Inflammatory Response, a Key Pathophysiological Mechanism of Obesity-Induced Depression
In recent years, with the acceleration of life rhythm and the increase of social competition, the incidence of obesity and depression has been increasing, which has seriously affected the quality of life and health of people. Obesity and depression, two seemingly unrelated physical and psychological...
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| Main Authors: | , , , |
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| Format: | Article |
| Language: | English |
| Published: |
Wiley
2020-01-01
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| Series: | Mediators of Inflammation |
| Online Access: | http://dx.doi.org/10.1155/2020/8893892 |
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| _version_ | 1832552814575878144 |
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| author | Shu-Lin Shao Da-Qian Liu Bo Zhang Shu-Ming Huang |
| author_facet | Shu-Lin Shao Da-Qian Liu Bo Zhang Shu-Ming Huang |
| author_sort | Shu-Lin Shao |
| collection | DOAJ |
| description | In recent years, with the acceleration of life rhythm and the increase of social competition, the incidence of obesity and depression has been increasing, which has seriously affected the quality of life and health of people. Obesity and depression, two seemingly unrelated physical and psychological diseases, in fact, are closely related: obese people are more likely to have depression than nonobese ones. We have reviewed and analyzed the relevant research literature and found that the inflammatory response plays a key role in obesity-induced depression. This article will discuss in detail the inflammatory mechanisms by which obesity induces depression. |
| format | Article |
| id | doaj-art-dc52e4685f59464da95faf4282f64d93 |
| institution | Kabale University |
| issn | 0962-9351 1466-1861 |
| language | English |
| publishDate | 2020-01-01 |
| publisher | Wiley |
| record_format | Article |
| series | Mediators of Inflammation |
| spelling | doaj-art-dc52e4685f59464da95faf4282f64d932025-02-03T05:57:51ZengWileyMediators of Inflammation0962-93511466-18612020-01-01202010.1155/2020/88938928893892Inflammatory Response, a Key Pathophysiological Mechanism of Obesity-Induced DepressionShu-Lin Shao0Da-Qian Liu1Bo Zhang2Shu-Ming Huang3Department of Neuroscience, Institute for Chinese Medicine, Heilongjiang University of Chinese Medicine, Harbin, ChinaDepartment of Orthopedics, The Second Affiliated Hospital of Harbin Medical University, Harbin 150001, ChinaDepartment of Neuroscience, Institute for Chinese Medicine, Heilongjiang University of Chinese Medicine, Harbin, ChinaDepartment of Neuroscience, Institute for Chinese Medicine, Heilongjiang University of Chinese Medicine, Harbin, ChinaIn recent years, with the acceleration of life rhythm and the increase of social competition, the incidence of obesity and depression has been increasing, which has seriously affected the quality of life and health of people. Obesity and depression, two seemingly unrelated physical and psychological diseases, in fact, are closely related: obese people are more likely to have depression than nonobese ones. We have reviewed and analyzed the relevant research literature and found that the inflammatory response plays a key role in obesity-induced depression. This article will discuss in detail the inflammatory mechanisms by which obesity induces depression.http://dx.doi.org/10.1155/2020/8893892 |
| spellingShingle | Shu-Lin Shao Da-Qian Liu Bo Zhang Shu-Ming Huang Inflammatory Response, a Key Pathophysiological Mechanism of Obesity-Induced Depression Mediators of Inflammation |
| title | Inflammatory Response, a Key Pathophysiological Mechanism of Obesity-Induced Depression |
| title_full | Inflammatory Response, a Key Pathophysiological Mechanism of Obesity-Induced Depression |
| title_fullStr | Inflammatory Response, a Key Pathophysiological Mechanism of Obesity-Induced Depression |
| title_full_unstemmed | Inflammatory Response, a Key Pathophysiological Mechanism of Obesity-Induced Depression |
| title_short | Inflammatory Response, a Key Pathophysiological Mechanism of Obesity-Induced Depression |
| title_sort | inflammatory response a key pathophysiological mechanism of obesity induced depression |
| url | http://dx.doi.org/10.1155/2020/8893892 |
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