The mechanism of ECT1/E6E7 cervical intraepithelial neoplasia cells regulated by Acinetobacter lwoffii through circ-LDHA/HMGB1

Abstract Background To investigate the mechanism by which Acinetobacter lwoffii-induced circ-LDHA/HMGB1 activation regulates the ability of ECT1/E6E7 cells to promote cervical intraepithelial neoplasia (CIN). Methods After establishing a co-culture model of Acinetobacter lwoffii and ECT1/E6E7 cells,...

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Main Authors: Jiayuan Du, Tianmeng Tan, Yaojie Yu, He Wang
Format: Article
Language:English
Published: BMC 2025-05-01
Series:BMC Microbiology
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Online Access:https://doi.org/10.1186/s12866-025-04043-y
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author Jiayuan Du
Tianmeng Tan
Yaojie Yu
He Wang
author_facet Jiayuan Du
Tianmeng Tan
Yaojie Yu
He Wang
author_sort Jiayuan Du
collection DOAJ
description Abstract Background To investigate the mechanism by which Acinetobacter lwoffii-induced circ-LDHA/HMGB1 activation regulates the ability of ECT1/E6E7 cells to promote cervical intraepithelial neoplasia (CIN). Methods After establishing a co-culture model of Acinetobacter lwoffii and ECT1/E6E7 cells, the proliferation, migration and apoptosis of ECT1/E6E7 cells were detected by CCK8, transwell and flow cytometry assays. The relative expression levels of circ-LDHA and HMGB1 were detected by real-time quantitative PCR (qRT-PCR). Results Circ-LDHA expression was lower in normal cervical tissues and greater in the cervical cancer group and CIN group than in the normal group (P < 0.05). The optimal coculture model with Acinetobacter lwoffii was an MOI of 100 following 18 h-24 h of coculture. The proliferative activity of the ECT1/E6E7 cells and the relative mRNA expression levels of circ-LDHA and HMGB1 were significantly increased within 36h of coculture, and the apoptosis rate of the ECT1/E6E7 cells was significantly increased after 12 h of coculture. The migration rate of the ECT1/E6E7 cells increased from 12 h to 24 h (all P < 0.05). Conclusion Acinetobacter lwoffii can regulate the development and progression of CIN by regulating the expression of circ-LDHA and HMGB1 in a time-dependent manner.
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spelling doaj-art-94e4a30f8ddb4d9fba463f4384e6fa1e2025-08-20T03:16:50ZengBMCBMC Microbiology1471-21802025-05-0125111210.1186/s12866-025-04043-yThe mechanism of ECT1/E6E7 cervical intraepithelial neoplasia cells regulated by Acinetobacter lwoffii through circ-LDHA/HMGB1Jiayuan Du0Tianmeng Tan1Yaojie Yu2He Wang3Department of Gynecology Oncology, Guangxi Medical University Cancer HospitalDepartment of Gynecology Oncology, Guangxi Medical University Cancer HospitalDepartment of Gynecology Oncology, Guangxi Medical University Cancer HospitalDepartment of Gynecology Oncology, Guangxi Medical University Cancer HospitalAbstract Background To investigate the mechanism by which Acinetobacter lwoffii-induced circ-LDHA/HMGB1 activation regulates the ability of ECT1/E6E7 cells to promote cervical intraepithelial neoplasia (CIN). Methods After establishing a co-culture model of Acinetobacter lwoffii and ECT1/E6E7 cells, the proliferation, migration and apoptosis of ECT1/E6E7 cells were detected by CCK8, transwell and flow cytometry assays. The relative expression levels of circ-LDHA and HMGB1 were detected by real-time quantitative PCR (qRT-PCR). Results Circ-LDHA expression was lower in normal cervical tissues and greater in the cervical cancer group and CIN group than in the normal group (P < 0.05). The optimal coculture model with Acinetobacter lwoffii was an MOI of 100 following 18 h-24 h of coculture. The proliferative activity of the ECT1/E6E7 cells and the relative mRNA expression levels of circ-LDHA and HMGB1 were significantly increased within 36h of coculture, and the apoptosis rate of the ECT1/E6E7 cells was significantly increased after 12 h of coculture. The migration rate of the ECT1/E6E7 cells increased from 12 h to 24 h (all P < 0.05). Conclusion Acinetobacter lwoffii can regulate the development and progression of CIN by regulating the expression of circ-LDHA and HMGB1 in a time-dependent manner.https://doi.org/10.1186/s12866-025-04043-yAcinetobacter lwoffiiCervical intraepithelial neoplasiaCocultureCirc-LDHAHMGB1
spellingShingle Jiayuan Du
Tianmeng Tan
Yaojie Yu
He Wang
The mechanism of ECT1/E6E7 cervical intraepithelial neoplasia cells regulated by Acinetobacter lwoffii through circ-LDHA/HMGB1
BMC Microbiology
Acinetobacter lwoffii
Cervical intraepithelial neoplasia
Coculture
Circ-LDHA
HMGB1
title The mechanism of ECT1/E6E7 cervical intraepithelial neoplasia cells regulated by Acinetobacter lwoffii through circ-LDHA/HMGB1
title_full The mechanism of ECT1/E6E7 cervical intraepithelial neoplasia cells regulated by Acinetobacter lwoffii through circ-LDHA/HMGB1
title_fullStr The mechanism of ECT1/E6E7 cervical intraepithelial neoplasia cells regulated by Acinetobacter lwoffii through circ-LDHA/HMGB1
title_full_unstemmed The mechanism of ECT1/E6E7 cervical intraepithelial neoplasia cells regulated by Acinetobacter lwoffii through circ-LDHA/HMGB1
title_short The mechanism of ECT1/E6E7 cervical intraepithelial neoplasia cells regulated by Acinetobacter lwoffii through circ-LDHA/HMGB1
title_sort mechanism of ect1 e6e7 cervical intraepithelial neoplasia cells regulated by acinetobacter lwoffii through circ ldha hmgb1
topic Acinetobacter lwoffii
Cervical intraepithelial neoplasia
Coculture
Circ-LDHA
HMGB1
url https://doi.org/10.1186/s12866-025-04043-y
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