The mTOR pathway is involved in the process of platelet-rich plasma improving intervertebral disc degeneration

Objective(s): Platelet-rich plasma (PRP) contains multiple growth hormones that may stimulate tissue repair. We aimed to assess PRP’s efficacy and explore possible mechanisms using the intervertebral disc degeneration (IDD) model. Materials and Methods: A total of 48 male Sprague-Dawley (SD) rats we...

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Main Authors: Jing Luan, Qi Wang, Wei Zheng, Yongjin He
Format: Article
Language:English
Published: Mashhad University of Medical Sciences 2025-03-01
Series:Iranian Journal of Basic Medical Sciences
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Online Access:https://ijbms.mums.ac.ir/article_25233_b6707a240a7962ae68e134e613620c50.pdf
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author Jing Luan
Qi Wang
Wei Zheng
Yongjin He
author_facet Jing Luan
Qi Wang
Wei Zheng
Yongjin He
author_sort Jing Luan
collection DOAJ
description Objective(s): Platelet-rich plasma (PRP) contains multiple growth hormones that may stimulate tissue repair. We aimed to assess PRP’s efficacy and explore possible mechanisms using the intervertebral disc degeneration (IDD) model. Materials and Methods: A total of 48 male Sprague-Dawley (SD) rats were randomly divided into three groups: sham, IDD+PBS, and IDD+PRP (n=16, respectively). IL-1β (10 ng/ml) was used to establish a humanized IDD model in human lumbar nucleus pulposus (NP) tissues from 36 patients with degenerative disc disease. These NP cells were randomly divided into three groups: sham, IDD+PBS, and IDD+PRP (n=12, respectively). RT-PCR and western blot were used to detect the expression of aggrecan, collagen II, IL-1β, IL-6, TNF-α, Bcl-2, cleaved-Caspase 3, Bax and Akt/mTOR/p70S6K signaling pathway. A related assay kit was used to detect MDA, SOD, and GSH.Results: PRP affected the expression of aggrecan, collagen II, IL-1β, IL-6, TNF-α, MDA, SOD, GSH, Bcl-2, cleaved-Caspase 3, and Bax in IDD rats. Compared with the IDD+PBS group, the expression of p-mTOR, p-p70/S6K, and p-Akt was much lower in the rat IDD+PRP group (P<0.05). Similarly, with PRP treatment in the humanized IDD model, the expression of p-mTOR, p-p70/S6K, and p-Akt was also inhibited.Conclusion: PRP may be a potential therapy for IDD via the mTOR signaling pathway in regulating and affecting extracellular matrix degradation, inflammatory factors, oxidative stress, and apoptosis.
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spelling doaj-art-9b0ba2523ac841cbbbed90e25b4564f52025-08-20T02:48:06ZengMashhad University of Medical SciencesIranian Journal of Basic Medical Sciences2008-38662008-38742025-03-0128339340010.22038/ijbms.2024.79218.1716325233The mTOR pathway is involved in the process of platelet-rich plasma improving intervertebral disc degenerationJing Luan0Qi Wang1Wei Zheng2Yongjin He3Department of Pain, Tianjin First Central Hospital, Tianjin, 300110, ChinaDepartment of Anesthesiology, Tianjin First Central Hospital, Tianjin, 300110, ChinaDepartment of Pain, Tianjin First Central Hospital, Tianjin, 300110, ChinaDepartment of Pain, Tianjin First Central Hospital, Tianjin, 300110, ChinaObjective(s): Platelet-rich plasma (PRP) contains multiple growth hormones that may stimulate tissue repair. We aimed to assess PRP’s efficacy and explore possible mechanisms using the intervertebral disc degeneration (IDD) model. Materials and Methods: A total of 48 male Sprague-Dawley (SD) rats were randomly divided into three groups: sham, IDD+PBS, and IDD+PRP (n=16, respectively). IL-1β (10 ng/ml) was used to establish a humanized IDD model in human lumbar nucleus pulposus (NP) tissues from 36 patients with degenerative disc disease. These NP cells were randomly divided into three groups: sham, IDD+PBS, and IDD+PRP (n=12, respectively). RT-PCR and western blot were used to detect the expression of aggrecan, collagen II, IL-1β, IL-6, TNF-α, Bcl-2, cleaved-Caspase 3, Bax and Akt/mTOR/p70S6K signaling pathway. A related assay kit was used to detect MDA, SOD, and GSH.Results: PRP affected the expression of aggrecan, collagen II, IL-1β, IL-6, TNF-α, MDA, SOD, GSH, Bcl-2, cleaved-Caspase 3, and Bax in IDD rats. Compared with the IDD+PBS group, the expression of p-mTOR, p-p70/S6K, and p-Akt was much lower in the rat IDD+PRP group (P<0.05). Similarly, with PRP treatment in the humanized IDD model, the expression of p-mTOR, p-p70/S6K, and p-Akt was also inhibited.Conclusion: PRP may be a potential therapy for IDD via the mTOR signaling pathway in regulating and affecting extracellular matrix degradation, inflammatory factors, oxidative stress, and apoptosis.https://ijbms.mums.ac.ir/article_25233_b6707a240a7962ae68e134e613620c50.pdfakt/mtor/p70s6k-signaling pathwayapoptosisextracellular matrix- degradationinflammatory factors intervertebral disc degenerationoxidative stressplatelet-rich plasma
spellingShingle Jing Luan
Qi Wang
Wei Zheng
Yongjin He
The mTOR pathway is involved in the process of platelet-rich plasma improving intervertebral disc degeneration
Iranian Journal of Basic Medical Sciences
akt/mtor/p70s6k-signaling pathway
apoptosis
extracellular matrix- degradation
inflammatory factors intervertebral disc degeneration
oxidative stress
platelet-rich plasma
title The mTOR pathway is involved in the process of platelet-rich plasma improving intervertebral disc degeneration
title_full The mTOR pathway is involved in the process of platelet-rich plasma improving intervertebral disc degeneration
title_fullStr The mTOR pathway is involved in the process of platelet-rich plasma improving intervertebral disc degeneration
title_full_unstemmed The mTOR pathway is involved in the process of platelet-rich plasma improving intervertebral disc degeneration
title_short The mTOR pathway is involved in the process of platelet-rich plasma improving intervertebral disc degeneration
title_sort mtor pathway is involved in the process of platelet rich plasma improving intervertebral disc degeneration
topic akt/mtor/p70s6k-signaling pathway
apoptosis
extracellular matrix- degradation
inflammatory factors intervertebral disc degeneration
oxidative stress
platelet-rich plasma
url https://ijbms.mums.ac.ir/article_25233_b6707a240a7962ae68e134e613620c50.pdf
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