Pore formation by the CDTb component of the Clostridioides difficile binary toxin is Ca2+-dependent

Abstract Clostridioides difficile infection (CDI) is one of the five most urgent bacterial threats in the United States. Furthermore, hypervirulent CDI strains express a third toxin termed the C. difficile binary toxin (CDT), and its molecular mechanism for entering host cells is not fully elucidate...

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Main Authors: Dinendra L. Abeyawardhane, Spiridon E. Sevdalis, Kaylin A. Adipietro, Raquel Godoy-Ruiz, Kristen M. Varney, Izza F. Nawaz, Alejandro X. Spittel, Daniel Hunter, Richard R. Rustandi, Vitalii I. Silin, Amedee des Georges, Mary E. Cook, Edwin Pozharski, David J. Weber
Format: Article
Language:English
Published: Nature Portfolio 2025-06-01
Series:Communications Biology
Online Access:https://doi.org/10.1038/s42003-025-08343-x
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author Dinendra L. Abeyawardhane
Spiridon E. Sevdalis
Kaylin A. Adipietro
Raquel Godoy-Ruiz
Kristen M. Varney
Izza F. Nawaz
Alejandro X. Spittel
Daniel Hunter
Richard R. Rustandi
Vitalii I. Silin
Amedee des Georges
Mary E. Cook
Edwin Pozharski
David J. Weber
author_facet Dinendra L. Abeyawardhane
Spiridon E. Sevdalis
Kaylin A. Adipietro
Raquel Godoy-Ruiz
Kristen M. Varney
Izza F. Nawaz
Alejandro X. Spittel
Daniel Hunter
Richard R. Rustandi
Vitalii I. Silin
Amedee des Georges
Mary E. Cook
Edwin Pozharski
David J. Weber
author_sort Dinendra L. Abeyawardhane
collection DOAJ
description Abstract Clostridioides difficile infection (CDI) is one of the five most urgent bacterial threats in the United States. Furthermore, hypervirulent CDI strains express a third toxin termed the C. difficile binary toxin (CDT), and its molecular mechanism for entering host cells is not fully elucidated. Like other AB-type binary toxins, CDT enters host cells via endosomes. Here we show via surface plasmon resonance and electrochemical impedance spectroscopy that the cell-binding component of CDT, termed CDTb, binds and form pores in lipid bilayers in the absence of its enzymatic component, CDTa. This occurs upon lowering free Ca2+ ion concentration, and not by decreasing pH, as found for other binary toxins (i.e., anthrax). Cryogenic electron microscopy (CryoEM), X-ray crystallography, and nuclear magnetic resonance (NMR) studies show that dissociation of Ca2+ from a single site in receptor binding domain 1 (RBD1) of CDTb triggers conformational exchange in CDTb. These and structure/function studies of a Ca2+-binding double mutant targeting RBD1 (i.e., D623A/D734A) support a model in which dissociation of Ca2+ from RBD1 induces dynamic properties in CDTb that enable it to bind and form pores in lipid bilayers.
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spelling doaj-art-d8d61c8064ea4fc6be04570da3fa8b2b2025-08-20T02:07:45ZengNature PortfolioCommunications Biology2399-36422025-06-018111410.1038/s42003-025-08343-xPore formation by the CDTb component of the Clostridioides difficile binary toxin is Ca2+-dependentDinendra L. Abeyawardhane0Spiridon E. Sevdalis1Kaylin A. Adipietro2Raquel Godoy-Ruiz3Kristen M. Varney4Izza F. Nawaz5Alejandro X. Spittel6Daniel Hunter7Richard R. Rustandi8Vitalii I. Silin9Amedee des Georges10Mary E. Cook11Edwin Pozharski12David J. Weber13Department of Biochemistry and Molecular Biology, University of Maryland School of MedicineDepartment of Biochemistry and Molecular Biology, University of Maryland School of MedicineDepartment of Biochemistry and Molecular Biology, University of Maryland School of MedicineDepartment of Biochemistry and Molecular Biology, University of Maryland School of MedicineDepartment of Biochemistry and Molecular Biology, University of Maryland School of MedicineDepartment of Biochemistry and Molecular Biology, University of Maryland School of MedicineDepartment of Biochemistry and Molecular Biology, University of Maryland School of MedicineDepartment of Biochemistry and Molecular Biology, University of Maryland School of MedicineMerck & Co. Inc.Institute for Bioscience and Biotechnology Research (IBBR), University of MarylandStructural Biology Initiative, CUNY Advanced Science Research CenterDepartment of Biochemistry and Molecular Biology, University of Maryland School of MedicineDepartment of Biochemistry and Molecular Biology, University of Maryland School of MedicineDepartment of Biochemistry and Molecular Biology, University of Maryland School of MedicineAbstract Clostridioides difficile infection (CDI) is one of the five most urgent bacterial threats in the United States. Furthermore, hypervirulent CDI strains express a third toxin termed the C. difficile binary toxin (CDT), and its molecular mechanism for entering host cells is not fully elucidated. Like other AB-type binary toxins, CDT enters host cells via endosomes. Here we show via surface plasmon resonance and electrochemical impedance spectroscopy that the cell-binding component of CDT, termed CDTb, binds and form pores in lipid bilayers in the absence of its enzymatic component, CDTa. This occurs upon lowering free Ca2+ ion concentration, and not by decreasing pH, as found for other binary toxins (i.e., anthrax). Cryogenic electron microscopy (CryoEM), X-ray crystallography, and nuclear magnetic resonance (NMR) studies show that dissociation of Ca2+ from a single site in receptor binding domain 1 (RBD1) of CDTb triggers conformational exchange in CDTb. These and structure/function studies of a Ca2+-binding double mutant targeting RBD1 (i.e., D623A/D734A) support a model in which dissociation of Ca2+ from RBD1 induces dynamic properties in CDTb that enable it to bind and form pores in lipid bilayers.https://doi.org/10.1038/s42003-025-08343-x
spellingShingle Dinendra L. Abeyawardhane
Spiridon E. Sevdalis
Kaylin A. Adipietro
Raquel Godoy-Ruiz
Kristen M. Varney
Izza F. Nawaz
Alejandro X. Spittel
Daniel Hunter
Richard R. Rustandi
Vitalii I. Silin
Amedee des Georges
Mary E. Cook
Edwin Pozharski
David J. Weber
Pore formation by the CDTb component of the Clostridioides difficile binary toxin is Ca2+-dependent
Communications Biology
title Pore formation by the CDTb component of the Clostridioides difficile binary toxin is Ca2+-dependent
title_full Pore formation by the CDTb component of the Clostridioides difficile binary toxin is Ca2+-dependent
title_fullStr Pore formation by the CDTb component of the Clostridioides difficile binary toxin is Ca2+-dependent
title_full_unstemmed Pore formation by the CDTb component of the Clostridioides difficile binary toxin is Ca2+-dependent
title_short Pore formation by the CDTb component of the Clostridioides difficile binary toxin is Ca2+-dependent
title_sort pore formation by the cdtb component of the clostridioides difficile binary toxin is ca2 dependent
url https://doi.org/10.1038/s42003-025-08343-x
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