Effect of Pro12Ala polymorphism of PPARG gene on lipid peroxidation and antioxidant defense in patients with type 2 diabetes depending on the duration of the disease

Aim – to study the impact of polymorphic marker rs1801282 of gene PPARG on the formation of oxidative stress in terms of LPO and AOS in patients with type 2 diabetes depending on the duration of the disease. Materials and methods. The study was conducted involving 138 patients, 88 of whom were pa...

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Main Authors: V. Y. Mokrii, S. V. Ziablytsev
Format: Article
Language:English
Published: Zaporizhzhia State Medical and Pharmaceutical University 2016-08-01
Series:Patologìâ
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Online Access:http://pat.zsmu.edu.ua/article/view/81332/78226
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author V. Y. Mokrii
S. V. Ziablytsev
author_facet V. Y. Mokrii
S. V. Ziablytsev
author_sort V. Y. Mokrii
collection DOAJ
description Aim – to study the impact of polymorphic marker rs1801282 of gene PPARG on the formation of oxidative stress in terms of LPO and AOS in patients with type 2 diabetes depending on the duration of the disease. Materials and methods. The study was conducted involving 138 patients, 88 of whom were patients with type 2 diabetes and the control group consisted of 50 people. Patients were divided into three groups according to disease duration: 5 years, 5-10 years and more than 10 years. LPO activity was assessed in terms of diene conjugates (DC) and malonic dialdehyde (MDA) and AOC status - the activity of superoxide dismutase (SOD), catalase and the level of α-tocopherol (α-TF). Molecular genetic studies were conducted by the method of PCR in real time, and to analyze polymorphic DNA loci a standardized test system TaqMan Mutation Detection Assays Life-Technology (USA) was used. Results. During 5-10 years disease levels of DC and MDA in patients with Pro12Ala genotype were increased by 34,9% and 34,7%, as compared to Pro12Pro (p=0,01). Availability of Pro12Pro stipulated the reduction of catalase activity during 5-10 years disease by 75% (p=0,001), and for those, who are ill for more than 10 years by 2,04 times (p=0,01), which is not different from the reference level (F=1,19; p=0,600), but in the case of Pro12Ala, this figure was 2 times higher. Conclusions. Pro12Ala polymorphism (rs1801282) of PPARG gene causes the development of oxidative stress in type 2 diabetes with 5-10 years durations, and genotype Pro12Pro - lack of the catalase enzyme level of antioxidant system in patients with durations of disease more than 5 years.
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spelling doaj-art-e555fea036e0474a981a1cd1e1ffb2c82025-08-20T02:04:30ZengZaporizhzhia State Medical and Pharmaceutical UniversityPatologìâ2306-80272310-12372016-08-012525710.14739/2310-1237.2016.2.81332Effect of Pro12Ala polymorphism of PPARG gene on lipid peroxidation and antioxidant defense in patients with type 2 diabetes depending on the duration of the diseaseV. Y. MokriiS. V. ZiablytsevAim – to study the impact of polymorphic marker rs1801282 of gene PPARG on the formation of oxidative stress in terms of LPO and AOS in patients with type 2 diabetes depending on the duration of the disease. Materials and methods. The study was conducted involving 138 patients, 88 of whom were patients with type 2 diabetes and the control group consisted of 50 people. Patients were divided into three groups according to disease duration: 5 years, 5-10 years and more than 10 years. LPO activity was assessed in terms of diene conjugates (DC) and malonic dialdehyde (MDA) and AOC status - the activity of superoxide dismutase (SOD), catalase and the level of α-tocopherol (α-TF). Molecular genetic studies were conducted by the method of PCR in real time, and to analyze polymorphic DNA loci a standardized test system TaqMan Mutation Detection Assays Life-Technology (USA) was used. Results. During 5-10 years disease levels of DC and MDA in patients with Pro12Ala genotype were increased by 34,9% and 34,7%, as compared to Pro12Pro (p=0,01). Availability of Pro12Pro stipulated the reduction of catalase activity during 5-10 years disease by 75% (p=0,001), and for those, who are ill for more than 10 years by 2,04 times (p=0,01), which is not different from the reference level (F=1,19; p=0,600), but in the case of Pro12Ala, this figure was 2 times higher. Conclusions. Pro12Ala polymorphism (rs1801282) of PPARG gene causes the development of oxidative stress in type 2 diabetes with 5-10 years durations, and genotype Pro12Pro - lack of the catalase enzyme level of antioxidant system in patients with durations of disease more than 5 years.http://pat.zsmu.edu.ua/article/view/81332/78226Pro12Ala polymorphism of PPARG genetype 2 diabetesoxidative stress
spellingShingle V. Y. Mokrii
S. V. Ziablytsev
Effect of Pro12Ala polymorphism of PPARG gene on lipid peroxidation and antioxidant defense in patients with type 2 diabetes depending on the duration of the disease
Patologìâ
Pro12Ala polymorphism of PPARG gene
type 2 diabetes
oxidative stress
title Effect of Pro12Ala polymorphism of PPARG gene on lipid peroxidation and antioxidant defense in patients with type 2 diabetes depending on the duration of the disease
title_full Effect of Pro12Ala polymorphism of PPARG gene on lipid peroxidation and antioxidant defense in patients with type 2 diabetes depending on the duration of the disease
title_fullStr Effect of Pro12Ala polymorphism of PPARG gene on lipid peroxidation and antioxidant defense in patients with type 2 diabetes depending on the duration of the disease
title_full_unstemmed Effect of Pro12Ala polymorphism of PPARG gene on lipid peroxidation and antioxidant defense in patients with type 2 diabetes depending on the duration of the disease
title_short Effect of Pro12Ala polymorphism of PPARG gene on lipid peroxidation and antioxidant defense in patients with type 2 diabetes depending on the duration of the disease
title_sort effect of pro12ala polymorphism of pparg gene on lipid peroxidation and antioxidant defense in patients with type 2 diabetes depending on the duration of the disease
topic Pro12Ala polymorphism of PPARG gene
type 2 diabetes
oxidative stress
url http://pat.zsmu.edu.ua/article/view/81332/78226
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AT svziablytsev effectofpro12alapolymorphismofpparggeneonlipidperoxidationandantioxidantdefenseinpatientswithtype2diabetesdependingonthedurationofthedisease