Congenital Heart Disease and Its Impact on the Development of Anastomotic Strictures after Reconstruction of Esophageal Atresia
Background. The aim was to explore if severe congenital heart disease (CHD) influenced the need for dilatation of anastomotic strictures (AS) after the repair of esophageal atresia (EA). Methods. A retrospective case-control study was conducted examining AS in children with EA and Gross type C. The...
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| Format: | Article |
| Language: | English |
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Wiley
2018-01-01
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| Series: | Gastroenterology Research and Practice |
| Online Access: | http://dx.doi.org/10.1155/2018/6021014 |
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| author | Pernilla Stenström Martin Salö Magnus Anderberg Einar Arnbjörnsson |
| author_facet | Pernilla Stenström Martin Salö Magnus Anderberg Einar Arnbjörnsson |
| author_sort | Pernilla Stenström |
| collection | DOAJ |
| description | Background. The aim was to explore if severe congenital heart disease (CHD) influenced the need for dilatation of anastomotic strictures (AS) after the repair of esophageal atresia (EA). Methods. A retrospective case-control study was conducted examining AS in children with EA and Gross type C. The spectra of CHD and cardiac interventions were reviewed. The frequency of dilatations of AS during the first year following EA reconstruction was compared between children with and without severe CHD requiring cardiac surgery during their first year of life. Endoscopic signs of stricture were an indication for dilatation. Results. Included in the follow-up for AS were 94 patients who had EA reconstructions, of whom 10 (11%) children had severe CHD requiring surgery during the first year including 19 different cardiac interventions. In total, 38 patients needed dilatation of esophageal AS, distributed as six (60%) with severe CHD and 32 (38%) without severe CHD (p=0.31). Conclusion. Severe CHD was present in 11% of children with EA. Esophageal AS developed in 60% children with concomitant CHD, but although high, it did not reach statistical difference from children without CHD (38%). |
| format | Article |
| id | doaj-art-d86f3722026c4ee2b1989f66322109ee |
| institution | Kabale University |
| issn | 1687-6121 1687-630X |
| language | English |
| publishDate | 2018-01-01 |
| publisher | Wiley |
| record_format | Article |
| series | Gastroenterology Research and Practice |
| spelling | doaj-art-d86f3722026c4ee2b1989f66322109ee2025-02-03T00:59:23ZengWileyGastroenterology Research and Practice1687-61211687-630X2018-01-01201810.1155/2018/60210146021014Congenital Heart Disease and Its Impact on the Development of Anastomotic Strictures after Reconstruction of Esophageal AtresiaPernilla Stenström0Martin Salö1Magnus Anderberg2Einar Arnbjörnsson3Department of Pediatric Surgery, Lund University and Skåne University Hospital, Lund, SwedenDepartment of Pediatric Surgery, Lund University and Skåne University Hospital, Lund, SwedenDepartment of Pediatric Surgery, Lund University and Skåne University Hospital, Lund, SwedenDepartment of Pediatric Surgery, Lund University and Skåne University Hospital, Lund, SwedenBackground. The aim was to explore if severe congenital heart disease (CHD) influenced the need for dilatation of anastomotic strictures (AS) after the repair of esophageal atresia (EA). Methods. A retrospective case-control study was conducted examining AS in children with EA and Gross type C. The spectra of CHD and cardiac interventions were reviewed. The frequency of dilatations of AS during the first year following EA reconstruction was compared between children with and without severe CHD requiring cardiac surgery during their first year of life. Endoscopic signs of stricture were an indication for dilatation. Results. Included in the follow-up for AS were 94 patients who had EA reconstructions, of whom 10 (11%) children had severe CHD requiring surgery during the first year including 19 different cardiac interventions. In total, 38 patients needed dilatation of esophageal AS, distributed as six (60%) with severe CHD and 32 (38%) without severe CHD (p=0.31). Conclusion. Severe CHD was present in 11% of children with EA. Esophageal AS developed in 60% children with concomitant CHD, but although high, it did not reach statistical difference from children without CHD (38%).http://dx.doi.org/10.1155/2018/6021014 |
| spellingShingle | Pernilla Stenström Martin Salö Magnus Anderberg Einar Arnbjörnsson Congenital Heart Disease and Its Impact on the Development of Anastomotic Strictures after Reconstruction of Esophageal Atresia Gastroenterology Research and Practice |
| title | Congenital Heart Disease and Its Impact on the Development of Anastomotic Strictures after Reconstruction of Esophageal Atresia |
| title_full | Congenital Heart Disease and Its Impact on the Development of Anastomotic Strictures after Reconstruction of Esophageal Atresia |
| title_fullStr | Congenital Heart Disease and Its Impact on the Development of Anastomotic Strictures after Reconstruction of Esophageal Atresia |
| title_full_unstemmed | Congenital Heart Disease and Its Impact on the Development of Anastomotic Strictures after Reconstruction of Esophageal Atresia |
| title_short | Congenital Heart Disease and Its Impact on the Development of Anastomotic Strictures after Reconstruction of Esophageal Atresia |
| title_sort | congenital heart disease and its impact on the development of anastomotic strictures after reconstruction of esophageal atresia |
| url | http://dx.doi.org/10.1155/2018/6021014 |
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