Inhibition of CYP450 family 1 subfamily B member 1 (CYP1B1) expression in macrophage reduces the inflammatory response in type 2 diabetes mellitus combined with tuberculosis

Type 2 diabetes (T2DM) and tuberculosis (TB) both regulate inflammation and may exert synergistic or antagonistic effects through shared immune pathways. Previous studies have demonstrated that T2DM is a risk factor for TB. However, at the level of gene regulatory networks, it remains unclear whethe...

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Main Authors: Qianqian Du, Kun Liu, Yanling Li, Xinyan Wang, Xin Liu, Jing Zhao, Xuemei Wang
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-08-01
Series:Frontiers in Endocrinology
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Online Access:https://www.frontiersin.org/articles/10.3389/fendo.2025.1617292/full
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author Qianqian Du
Kun Liu
Yanling Li
Xinyan Wang
Xin Liu
Jing Zhao
Xuemei Wang
author_facet Qianqian Du
Kun Liu
Yanling Li
Xinyan Wang
Xin Liu
Jing Zhao
Xuemei Wang
author_sort Qianqian Du
collection DOAJ
description Type 2 diabetes (T2DM) and tuberculosis (TB) both regulate inflammation and may exert synergistic or antagonistic effects through shared immune pathways. Previous studies have demonstrated that T2DM is a risk factor for TB. However, at the level of gene regulatory networks, it remains unclear whether there are key interaction nodes linking these two diseases. In this study, we integrated bioinformatic analysis from the Gene Expression Omnibus (GEO) database and performed differential gene expression analysis and weighted gene co-expression network analysis (WGCNA). Furthermore, we applied machine learning techniques to identify key genes among the commonly differentially expressed genes (DEGs). In addition, this study employed siRNA in THP-1 cells to validate the cross-talk genes selected through bioinformatic analysis. The THP-1 cells were treated with high-concentration glucose (15.5 μM, Glu), Mycobacterium tuberculosis ESAT-6, or Glu+ESAT-6. We identified a total of 23 common genes between TB and T2DM using DEGs and WGCNA. Furthermore, expression patterns from external datasets revealed three key cross-talk genes linking TB-T2DM: CYP1B1, SERPING1, and CHPT1. Notably, only CYP1B1 was significantly upregulated in the THP-1 detection test, compared to the unstimulated (control) group (P < 0.05). Moreover, CYP1B1 significantly reduced the expression of pro-inflammatory cytokines (TNF-α, IL-6, IL-1β, IL-10), M2 macrophage polarization markers (CD163, Arg-1), and chemokines (CXCL-10), and was associated with the NOD2 and TRAF6 signaling pathways (P < 0.05). These findings elucidate the regulatory mechanisms underlying the comorbidity of TB and T2DM, providing a theoretical basis for the development of precise combination therapies and novel therapeutic targets.
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spelling doaj-art-a947f19a432e4e2d932d89859758ea042025-08-21T04:10:31ZengFrontiers Media S.A.Frontiers in Endocrinology1664-23922025-08-011610.3389/fendo.2025.16172921617292Inhibition of CYP450 family 1 subfamily B member 1 (CYP1B1) expression in macrophage reduces the inflammatory response in type 2 diabetes mellitus combined with tuberculosisQianqian DuKun LiuYanling LiXinyan WangXin LiuJing ZhaoXuemei WangType 2 diabetes (T2DM) and tuberculosis (TB) both regulate inflammation and may exert synergistic or antagonistic effects through shared immune pathways. Previous studies have demonstrated that T2DM is a risk factor for TB. However, at the level of gene regulatory networks, it remains unclear whether there are key interaction nodes linking these two diseases. In this study, we integrated bioinformatic analysis from the Gene Expression Omnibus (GEO) database and performed differential gene expression analysis and weighted gene co-expression network analysis (WGCNA). Furthermore, we applied machine learning techniques to identify key genes among the commonly differentially expressed genes (DEGs). In addition, this study employed siRNA in THP-1 cells to validate the cross-talk genes selected through bioinformatic analysis. The THP-1 cells were treated with high-concentration glucose (15.5 μM, Glu), Mycobacterium tuberculosis ESAT-6, or Glu+ESAT-6. We identified a total of 23 common genes between TB and T2DM using DEGs and WGCNA. Furthermore, expression patterns from external datasets revealed three key cross-talk genes linking TB-T2DM: CYP1B1, SERPING1, and CHPT1. Notably, only CYP1B1 was significantly upregulated in the THP-1 detection test, compared to the unstimulated (control) group (P < 0.05). Moreover, CYP1B1 significantly reduced the expression of pro-inflammatory cytokines (TNF-α, IL-6, IL-1β, IL-10), M2 macrophage polarization markers (CD163, Arg-1), and chemokines (CXCL-10), and was associated with the NOD2 and TRAF6 signaling pathways (P < 0.05). These findings elucidate the regulatory mechanisms underlying the comorbidity of TB and T2DM, providing a theoretical basis for the development of precise combination therapies and novel therapeutic targets.https://www.frontiersin.org/articles/10.3389/fendo.2025.1617292/fulltuberculosistype 2 diabetesmacrophagesCYP1B1inflammatory response
spellingShingle Qianqian Du
Kun Liu
Yanling Li
Xinyan Wang
Xin Liu
Jing Zhao
Xuemei Wang
Inhibition of CYP450 family 1 subfamily B member 1 (CYP1B1) expression in macrophage reduces the inflammatory response in type 2 diabetes mellitus combined with tuberculosis
Frontiers in Endocrinology
tuberculosis
type 2 diabetes
macrophages
CYP1B1
inflammatory response
title Inhibition of CYP450 family 1 subfamily B member 1 (CYP1B1) expression in macrophage reduces the inflammatory response in type 2 diabetes mellitus combined with tuberculosis
title_full Inhibition of CYP450 family 1 subfamily B member 1 (CYP1B1) expression in macrophage reduces the inflammatory response in type 2 diabetes mellitus combined with tuberculosis
title_fullStr Inhibition of CYP450 family 1 subfamily B member 1 (CYP1B1) expression in macrophage reduces the inflammatory response in type 2 diabetes mellitus combined with tuberculosis
title_full_unstemmed Inhibition of CYP450 family 1 subfamily B member 1 (CYP1B1) expression in macrophage reduces the inflammatory response in type 2 diabetes mellitus combined with tuberculosis
title_short Inhibition of CYP450 family 1 subfamily B member 1 (CYP1B1) expression in macrophage reduces the inflammatory response in type 2 diabetes mellitus combined with tuberculosis
title_sort inhibition of cyp450 family 1 subfamily b member 1 cyp1b1 expression in macrophage reduces the inflammatory response in type 2 diabetes mellitus combined with tuberculosis
topic tuberculosis
type 2 diabetes
macrophages
CYP1B1
inflammatory response
url https://www.frontiersin.org/articles/10.3389/fendo.2025.1617292/full
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