Hypertrophic Cardiomyopathy Through the Lens of Mitochondria

The mechanisms of pathogenesis of hypertrophic cardiomyopathy are associated with mutations in the sarcomere genes of cardiomyocytes and metabolic disorders of the cell, including mitochondrial dysfunction. Mitochondria are characterized by the presence of their own DNA and enzyme complexes involved...

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Main Authors: Tatiana V. Kirichenko, Ivan V. Zhivodernikov, Maria A. Kozlova, Alexander M. Markin, Vasily V. Sinyov, Yuliya V. Markina
Format: Article
Language:English
Published: MDPI AG 2025-02-01
Series:Biomedicines
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Online Access:https://www.mdpi.com/2227-9059/13/3/591
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author Tatiana V. Kirichenko
Ivan V. Zhivodernikov
Maria A. Kozlova
Alexander M. Markin
Vasily V. Sinyov
Yuliya V. Markina
author_facet Tatiana V. Kirichenko
Ivan V. Zhivodernikov
Maria A. Kozlova
Alexander M. Markin
Vasily V. Sinyov
Yuliya V. Markina
author_sort Tatiana V. Kirichenko
collection DOAJ
description The mechanisms of pathogenesis of hypertrophic cardiomyopathy are associated with mutations in the sarcomere genes of cardiomyocytes and metabolic disorders of the cell, including mitochondrial dysfunction. Mitochondria are characterized by the presence of their own DNA and enzyme complexes involved in oxidative reactions, which cause damage to mitochondrial protein structures and membranes by reactive oxygen species. Mitochondrial dysfunctions can also be associated with mutations in the genes encoding mitochondrial proteins and lead to a violation of protective functions such as mitophagy, mitochondrial fusion, and fission. Mutations in myofibril proteins can negatively affect mitochondria through increased oxidative stress due to an increased need for ATP. Mitochondrial dysfunction is associated with impaired ATP synthesis and cardiac contractility, leading to clinical manifestations of hypertrophic cardiomyopathy. The current review was designed to characterize the role of mitochondria in the pathogenesis of hypertrophic cardiomyopathy based on published data; the search for publications was based on the analysis of articles including the keywords “hypertrophic cardiomyopathy, mitochondria, dysfunction” in the PubMed and Scopus databases up to January 2025.
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series Biomedicines
spelling doaj-art-df0c8d372de6430abff1856c3f7b2c572025-08-20T02:42:35ZengMDPI AGBiomedicines2227-90592025-02-0113359110.3390/biomedicines13030591Hypertrophic Cardiomyopathy Through the Lens of MitochondriaTatiana V. Kirichenko0Ivan V. Zhivodernikov1Maria A. Kozlova2Alexander M. Markin3Vasily V. Sinyov4Yuliya V. Markina5Petrovsky National Research Centre of Surgery, 119435 Moscow, RussiaPetrovsky National Research Centre of Surgery, 119435 Moscow, RussiaPetrovsky National Research Centre of Surgery, 119435 Moscow, RussiaPetrovsky National Research Centre of Surgery, 119435 Moscow, RussiaPetrovsky National Research Centre of Surgery, 119435 Moscow, RussiaPetrovsky National Research Centre of Surgery, 119435 Moscow, RussiaThe mechanisms of pathogenesis of hypertrophic cardiomyopathy are associated with mutations in the sarcomere genes of cardiomyocytes and metabolic disorders of the cell, including mitochondrial dysfunction. Mitochondria are characterized by the presence of their own DNA and enzyme complexes involved in oxidative reactions, which cause damage to mitochondrial protein structures and membranes by reactive oxygen species. Mitochondrial dysfunctions can also be associated with mutations in the genes encoding mitochondrial proteins and lead to a violation of protective functions such as mitophagy, mitochondrial fusion, and fission. Mutations in myofibril proteins can negatively affect mitochondria through increased oxidative stress due to an increased need for ATP. Mitochondrial dysfunction is associated with impaired ATP synthesis and cardiac contractility, leading to clinical manifestations of hypertrophic cardiomyopathy. The current review was designed to characterize the role of mitochondria in the pathogenesis of hypertrophic cardiomyopathy based on published data; the search for publications was based on the analysis of articles including the keywords “hypertrophic cardiomyopathy, mitochondria, dysfunction” in the PubMed and Scopus databases up to January 2025.https://www.mdpi.com/2227-9059/13/3/591hypertrophic cardiomyopathymitochondrial dysfunctionoxidative stress
spellingShingle Tatiana V. Kirichenko
Ivan V. Zhivodernikov
Maria A. Kozlova
Alexander M. Markin
Vasily V. Sinyov
Yuliya V. Markina
Hypertrophic Cardiomyopathy Through the Lens of Mitochondria
Biomedicines
hypertrophic cardiomyopathy
mitochondrial dysfunction
oxidative stress
title Hypertrophic Cardiomyopathy Through the Lens of Mitochondria
title_full Hypertrophic Cardiomyopathy Through the Lens of Mitochondria
title_fullStr Hypertrophic Cardiomyopathy Through the Lens of Mitochondria
title_full_unstemmed Hypertrophic Cardiomyopathy Through the Lens of Mitochondria
title_short Hypertrophic Cardiomyopathy Through the Lens of Mitochondria
title_sort hypertrophic cardiomyopathy through the lens of mitochondria
topic hypertrophic cardiomyopathy
mitochondrial dysfunction
oxidative stress
url https://www.mdpi.com/2227-9059/13/3/591
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AT ivanvzhivodernikov hypertrophiccardiomyopathythroughthelensofmitochondria
AT mariaakozlova hypertrophiccardiomyopathythroughthelensofmitochondria
AT alexandermmarkin hypertrophiccardiomyopathythroughthelensofmitochondria
AT vasilyvsinyov hypertrophiccardiomyopathythroughthelensofmitochondria
AT yuliyavmarkina hypertrophiccardiomyopathythroughthelensofmitochondria