Treatment Combining Focused Ultrasound with Gastrodin Alleviates Memory Deficit and Neuropathology in an Alzheimer’s Disease-Like Experimental Mouse Model

Alzheimer’s disease (AD) is the most common type of dementia but lacks effective treatment at present. Gastrodin (GAS) is a phenolic glycoside extracted from the traditional Chinese herb—Gastrodia elata—and has been reported as a potential therapeutic agent for AD. However, its efficiency is reduced...

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Main Authors: Kaixuan Luo, Yuhong Wang, Wen-Shiang Chen, Xiangjun Feng, Yehui Liao, Shaochun Chen, Yao Liu, Chengde Liao, Moxian Chen, Lijuan Ao
Format: Article
Language:English
Published: Wiley 2022-01-01
Series:Neural Plasticity
Online Access:http://dx.doi.org/10.1155/2022/5241449
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author Kaixuan Luo
Yuhong Wang
Wen-Shiang Chen
Xiangjun Feng
Yehui Liao
Shaochun Chen
Yao Liu
Chengde Liao
Moxian Chen
Lijuan Ao
author_facet Kaixuan Luo
Yuhong Wang
Wen-Shiang Chen
Xiangjun Feng
Yehui Liao
Shaochun Chen
Yao Liu
Chengde Liao
Moxian Chen
Lijuan Ao
author_sort Kaixuan Luo
collection DOAJ
description Alzheimer’s disease (AD) is the most common type of dementia but lacks effective treatment at present. Gastrodin (GAS) is a phenolic glycoside extracted from the traditional Chinese herb—Gastrodia elata—and has been reported as a potential therapeutic agent for AD. However, its efficiency is reduced for AD patients due to its limited BBB permeability. Studies have demonstrated the feasibility of opening the blood-brain barrier (BBB) via focused ultrasound (FUS) to overcome the obstacles preventing medicines from blood flow into the brain tissue. We explored the therapeutic potential of FUS-mediated BBB opening combined with GAS in an AD-like mouse model induced by unilateral intracerebroventricular (ICV) injection of Aβ1-42. Mice were divided into 5 groups: control, untreated, GAS, FUS and FUS+GAS. Combined treatment (FUS+GAS) rather than single intervention (GAS or FUS) alleviated memory deficit and neuropathology of AD-like mice. The time that mice spent in the novel arm was prolonged in the Y-maze test after 15-day intervention, and the waste-cleaning effect was remarkably increased. Contents of Aβ, tau, and P-tau in the observed (also the targeted) hippocampus were reduced. BDNF, synaptophysin (SYN), and PSD-95 were upregulated in the combined group. Overall, our results demonstrate that FUS-mediated BBB opening combined with GAS injection exerts the potential to alleviate memory deficit and neuropathology in the AD-like experimental mouse model, which may be a novel strategy for AD treatment.
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spelling doaj-art-7d5e555280f34f49b533a0c543e8ce882025-08-20T02:07:12ZengWileyNeural Plasticity1687-54432022-01-01202210.1155/2022/5241449Treatment Combining Focused Ultrasound with Gastrodin Alleviates Memory Deficit and Neuropathology in an Alzheimer’s Disease-Like Experimental Mouse ModelKaixuan Luo0Yuhong Wang1Wen-Shiang Chen2Xiangjun Feng3Yehui Liao4Shaochun Chen5Yao Liu6Chengde Liao7Moxian Chen8Lijuan Ao9School of RehabilitationSchool of RehabilitationDepartment of Physical Medicine and RehabilitationSchool of RehabilitationSchool of RehabilitationSchool of RehabilitationSchool of RehabilitationDepartment of RadiologySchool of RehabilitationSchool of RehabilitationAlzheimer’s disease (AD) is the most common type of dementia but lacks effective treatment at present. Gastrodin (GAS) is a phenolic glycoside extracted from the traditional Chinese herb—Gastrodia elata—and has been reported as a potential therapeutic agent for AD. However, its efficiency is reduced for AD patients due to its limited BBB permeability. Studies have demonstrated the feasibility of opening the blood-brain barrier (BBB) via focused ultrasound (FUS) to overcome the obstacles preventing medicines from blood flow into the brain tissue. We explored the therapeutic potential of FUS-mediated BBB opening combined with GAS in an AD-like mouse model induced by unilateral intracerebroventricular (ICV) injection of Aβ1-42. Mice were divided into 5 groups: control, untreated, GAS, FUS and FUS+GAS. Combined treatment (FUS+GAS) rather than single intervention (GAS or FUS) alleviated memory deficit and neuropathology of AD-like mice. The time that mice spent in the novel arm was prolonged in the Y-maze test after 15-day intervention, and the waste-cleaning effect was remarkably increased. Contents of Aβ, tau, and P-tau in the observed (also the targeted) hippocampus were reduced. BDNF, synaptophysin (SYN), and PSD-95 were upregulated in the combined group. Overall, our results demonstrate that FUS-mediated BBB opening combined with GAS injection exerts the potential to alleviate memory deficit and neuropathology in the AD-like experimental mouse model, which may be a novel strategy for AD treatment.http://dx.doi.org/10.1155/2022/5241449
spellingShingle Kaixuan Luo
Yuhong Wang
Wen-Shiang Chen
Xiangjun Feng
Yehui Liao
Shaochun Chen
Yao Liu
Chengde Liao
Moxian Chen
Lijuan Ao
Treatment Combining Focused Ultrasound with Gastrodin Alleviates Memory Deficit and Neuropathology in an Alzheimer’s Disease-Like Experimental Mouse Model
Neural Plasticity
title Treatment Combining Focused Ultrasound with Gastrodin Alleviates Memory Deficit and Neuropathology in an Alzheimer’s Disease-Like Experimental Mouse Model
title_full Treatment Combining Focused Ultrasound with Gastrodin Alleviates Memory Deficit and Neuropathology in an Alzheimer’s Disease-Like Experimental Mouse Model
title_fullStr Treatment Combining Focused Ultrasound with Gastrodin Alleviates Memory Deficit and Neuropathology in an Alzheimer’s Disease-Like Experimental Mouse Model
title_full_unstemmed Treatment Combining Focused Ultrasound with Gastrodin Alleviates Memory Deficit and Neuropathology in an Alzheimer’s Disease-Like Experimental Mouse Model
title_short Treatment Combining Focused Ultrasound with Gastrodin Alleviates Memory Deficit and Neuropathology in an Alzheimer’s Disease-Like Experimental Mouse Model
title_sort treatment combining focused ultrasound with gastrodin alleviates memory deficit and neuropathology in an alzheimer s disease like experimental mouse model
url http://dx.doi.org/10.1155/2022/5241449
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