The Size of the Human Proteome: The Width and Depth

This work discusses bioinformatics and experimental approaches to explore the human proteome, a constellation of proteins expressed in different tissues and organs. As the human proteome is not a static entity, it seems necessary to estimate the number of different protein species (proteoforms) and...

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Main Authors: Elena A. Ponomarenko, Ekaterina V. Poverennaya, Ekaterina V. Ilgisonis, Mikhail A. Pyatnitskiy, Arthur T. Kopylov, Victor G. Zgoda, Andrey V. Lisitsa, Alexander I. Archakov
Format: Article
Language:English
Published: Wiley 2016-01-01
Series:International Journal of Analytical Chemistry
Online Access:http://dx.doi.org/10.1155/2016/7436849
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author Elena A. Ponomarenko
Ekaterina V. Poverennaya
Ekaterina V. Ilgisonis
Mikhail A. Pyatnitskiy
Arthur T. Kopylov
Victor G. Zgoda
Andrey V. Lisitsa
Alexander I. Archakov
author_facet Elena A. Ponomarenko
Ekaterina V. Poverennaya
Ekaterina V. Ilgisonis
Mikhail A. Pyatnitskiy
Arthur T. Kopylov
Victor G. Zgoda
Andrey V. Lisitsa
Alexander I. Archakov
author_sort Elena A. Ponomarenko
collection DOAJ
description This work discusses bioinformatics and experimental approaches to explore the human proteome, a constellation of proteins expressed in different tissues and organs. As the human proteome is not a static entity, it seems necessary to estimate the number of different protein species (proteoforms) and measure the number of copies of the same protein in a specific tissue. Here, meta-analysis of neXtProt knowledge base is proposed for theoretical prediction of the number of different proteoforms that arise from alternative splicing (AS), single amino acid polymorphisms (SAPs), and posttranslational modifications (PTMs). Three possible cases are considered: (1) PTMs and SAPs appear exclusively in the canonical sequences of proteins, but not in splice variants; (2) PTMs and SAPs can occur in both proteins encoded by canonical sequences and in splice variants; (3) all modification types (AS, SAP, and PTM) occur as independent events. Experimental validation of proteoforms is limited by the analytical sensitivity of proteomic technology. A bell-shaped distribution histogram was generated for proteins encoded by a single chromosome, with the estimation of copy numbers in plasma, liver, and HepG2 cell line. The proposed metabioinformatics approaches can be used for estimation of the number of different proteoforms for any group of protein-coding genes.
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institution Kabale University
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spelling doaj-art-da6285a6970a492590c51f1d49e17f4d2025-08-20T03:24:25ZengWileyInternational Journal of Analytical Chemistry1687-87601687-87792016-01-01201610.1155/2016/74368497436849The Size of the Human Proteome: The Width and DepthElena A. Ponomarenko0Ekaterina V. Poverennaya1Ekaterina V. Ilgisonis2Mikhail A. Pyatnitskiy3Arthur T. Kopylov4Victor G. Zgoda5Andrey V. Lisitsa6Alexander I. Archakov7Institute of Biomedical Chemistry, Moscow 119121, RussiaInstitute of Biomedical Chemistry, Moscow 119121, RussiaInstitute of Biomedical Chemistry, Moscow 119121, RussiaInstitute of Biomedical Chemistry, Moscow 119121, RussiaInstitute of Biomedical Chemistry, Moscow 119121, RussiaInstitute of Biomedical Chemistry, Moscow 119121, RussiaInstitute of Biomedical Chemistry, Moscow 119121, RussiaInstitute of Biomedical Chemistry, Moscow 119121, RussiaThis work discusses bioinformatics and experimental approaches to explore the human proteome, a constellation of proteins expressed in different tissues and organs. As the human proteome is not a static entity, it seems necessary to estimate the number of different protein species (proteoforms) and measure the number of copies of the same protein in a specific tissue. Here, meta-analysis of neXtProt knowledge base is proposed for theoretical prediction of the number of different proteoforms that arise from alternative splicing (AS), single amino acid polymorphisms (SAPs), and posttranslational modifications (PTMs). Three possible cases are considered: (1) PTMs and SAPs appear exclusively in the canonical sequences of proteins, but not in splice variants; (2) PTMs and SAPs can occur in both proteins encoded by canonical sequences and in splice variants; (3) all modification types (AS, SAP, and PTM) occur as independent events. Experimental validation of proteoforms is limited by the analytical sensitivity of proteomic technology. A bell-shaped distribution histogram was generated for proteins encoded by a single chromosome, with the estimation of copy numbers in plasma, liver, and HepG2 cell line. The proposed metabioinformatics approaches can be used for estimation of the number of different proteoforms for any group of protein-coding genes.http://dx.doi.org/10.1155/2016/7436849
spellingShingle Elena A. Ponomarenko
Ekaterina V. Poverennaya
Ekaterina V. Ilgisonis
Mikhail A. Pyatnitskiy
Arthur T. Kopylov
Victor G. Zgoda
Andrey V. Lisitsa
Alexander I. Archakov
The Size of the Human Proteome: The Width and Depth
International Journal of Analytical Chemistry
title The Size of the Human Proteome: The Width and Depth
title_full The Size of the Human Proteome: The Width and Depth
title_fullStr The Size of the Human Proteome: The Width and Depth
title_full_unstemmed The Size of the Human Proteome: The Width and Depth
title_short The Size of the Human Proteome: The Width and Depth
title_sort size of the human proteome the width and depth
url http://dx.doi.org/10.1155/2016/7436849
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