Sodium Lactate Accelerates M2 Macrophage Polarization and Improves Cardiac Function after Myocardial Infarction in Mice
Background. After myocardial infarction, anti-inflammatory macrophages perform key homeostatic functions that facilitate cardiac recovery and remodeling. Several studies have shown that lactate may serve as a modifier that influences phenotype of macrophage. However, the therapeutic role of sodium l...
Saved in:
Main Authors: | Jialiang Zhang, Fangyang Huang, Li Chen, Guoyong Li, Wenhua Lei, Jiahao Zhao, Yanbiao Liao, Yijian Li, Changming Li, Mao Chen |
---|---|
Format: | Article |
Language: | English |
Published: |
Wiley
2021-01-01
|
Series: | Cardiovascular Therapeutics |
Online Access: | http://dx.doi.org/10.1155/2021/5530541 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Macrophage Activities in Myocardial Infarction and Heart Failure
by: Sophia Esi Duncan, et al.
Published: (2020-01-01) -
Sodium butyrate regulates macrophage polarization by TGR5/β-arrestin2 in vitro
by: Miao Liu, et al.
Published: (2025-01-01) -
Increased Expression of Visfatin in Monocytes and Macrophages in Male Acute Myocardial Infarction Patients
by: Cheng-An Chiu, et al.
Published: (2012-01-01) -
Regenerative therapies for myocardial infarction: exploring the critical role of energy metabolism in achieving cardiac repair
by: Jiahao Ren, et al.
Published: (2025-02-01) -
Endothelial cells under disturbed flow release extracellular vesicles to promote inflammatory polarization of macrophages and accelerate atherosclerosis
by: Zhe Hou, et al.
Published: (2025-01-01)