally been attributed to the accumulation of genetic mutations in oncogenes and tumor suppressor genes, increasing attention has been directed toward the role of epigenetic regulation in cancer developme...

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Main Authors: Yanmei Han, Bian Wu, Jie Tan, Ruolin Wu, Jingjing Wang, Xiaojing Ren, Yajing Zhang, Zairong Gao, Xiaotian Xia
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-05-01
Series:Frontiers in Oncology
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Online Access:https://www.frontiersin.org/articles/10.3389/fonc.2025.1549477/full
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author Yanmei Han
Yanmei Han
Yanmei Han
Bian Wu
Jie Tan
Ruolin Wu
Ruolin Wu
Ruolin Wu
Jingjing Wang
Jingjing Wang
Jingjing Wang
Xiaojing Ren
Xiaojing Ren
Xiaojing Ren
Yajing Zhang
Yajing Zhang
Yajing Zhang
Zairong Gao
Zairong Gao
Zairong Gao
Xiaotian Xia
Xiaotian Xia
Xiaotian Xia
author_facet Yanmei Han
Yanmei Han
Yanmei Han
Bian Wu
Jie Tan
Ruolin Wu
Ruolin Wu
Ruolin Wu
Jingjing Wang
Jingjing Wang
Jingjing Wang
Xiaojing Ren
Xiaojing Ren
Xiaojing Ren
Yajing Zhang
Yajing Zhang
Yajing Zhang
Zairong Gao
Zairong Gao
Zairong Gao
Xiaotian Xia
Xiaotian Xia
Xiaotian Xia
author_sort Yanmei Han
collection DOAJ
description The incidence of thyroid cancer has been rising in recent years. While tumorigenesis has traditionally been attributed to the accumulation of genetic mutations in oncogenes and tumor suppressor genes, increasing attention has been directed toward the role of epigenetic regulation in cancer development. Since the 1980s, however, it has been acknowledged that the role of another key regulatory system in carcinogenesis: epigenetics, shedding light on the regulation of gene expression without altering the DNA sequence.This review synthesizes current literature on epigenetic alterations in follicular cell-derived thyroid cancers, focusing on DNA methylation, histone modifications, chromatin remodeling, and RNA regulation. Evidence indicates that dysregulation of these epigenetic processes is prevalent in thyroid cancer, influencing tumor initiation, progression, and resistance to therapy. Several epigenetic inhibitors are under development, some demonstrating synergy with existing chemotherapies and immunotherapies. Understanding these mechanisms may facilitate the development of novel, more effective strategies for early detection and treatment.
format Article
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institution Kabale University
issn 2234-943X
language English
publishDate 2025-05-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Oncology
spelling doaj-art-ee669c8dbdfa43d18a8dd5bc75cc16c82025-08-20T03:48:06ZengFrontiers Media S.A.Frontiers in Oncology2234-943X2025-05-011510.3389/fonc.2025.15494771549477Epigenetic modifications in follicular cell-derived thyroid cancer: new dimensions in pathogenesis and treatmentYanmei Han0Yanmei Han1Yanmei Han2Bian Wu3Jie Tan4Ruolin Wu5Ruolin Wu6Ruolin Wu7Jingjing Wang8Jingjing Wang9Jingjing Wang10Xiaojing Ren11Xiaojing Ren12Xiaojing Ren13Yajing Zhang14Yajing Zhang15Yajing Zhang16Zairong Gao17Zairong Gao18Zairong Gao19Xiaotian Xia20Xiaotian Xia21Xiaotian Xia22Department of Nuclear Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaHubei Province Key Laboratory of Molecular Imaging, Wuhan, ChinaKey Laboratory of Biological Targeted Therapy, the Ministry of Education, Wuhan, ChinaCancer Center, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, ChinaDepartment of Breast and Thyroid Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, ChinaDepartment of Nuclear Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaHubei Province Key Laboratory of Molecular Imaging, Wuhan, ChinaKey Laboratory of Biological Targeted Therapy, the Ministry of Education, Wuhan, ChinaDepartment of Nuclear Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaHubei Province Key Laboratory of Molecular Imaging, Wuhan, ChinaKey Laboratory of Biological Targeted Therapy, the Ministry of Education, Wuhan, ChinaDepartment of Nuclear Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaHubei Province Key Laboratory of Molecular Imaging, Wuhan, ChinaKey Laboratory of Biological Targeted Therapy, the Ministry of Education, Wuhan, ChinaDepartment of Nuclear Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaHubei Province Key Laboratory of Molecular Imaging, Wuhan, ChinaKey Laboratory of Biological Targeted Therapy, the Ministry of Education, Wuhan, ChinaDepartment of Nuclear Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaHubei Province Key Laboratory of Molecular Imaging, Wuhan, ChinaKey Laboratory of Biological Targeted Therapy, the Ministry of Education, Wuhan, ChinaDepartment of Nuclear Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaHubei Province Key Laboratory of Molecular Imaging, Wuhan, ChinaKey Laboratory of Biological Targeted Therapy, the Ministry of Education, Wuhan, ChinaThe incidence of thyroid cancer has been rising in recent years. While tumorigenesis has traditionally been attributed to the accumulation of genetic mutations in oncogenes and tumor suppressor genes, increasing attention has been directed toward the role of epigenetic regulation in cancer development. Since the 1980s, however, it has been acknowledged that the role of another key regulatory system in carcinogenesis: epigenetics, shedding light on the regulation of gene expression without altering the DNA sequence.This review synthesizes current literature on epigenetic alterations in follicular cell-derived thyroid cancers, focusing on DNA methylation, histone modifications, chromatin remodeling, and RNA regulation. Evidence indicates that dysregulation of these epigenetic processes is prevalent in thyroid cancer, influencing tumor initiation, progression, and resistance to therapy. Several epigenetic inhibitors are under development, some demonstrating synergy with existing chemotherapies and immunotherapies. Understanding these mechanisms may facilitate the development of novel, more effective strategies for early detection and treatment.https://www.frontiersin.org/articles/10.3389/fonc.2025.1549477/fullepigeneticsfollicular cell-derived thyroid cancerbiomarkersinhibitorstherapy
spellingShingle Yanmei Han
Yanmei Han
Yanmei Han
Bian Wu
Jie Tan
Ruolin Wu
Ruolin Wu
Ruolin Wu
Jingjing Wang
Jingjing Wang
Jingjing Wang
Xiaojing Ren
Xiaojing Ren
Xiaojing Ren
Yajing Zhang
Yajing Zhang
Yajing Zhang
Zairong Gao
Zairong Gao
Zairong Gao
Xiaotian Xia
Xiaotian Xia
Xiaotian Xia
Epigenetic modifications in follicular cell-derived thyroid cancer: new dimensions in pathogenesis and treatment
Frontiers in Oncology
epigenetics
follicular cell-derived thyroid cancer
biomarkers
inhibitors
therapy
title Epigenetic modifications in follicular cell-derived thyroid cancer: new dimensions in pathogenesis and treatment
title_full Epigenetic modifications in follicular cell-derived thyroid cancer: new dimensions in pathogenesis and treatment
title_fullStr Epigenetic modifications in follicular cell-derived thyroid cancer: new dimensions in pathogenesis and treatment
title_full_unstemmed Epigenetic modifications in follicular cell-derived thyroid cancer: new dimensions in pathogenesis and treatment
title_short Epigenetic modifications in follicular cell-derived thyroid cancer: new dimensions in pathogenesis and treatment
title_sort epigenetic modifications in follicular cell derived thyroid cancer new dimensions in pathogenesis and treatment
topic epigenetics
follicular cell-derived thyroid cancer
biomarkers
inhibitors
therapy
url https://www.frontiersin.org/articles/10.3389/fonc.2025.1549477/full
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