Insightful chemistry of molecular docking studies and ADMET profiling of 4-amino-1,2,4-triazole scaffolds as potential inhibitor of DprE1 enzymes as antitubercular agents

Abstract Background Airborne transmission of the bacterial illness tuberculosis (TB) is attributed to Mycobacterium tuberculosis (Mtb). Mycobacterium tuberculosis, a multidrug-resistant strain, spreads quickly and is the cause of multidrug-resistant tuberculosis (MDRTB). A study was conducted to inv...

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Main Authors: Santosh Prasad Chaudhary Kurmi, Dipanjan Karati
Format: Article
Language:English
Published: Springer 2025-06-01
Series:Discover Chemistry
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Online Access:https://doi.org/10.1007/s44371-025-00208-w
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author Santosh Prasad Chaudhary Kurmi
Dipanjan Karati
author_facet Santosh Prasad Chaudhary Kurmi
Dipanjan Karati
author_sort Santosh Prasad Chaudhary Kurmi
collection DOAJ
description Abstract Background Airborne transmission of the bacterial illness tuberculosis (TB) is attributed to Mycobacterium tuberculosis (Mtb). Mycobacterium tuberculosis, a multidrug-resistant strain, spreads quickly and is the cause of multidrug-resistant tuberculosis (MDRTB). A study was conducted to investigate the possible antitubercular interactions between produced 4-amino-1,2,4-triazole scaffolds and the DprE1 enzyme in mycobacterium. Methods The binding affinity of ligands with the target PDB ID 6hfw was predicted using Pyrex 0.8 version software. Normal mode analysis and RMSF of protein–ligand complexes were performed using the imods server and CABSflex server. The Stoptox and Pkcsm servers were used to forecast the drug-likeness and toxicity of ligands. Results C20 has a significant negative binding energy of −10.4 kcal/mol and eight conventional hydrogen bonds, indicating a strong binding affinity for DprE1. In contrast, the reference medication isoniazid has a significantly lower binding affinity with two conventional hydrogen bonds of −5.5 kcal/mol. The binding energies of the remaining compounds, C21, C22, and C23, were −10.3 kcal/mol, correspondingly. Additionally, the absorption, distribution, metabolism, and excretion (ADME) features of designed compounds were assessed, showing the possibility for further development into more effective anti-tubercular medicines. Conclusion As a result, compounds C20, C21, C22, C23, and C24 are the best candidates as DprE1 enzyme protein inhibitors; however, in-vitro animal studies and toxicity studies are required to confirm these findings. Graphical Abstract
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spelling doaj-art-e4c84537856442eaa06816eca3058f9a2025-08-20T02:07:45ZengSpringerDiscover Chemistry3005-11932025-06-012111910.1007/s44371-025-00208-wInsightful chemistry of molecular docking studies and ADMET profiling of 4-amino-1,2,4-triazole scaffolds as potential inhibitor of DprE1 enzymes as antitubercular agentsSantosh Prasad Chaudhary Kurmi0Dipanjan Karati1Department of Pharmacy, Universal College of Medical SciencesDepartment of Pharmaceutical Technology, School of Pharmacy, Techno India University, West BengalAbstract Background Airborne transmission of the bacterial illness tuberculosis (TB) is attributed to Mycobacterium tuberculosis (Mtb). Mycobacterium tuberculosis, a multidrug-resistant strain, spreads quickly and is the cause of multidrug-resistant tuberculosis (MDRTB). A study was conducted to investigate the possible antitubercular interactions between produced 4-amino-1,2,4-triazole scaffolds and the DprE1 enzyme in mycobacterium. Methods The binding affinity of ligands with the target PDB ID 6hfw was predicted using Pyrex 0.8 version software. Normal mode analysis and RMSF of protein–ligand complexes were performed using the imods server and CABSflex server. The Stoptox and Pkcsm servers were used to forecast the drug-likeness and toxicity of ligands. Results C20 has a significant negative binding energy of −10.4 kcal/mol and eight conventional hydrogen bonds, indicating a strong binding affinity for DprE1. In contrast, the reference medication isoniazid has a significantly lower binding affinity with two conventional hydrogen bonds of −5.5 kcal/mol. The binding energies of the remaining compounds, C21, C22, and C23, were −10.3 kcal/mol, correspondingly. Additionally, the absorption, distribution, metabolism, and excretion (ADME) features of designed compounds were assessed, showing the possibility for further development into more effective anti-tubercular medicines. Conclusion As a result, compounds C20, C21, C22, C23, and C24 are the best candidates as DprE1 enzyme protein inhibitors; however, in-vitro animal studies and toxicity studies are required to confirm these findings. Graphical Abstracthttps://doi.org/10.1007/s44371-025-00208-wDprE1 enzymeTuberculosisToxicity predictionRetrobiocat routeBioactivity radar analysis
spellingShingle Santosh Prasad Chaudhary Kurmi
Dipanjan Karati
Insightful chemistry of molecular docking studies and ADMET profiling of 4-amino-1,2,4-triazole scaffolds as potential inhibitor of DprE1 enzymes as antitubercular agents
Discover Chemistry
DprE1 enzyme
Tuberculosis
Toxicity prediction
Retrobiocat route
Bioactivity radar analysis
title Insightful chemistry of molecular docking studies and ADMET profiling of 4-amino-1,2,4-triazole scaffolds as potential inhibitor of DprE1 enzymes as antitubercular agents
title_full Insightful chemistry of molecular docking studies and ADMET profiling of 4-amino-1,2,4-triazole scaffolds as potential inhibitor of DprE1 enzymes as antitubercular agents
title_fullStr Insightful chemistry of molecular docking studies and ADMET profiling of 4-amino-1,2,4-triazole scaffolds as potential inhibitor of DprE1 enzymes as antitubercular agents
title_full_unstemmed Insightful chemistry of molecular docking studies and ADMET profiling of 4-amino-1,2,4-triazole scaffolds as potential inhibitor of DprE1 enzymes as antitubercular agents
title_short Insightful chemistry of molecular docking studies and ADMET profiling of 4-amino-1,2,4-triazole scaffolds as potential inhibitor of DprE1 enzymes as antitubercular agents
title_sort insightful chemistry of molecular docking studies and admet profiling of 4 amino 1 2 4 triazole scaffolds as potential inhibitor of dpre1 enzymes as antitubercular agents
topic DprE1 enzyme
Tuberculosis
Toxicity prediction
Retrobiocat route
Bioactivity radar analysis
url https://doi.org/10.1007/s44371-025-00208-w
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