Abdominal attack in a patient with hereditary angioedema due to C1 inhibitor deficiency complicated by a perforated peptic ulcer
A 30-year-old male with hereditary angioedema (HAE) due to C1 inhibitor deficiency was admitted to the Emergency Department (ED) due to severe abdominal pain lasting for 3 hs with severe bloating and weakness. pdC1INH and fluids were applied as usual and the patient was discharged with feeling bette...
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Main Authors: | , , , , |
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Format: | Article |
Language: | English |
Published: |
Termedia Publishing House
2024-02-01
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Series: | Alergologia Polska |
Subjects: | |
Online Access: | https://www.termedia.pl/Abdominal-attack-in-a-patient-with-hereditary-angioedema-due-to-C1-inhibitor-deficiency-complicated-by-a-perforated-peptic-ulcer,123,52432,1,1.html |
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Summary: | A 30-year-old male with hereditary angioedema (HAE) due to C1 inhibitor deficiency was admitted to the Emergency Department (ED) due to severe abdominal pain lasting for 3 hs with severe bloating and weakness. pdC1INH and fluids were applied as usual and the patient was discharged with feeling better. A few hours later abdominal symptoms recurred and erythema appeared, thus the allergy to antispasmodic drug taken by the patient was suspected. In the ED, the patient received anti-allergic treatment and was discharged again. After 3 h severe abdominal symptoms returned with fever. The patient was readmitted to the ED with peritoneal symptoms. Abdominal X-ray confirmed digestive tract perforation. Laparotomy revealed perforation of the chronic duodenal ulcer. In conclusion, we indicate that a HAE patient with an abdominal pain not resolving after usually effective treatment, should be hospitalized and monitored. Differential diagnosis of reasons other than HAE attack should be made. |
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ISSN: | 2353-3854 2391-6052 |