The myocardial remodeling at ischemic chronic heart failure: the features of Ivabradine influence in experimental model

Aim. Experimental study of Ivabradine influence on myocardial remodeling in patients with ischemic chronic heart failure. Material and Methods. The ischemic model of heart failure (HF) was modeled in 30 mature rats by isoproterenol injection. All animals were divided into 3 groups (10 each): I –...

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Main Authors: S. V. Fedorov, I. V. Kozlova, G. P. Gamorak, A. S. Geraschenko
Format: Article
Language:English
Published: Zaporizhzhia State Medical and Pharmaceutical University 2016-12-01
Series:Zaporožskij Medicinskij Žurnal
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Online Access:http://zmj.zsmu.edu.ua/article/view/85500/83149
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author S. V. Fedorov
I. V. Kozlova
G. P. Gamorak
A. S. Geraschenko
author_facet S. V. Fedorov
I. V. Kozlova
G. P. Gamorak
A. S. Geraschenko
author_sort S. V. Fedorov
collection DOAJ
description Aim. Experimental study of Ivabradine influence on myocardial remodeling in patients with ischemic chronic heart failure. Material and Methods. The ischemic model of heart failure (HF) was modeled in 30 mature rats by isoproterenol injection. All animals were divided into 3 groups (10 each): I – control; II – with model of HF; III – with HF model and Ivabradine usage. For calculation of metric characteristics of the cardiomyocytes UTHSCSA Image Tool® for Windows® (version 2.00) program equipment in interactive regime was used. Morphometric analysis was made by point-based calculation method with 72-network. Statistical analysis was made by standard program equipment “Statistica for Windows12.0” (StatSoft,Tulsa,OK,USA). Results. In experimental animals with model of ischemic HF the substantial hypertrophy of cardiomyocytes was observed. This process was characterized by increase in cardiomyocytes cross-sectional area, cross-sectional area of nuclei, decrease of nucleus-to-cytoplasm ratio and some discoordination of this parameter. In case of Ivabradine prescription we have noted reduction in myocardial hypertrophy processes with the following sings: decrease in mean cardiomyocytes cross-sectional area in 1.31 times (p<0,05), and mean cross-sectional area of nuclei – in 1.41 times (p<0,05). Conclusions. 1. In case of ischemic heart failure the substantial hypertrophy of cardiomyocytes with increase in mean parameters of their nuclei volume and nucleus-to-cytoplasm ratio disturbances were observed. 2. Ivabradine caused a mild regression of myocardial remodeling, as evidenced by a decrease in the degree of cardiomyocytes and their nuclei hypertrophy, severity of parenchyma dystrophy and fibrosis of heart muscle.
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spelling doaj-art-e5aa4a6c46ba4cbbbbfb26b47a02b0f42025-08-20T03:26:16ZengZaporizhzhia State Medical and Pharmaceutical UniversityZaporožskij Medicinskij Žurnal2306-41452310-12102016-12-0163943http://dx.doi.org/10.14739/2310-1210.2016.6.85500The myocardial remodeling at ischemic chronic heart failure: the features of Ivabradine influence in experimental modelS. V. FedorovI. V. KozlovaG. P. GamorakA. S. GeraschenkoAim. Experimental study of Ivabradine influence on myocardial remodeling in patients with ischemic chronic heart failure. Material and Methods. The ischemic model of heart failure (HF) was modeled in 30 mature rats by isoproterenol injection. All animals were divided into 3 groups (10 each): I – control; II – with model of HF; III – with HF model and Ivabradine usage. For calculation of metric characteristics of the cardiomyocytes UTHSCSA Image Tool® for Windows® (version 2.00) program equipment in interactive regime was used. Morphometric analysis was made by point-based calculation method with 72-network. Statistical analysis was made by standard program equipment “Statistica for Windows12.0” (StatSoft,Tulsa,OK,USA). Results. In experimental animals with model of ischemic HF the substantial hypertrophy of cardiomyocytes was observed. This process was characterized by increase in cardiomyocytes cross-sectional area, cross-sectional area of nuclei, decrease of nucleus-to-cytoplasm ratio and some discoordination of this parameter. In case of Ivabradine prescription we have noted reduction in myocardial hypertrophy processes with the following sings: decrease in mean cardiomyocytes cross-sectional area in 1.31 times (p<0,05), and mean cross-sectional area of nuclei – in 1.41 times (p<0,05). Conclusions. 1. In case of ischemic heart failure the substantial hypertrophy of cardiomyocytes with increase in mean parameters of their nuclei volume and nucleus-to-cytoplasm ratio disturbances were observed. 2. Ivabradine caused a mild regression of myocardial remodeling, as evidenced by a decrease in the degree of cardiomyocytes and their nuclei hypertrophy, severity of parenchyma dystrophy and fibrosis of heart muscle.http://zmj.zsmu.edu.ua/article/view/85500/83149chronic heart failuremyocardial remodelingexperimental modelivabradine
spellingShingle S. V. Fedorov
I. V. Kozlova
G. P. Gamorak
A. S. Geraschenko
The myocardial remodeling at ischemic chronic heart failure: the features of Ivabradine influence in experimental model
Zaporožskij Medicinskij Žurnal
chronic heart failure
myocardial remodeling
experimental model
ivabradine
title The myocardial remodeling at ischemic chronic heart failure: the features of Ivabradine influence in experimental model
title_full The myocardial remodeling at ischemic chronic heart failure: the features of Ivabradine influence in experimental model
title_fullStr The myocardial remodeling at ischemic chronic heart failure: the features of Ivabradine influence in experimental model
title_full_unstemmed The myocardial remodeling at ischemic chronic heart failure: the features of Ivabradine influence in experimental model
title_short The myocardial remodeling at ischemic chronic heart failure: the features of Ivabradine influence in experimental model
title_sort myocardial remodeling at ischemic chronic heart failure the features of ivabradine influence in experimental model
topic chronic heart failure
myocardial remodeling
experimental model
ivabradine
url http://zmj.zsmu.edu.ua/article/view/85500/83149
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