Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?
Objectives. Acute acquired comitant esotropia (AACE) can be a diagnostic challenge for ophthalmologists and neurologists because of its association with neurological pathologies. Our study describes a series of adult patients with AACE of undetermined etiology. Methods. Data on the clinical findings...
Saved in:
| Main Authors: | , |
|---|---|
| Format: | Article |
| Language: | English |
| Published: |
Wiley
2016-01-01
|
| Series: | Journal of Ophthalmology |
| Online Access: | http://dx.doi.org/10.1155/2016/2856128 |
| Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
| _version_ | 1832548158667751424 |
|---|---|
| author | Kadriye Erkan Turan Tulay Kansu |
| author_facet | Kadriye Erkan Turan Tulay Kansu |
| author_sort | Kadriye Erkan Turan |
| collection | DOAJ |
| description | Objectives. Acute acquired comitant esotropia (AACE) can be a diagnostic challenge for ophthalmologists and neurologists because of its association with neurological pathologies. Our study describes a series of adult patients with AACE of undetermined etiology. Methods. Data on the clinical findings of patients presented with AACE of undetermined etiology with a minimum follow-up of 1 year were retrieved from the medical records and the results analyzed. Results. A series of 9 esotropia cases (age range: 20–43 years) was reviewed. All patients had full duction and versions, without an A-pattern or V-pattern. All patients had esotropia for distance and near. Neurological evaluation in all cases was normal. Among patients, 3 were treated with prisms, 4 were treated with strabismus surgery, and 1 was treated with botulinum toxin injections; 1 patient declined treatment. In treated patients posttreatment sensory testing indicated restoration of binocularity that remained stable throughout follow-up of 1–9 years. The patient that declined treatment had binocular function with base-out prisms. Conclusion. Acute onset esotropia may be seen without a neurological pathology in adults. Good motor and sensory outcomes can be achieved in these patients with AACE of undetermined etiology via surgical and nonsurgical methods. |
| format | Article |
| id | doaj-art-eb60635e235b424980e7c72c6d58445d |
| institution | Kabale University |
| issn | 2090-004X 2090-0058 |
| language | English |
| publishDate | 2016-01-01 |
| publisher | Wiley |
| record_format | Article |
| series | Journal of Ophthalmology |
| spelling | doaj-art-eb60635e235b424980e7c72c6d58445d2025-02-03T06:42:00ZengWileyJournal of Ophthalmology2090-004X2090-00582016-01-01201610.1155/2016/28561282856128Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?Kadriye Erkan Turan0Tulay Kansu1Department of Ophthalmology, Faculty of Medicine, Hacettepe University, Ankara, TurkeyDepartment of Neurology, Faculty of Medicine, Hacettepe University, Ankara, TurkeyObjectives. Acute acquired comitant esotropia (AACE) can be a diagnostic challenge for ophthalmologists and neurologists because of its association with neurological pathologies. Our study describes a series of adult patients with AACE of undetermined etiology. Methods. Data on the clinical findings of patients presented with AACE of undetermined etiology with a minimum follow-up of 1 year were retrieved from the medical records and the results analyzed. Results. A series of 9 esotropia cases (age range: 20–43 years) was reviewed. All patients had full duction and versions, without an A-pattern or V-pattern. All patients had esotropia for distance and near. Neurological evaluation in all cases was normal. Among patients, 3 were treated with prisms, 4 were treated with strabismus surgery, and 1 was treated with botulinum toxin injections; 1 patient declined treatment. In treated patients posttreatment sensory testing indicated restoration of binocularity that remained stable throughout follow-up of 1–9 years. The patient that declined treatment had binocular function with base-out prisms. Conclusion. Acute onset esotropia may be seen without a neurological pathology in adults. Good motor and sensory outcomes can be achieved in these patients with AACE of undetermined etiology via surgical and nonsurgical methods.http://dx.doi.org/10.1155/2016/2856128 |
| spellingShingle | Kadriye Erkan Turan Tulay Kansu Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not? Journal of Ophthalmology |
| title | Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not? |
| title_full | Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not? |
| title_fullStr | Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not? |
| title_full_unstemmed | Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not? |
| title_short | Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not? |
| title_sort | acute acquired comitant esotropia in adults is it neurologic or not |
| url | http://dx.doi.org/10.1155/2016/2856128 |
| work_keys_str_mv | AT kadriyeerkanturan acuteacquiredcomitantesotropiainadultsisitneurologicornot AT tulaykansu acuteacquiredcomitantesotropiainadultsisitneurologicornot |