CXCL4 Contributes to the Pathogenesis of Chronic Liver Allograft Dysfunction

Chronic liver allograft dysfunction (CLAD) remains the most common cause of patient morbidity and allograft loss in liver transplant patients. However, the pathogenesis of CLAD has not been completely elucidated. By establishing rat CLAD models, in this study, we identified the informative CLAD-asso...

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Main Authors: Jing Li, Bin Liu, Yuan Shi, Ke-Liang Xie, Hai-Fang Yin, Lu-nan Yan, Wan-yee Lau, Guo-Lin Wang
Format: Article
Language:English
Published: Wiley 2016-01-01
Series:Journal of Immunology Research
Online Access:http://dx.doi.org/10.1155/2016/9276986
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author Jing Li
Bin Liu
Yuan Shi
Ke-Liang Xie
Hai-Fang Yin
Lu-nan Yan
Wan-yee Lau
Guo-Lin Wang
author_facet Jing Li
Bin Liu
Yuan Shi
Ke-Liang Xie
Hai-Fang Yin
Lu-nan Yan
Wan-yee Lau
Guo-Lin Wang
author_sort Jing Li
collection DOAJ
description Chronic liver allograft dysfunction (CLAD) remains the most common cause of patient morbidity and allograft loss in liver transplant patients. However, the pathogenesis of CLAD has not been completely elucidated. By establishing rat CLAD models, in this study, we identified the informative CLAD-associated genes using isobaric tags for relative and absolute quantification (iTRAQ) proteomics analysis and validated these results in recipient rat liver allografts. CXCL4, CXCR3, EGFR, JAK2, STAT3, and Collagen IV were associated with CLAD pathogenesis. We validated that CXCL4 is upstream of these informative genes in the isolated hepatic stellate cells (HSC). Blocking CXCL4 protects against CLAD by reducing liver fibrosis. Therefore, our results indicated that therapeutic approaches that neutralize CXCL4, a newly identified target of fibrosis, may represent a novel strategy for preventing and treating CLAD after liver transplantation.
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publisher Wiley
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spelling doaj-art-b51da7c5d66b4ad58dadb46a8a05f6a12025-08-20T02:23:31ZengWileyJournal of Immunology Research2314-88612314-71562016-01-01201610.1155/2016/92769869276986CXCL4 Contributes to the Pathogenesis of Chronic Liver Allograft DysfunctionJing Li0Bin Liu1Yuan Shi2Ke-Liang Xie3Hai-Fang Yin4Lu-nan Yan5Wan-yee Lau6Guo-Lin Wang7Department of Anesthesiology, General Hospital of Tianjin Medical University, Tianjin 300052, ChinaDepartment of Critical Care Medicine, General Hospital of Tianjin Medical University, Tianjin 300052, ChinaOrgan Transplantation Center, Tianjin First Center Hospital, Nankai District, Tianjin 300192, ChinaDepartment of Critical Care Medicine, General Hospital of Tianjin Medical University, Tianjin 300052, ChinaDepartment of Cell Biology and Research Center of Basic Medical Science, Tianjin Medical University, Qixiangtai Road, Heping District, Tianjin 300070, ChinaLiver Transplantation Center, West China Hospital of Sichuan University, Chengdu 610041, Sichuan Province, ChinaFaculty of Medicine, The Chinese University of Hong Kong, Prince of Wales Hospital, Sha Tin, Hong KongDepartment of Anesthesiology, General Hospital of Tianjin Medical University, Tianjin 300052, ChinaChronic liver allograft dysfunction (CLAD) remains the most common cause of patient morbidity and allograft loss in liver transplant patients. However, the pathogenesis of CLAD has not been completely elucidated. By establishing rat CLAD models, in this study, we identified the informative CLAD-associated genes using isobaric tags for relative and absolute quantification (iTRAQ) proteomics analysis and validated these results in recipient rat liver allografts. CXCL4, CXCR3, EGFR, JAK2, STAT3, and Collagen IV were associated with CLAD pathogenesis. We validated that CXCL4 is upstream of these informative genes in the isolated hepatic stellate cells (HSC). Blocking CXCL4 protects against CLAD by reducing liver fibrosis. Therefore, our results indicated that therapeutic approaches that neutralize CXCL4, a newly identified target of fibrosis, may represent a novel strategy for preventing and treating CLAD after liver transplantation.http://dx.doi.org/10.1155/2016/9276986
spellingShingle Jing Li
Bin Liu
Yuan Shi
Ke-Liang Xie
Hai-Fang Yin
Lu-nan Yan
Wan-yee Lau
Guo-Lin Wang
CXCL4 Contributes to the Pathogenesis of Chronic Liver Allograft Dysfunction
Journal of Immunology Research
title CXCL4 Contributes to the Pathogenesis of Chronic Liver Allograft Dysfunction
title_full CXCL4 Contributes to the Pathogenesis of Chronic Liver Allograft Dysfunction
title_fullStr CXCL4 Contributes to the Pathogenesis of Chronic Liver Allograft Dysfunction
title_full_unstemmed CXCL4 Contributes to the Pathogenesis of Chronic Liver Allograft Dysfunction
title_short CXCL4 Contributes to the Pathogenesis of Chronic Liver Allograft Dysfunction
title_sort cxcl4 contributes to the pathogenesis of chronic liver allograft dysfunction
url http://dx.doi.org/10.1155/2016/9276986
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