Tamsulosin-induced priapism: A case report

Background: Priapism is a persistent erection generally lasting more than 4 h. Types of priapism include recurrent, ischemic, and non-ischemic and are generally classified based on penile arterial blood flow. Priapism can be induced by hematologic disorders (i.e. sickle cell anemia), infections, rec...

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Main Authors: Julie Martino, Zane Elfessi, Heather Webster
Format: Article
Language:English
Published: Elsevier 2024-12-01
Series:JEM Reports
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Online Access:http://www.sciencedirect.com/science/article/pii/S2773232024000555
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author Julie Martino
Zane Elfessi
Heather Webster
author_facet Julie Martino
Zane Elfessi
Heather Webster
author_sort Julie Martino
collection DOAJ
description Background: Priapism is a persistent erection generally lasting more than 4 h. Types of priapism include recurrent, ischemic, and non-ischemic and are generally classified based on penile arterial blood flow. Priapism can be induced by hematologic disorders (i.e. sickle cell anemia), infections, recreational drug use, and medication use. Tamsulosin is an α1A receptor antagonist commonly used in the treatment of LUTS and benign prostatic hyperplasia (BPH). Case report: We report a case of priapism lasting 48 hours due to tamsulosin. Aspiration and irrigation of the corpus cavernosa and intracavernosal injection of phenylephrine were performed without success. A penile blood gas revealed a pH of 6.92, pCO2 of 108 mmHg, pO2 of 40 mmHg, and HCO3 of 22.2 mmol/L—indicating ischemic priapism. A T-shunt was performed through the glans with moderate detumescence—the penis was able to bend at least 60° in all directions and 90° ventrally. The patient had a computed tomographic angiography (CTA) of the pelvis and a magnetic resonance angiography (MRA) 1 and 3 months later, respectively, which indicated proper flow and the absence of arteriovenous malformations.Why should an EM Physician be aware of this?Despite its attractive nature for the treatment of LUTS or BPH, because of α1A receptor selectivity, tamsulosin should be recognized as a culprit of priapism. Because of the high affinity for the α1A receptor, patients on tamsulosin may not respond to traditional medical therapies for priapism and therefore may require surgical intervention.
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spelling doaj-art-a41a5d81dcec4e42ad6a12d4edda9e4f2025-08-20T01:54:19ZengElsevierJEM Reports2773-23202024-12-013410012510.1016/j.jemrpt.2024.100125Tamsulosin-induced priapism: A case reportJulie Martino0Zane Elfessi1Heather Webster2Department of Emergency Medicine, Jesse Brown Veterans Affairs Medical Center, 820 S Damen Avenue, Chicago, IL, 60612, USADepartment of Emergency Medicine, Jesse Brown Veterans Affairs Medical Center, 820 S Damen Avenue, Chicago, IL, 60612, USA; University of Illinois-Chicago College of Pharmacy, Department of Pharmacy Practice, 833 S Wood Street, Chicago, IL, 60612, USA; Corresponding author. Department of Emergency Medicine, Jesse Brown Veterans Affairs Medical Center, 820 S Damen Avenue, Chicago, IL, 60612, USA.University of Illinois-Chicago College of Pharmacy, Department of Pharmacy Practice, 833 S Wood Street, Chicago, IL, 60612, USABackground: Priapism is a persistent erection generally lasting more than 4 h. Types of priapism include recurrent, ischemic, and non-ischemic and are generally classified based on penile arterial blood flow. Priapism can be induced by hematologic disorders (i.e. sickle cell anemia), infections, recreational drug use, and medication use. Tamsulosin is an α1A receptor antagonist commonly used in the treatment of LUTS and benign prostatic hyperplasia (BPH). Case report: We report a case of priapism lasting 48 hours due to tamsulosin. Aspiration and irrigation of the corpus cavernosa and intracavernosal injection of phenylephrine were performed without success. A penile blood gas revealed a pH of 6.92, pCO2 of 108 mmHg, pO2 of 40 mmHg, and HCO3 of 22.2 mmol/L—indicating ischemic priapism. A T-shunt was performed through the glans with moderate detumescence—the penis was able to bend at least 60° in all directions and 90° ventrally. The patient had a computed tomographic angiography (CTA) of the pelvis and a magnetic resonance angiography (MRA) 1 and 3 months later, respectively, which indicated proper flow and the absence of arteriovenous malformations.Why should an EM Physician be aware of this?Despite its attractive nature for the treatment of LUTS or BPH, because of α1A receptor selectivity, tamsulosin should be recognized as a culprit of priapism. Because of the high affinity for the α1A receptor, patients on tamsulosin may not respond to traditional medical therapies for priapism and therefore may require surgical intervention.http://www.sciencedirect.com/science/article/pii/S2773232024000555PriapismTamsulosinIschemic
spellingShingle Julie Martino
Zane Elfessi
Heather Webster
Tamsulosin-induced priapism: A case report
JEM Reports
Priapism
Tamsulosin
Ischemic
title Tamsulosin-induced priapism: A case report
title_full Tamsulosin-induced priapism: A case report
title_fullStr Tamsulosin-induced priapism: A case report
title_full_unstemmed Tamsulosin-induced priapism: A case report
title_short Tamsulosin-induced priapism: A case report
title_sort tamsulosin induced priapism a case report
topic Priapism
Tamsulosin
Ischemic
url http://www.sciencedirect.com/science/article/pii/S2773232024000555
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AT zaneelfessi tamsulosininducedpriapismacasereport
AT heatherwebster tamsulosininducedpriapismacasereport