Selenoprotein S Attenuates Tumor Necrosis Factor-α-Induced Dysfunction in Endothelial Cells
Endothelial dysfunction, partly induced by inflammatory mediators, is known to initiate and promote several cardiovascular diseases. Selenoprotein S (SelS) has been identified in endothelial cells and is associated with inflammation; however, its function in inflammation-induced endothelial dysfunct...
Saved in:
Main Authors: | Siyuan Cui, Lili Men, Yu Li, Yingshuo Zhong, Shanshan Yu, Fang Li, Jianling Du |
---|---|
Format: | Article |
Language: | English |
Published: |
Wiley
2018-01-01
|
Series: | Mediators of Inflammation |
Online Access: | http://dx.doi.org/10.1155/2018/1625414 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
A Possible Involvement of Ion Transporter in Tumor Necrosis Factor α and Cycloheximide-Induced Apoptosis of Endothelial Cells
by: Hiroshi Fujita, et al.
Published: (1999-01-01) -
Palmitic Acid Accelerates Endothelial Cell Injury and Cardiovascular Dysfunction via Palmitoylation of PKM2
by: Yu He, et al.
Published: (2025-02-01) -
Upregulation of Tumor Necrosis Factor-α-Induced Protein 8-Like 2 mRNA Is Negatively Correlated with Serum Concentrations of Tumor Necrosis Factor-α and Interleukin 6 in Type 2 Diabetes Mellitus
by: Yongliang Liu, et al.
Published: (2017-01-01) -
Interleukin-15 and Tumor Necrosis Factor-α in Iraqi Patients with Alopecia Areata
by: Zainab A. Kamil, et al.
Published: (2023-01-01) -
Dietary Magnesium Intake and Leukocyte Telomere Attrition in Adults: The Regulatory Role of Serum Tumor Necrosis Factor α
by: Jie Yu, et al.
Published: (2020-01-01)