Reduced miR-338-3p contributes to polycystic ovarian syndrome by inhibiting proliferation and enhancing apoptosis

Abstract Background Polycystic ovarian syndrome (PCOS) is a frequently occurring disorder affecting reproductive and metabolic health. miR-338-3p is implicated in early follicular development. We aimed to investigate the expression of miR-338-3p in PCOS patients and its effects on proliferation and...

Full description

Saved in:
Bibliographic Details
Main Authors: Lin Liang, Jie Lv, Wei Li, Chengwen Song, Ying Chen, Huafang Wei
Format: Article
Language:English
Published: BMC 2025-07-01
Series:Hereditas
Subjects:
Online Access:https://doi.org/10.1186/s41065-025-00498-1
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849332782197637120
author Lin Liang
Jie Lv
Wei Li
Chengwen Song
Ying Chen
Huafang Wei
author_facet Lin Liang
Jie Lv
Wei Li
Chengwen Song
Ying Chen
Huafang Wei
author_sort Lin Liang
collection DOAJ
description Abstract Background Polycystic ovarian syndrome (PCOS) is a frequently occurring disorder affecting reproductive and metabolic health. miR-338-3p is implicated in early follicular development. We aimed to investigate the expression of miR-338-3p in PCOS patients and its effects on proliferation and apoptosis of ovarian granulosa cells. Methods The study included 100 healthy women and 110 women diagnosed with PCOS as participants. Reverse transcription quantitative PCR (RT-qPCR) was used to detect the expression of miR-338-3p and phosphatase and tensin homolog (PTEN), and receiver operating characteristic (ROC) was employed to evaluate the diagnostic efficacy of miR-338-3p. Cell proliferation and apoptosis were detected by Cell Counting Kit-8 (CCK-8) and flow cytometry. Pearson correlation analysis was used to assess the correlations between miR-338-3p and luteinizing hormone (LH), testosterone, or PTEN. The target relationship of miR-338-3p and PTEN was confirmed via dual-luciferase assay. Results Serum miR-338-3p was decreased in PCOS patients, and it was negatively correlated with both LH and testosterone. Downregulation of miR-338-3p inhibits the proliferation of ovarian granulosa cells and enhances cell apoptosis, whereas upregulation produces the opposite effect. PTEN inhibition subverted the inhibited proliferation and enhanced apoptosis regulated by miR-338-3p inhibitor. Conclusions Reduced miR-338-3p levels have potential predictive value in distinguishing individuals with PCOS patients from normal population. Mechanistically, this microRNA regulates the PTEN gene to inhibit granulosa cell proliferation and promote apoptosis, thereby contributing to the pathological processes of PCOS.
format Article
id doaj-art-7a3a77893c5c4279b5481379589f9d87
institution Kabale University
issn 1601-5223
language English
publishDate 2025-07-01
publisher BMC
record_format Article
series Hereditas
spelling doaj-art-7a3a77893c5c4279b5481379589f9d872025-08-20T03:46:07ZengBMCHereditas1601-52232025-07-0116211910.1186/s41065-025-00498-1Reduced miR-338-3p contributes to polycystic ovarian syndrome by inhibiting proliferation and enhancing apoptosisLin Liang0Jie Lv1Wei Li2Chengwen Song3Ying Chen4Huafang Wei5Department of Gynecology, General Hospital of Central Theater Command of Chinese People’s Liberation ArmyDepartment of Gynecology, General Hospital of Central Theater Command of Chinese People’s Liberation ArmyDepartment of Gynecology, General Hospital of Central Theater Command of Chinese People’s Liberation ArmyDepartment of Gynecology, General Hospital of Central Theater Command of Chinese People’s Liberation ArmyDepartment of Gynecology, General Hospital of Central Theater Command of Chinese People’s Liberation ArmyDepartment of Gynecology, General Hospital of Central Theater Command of Chinese People’s Liberation ArmyAbstract Background Polycystic ovarian syndrome (PCOS) is a frequently occurring disorder affecting reproductive and metabolic health. miR-338-3p is implicated in early follicular development. We aimed to investigate the expression of miR-338-3p in PCOS patients and its effects on proliferation and apoptosis of ovarian granulosa cells. Methods The study included 100 healthy women and 110 women diagnosed with PCOS as participants. Reverse transcription quantitative PCR (RT-qPCR) was used to detect the expression of miR-338-3p and phosphatase and tensin homolog (PTEN), and receiver operating characteristic (ROC) was employed to evaluate the diagnostic efficacy of miR-338-3p. Cell proliferation and apoptosis were detected by Cell Counting Kit-8 (CCK-8) and flow cytometry. Pearson correlation analysis was used to assess the correlations between miR-338-3p and luteinizing hormone (LH), testosterone, or PTEN. The target relationship of miR-338-3p and PTEN was confirmed via dual-luciferase assay. Results Serum miR-338-3p was decreased in PCOS patients, and it was negatively correlated with both LH and testosterone. Downregulation of miR-338-3p inhibits the proliferation of ovarian granulosa cells and enhances cell apoptosis, whereas upregulation produces the opposite effect. PTEN inhibition subverted the inhibited proliferation and enhanced apoptosis regulated by miR-338-3p inhibitor. Conclusions Reduced miR-338-3p levels have potential predictive value in distinguishing individuals with PCOS patients from normal population. Mechanistically, this microRNA regulates the PTEN gene to inhibit granulosa cell proliferation and promote apoptosis, thereby contributing to the pathological processes of PCOS.https://doi.org/10.1186/s41065-025-00498-1Polycystic ovarian syndromemiR-338-3pApoptosisProliferation
spellingShingle Lin Liang
Jie Lv
Wei Li
Chengwen Song
Ying Chen
Huafang Wei
Reduced miR-338-3p contributes to polycystic ovarian syndrome by inhibiting proliferation and enhancing apoptosis
Hereditas
Polycystic ovarian syndrome
miR-338-3p
Apoptosis
Proliferation
title Reduced miR-338-3p contributes to polycystic ovarian syndrome by inhibiting proliferation and enhancing apoptosis
title_full Reduced miR-338-3p contributes to polycystic ovarian syndrome by inhibiting proliferation and enhancing apoptosis
title_fullStr Reduced miR-338-3p contributes to polycystic ovarian syndrome by inhibiting proliferation and enhancing apoptosis
title_full_unstemmed Reduced miR-338-3p contributes to polycystic ovarian syndrome by inhibiting proliferation and enhancing apoptosis
title_short Reduced miR-338-3p contributes to polycystic ovarian syndrome by inhibiting proliferation and enhancing apoptosis
title_sort reduced mir 338 3p contributes to polycystic ovarian syndrome by inhibiting proliferation and enhancing apoptosis
topic Polycystic ovarian syndrome
miR-338-3p
Apoptosis
Proliferation
url https://doi.org/10.1186/s41065-025-00498-1
work_keys_str_mv AT linliang reducedmir3383pcontributestopolycysticovariansyndromebyinhibitingproliferationandenhancingapoptosis
AT jielv reducedmir3383pcontributestopolycysticovariansyndromebyinhibitingproliferationandenhancingapoptosis
AT weili reducedmir3383pcontributestopolycysticovariansyndromebyinhibitingproliferationandenhancingapoptosis
AT chengwensong reducedmir3383pcontributestopolycysticovariansyndromebyinhibitingproliferationandenhancingapoptosis
AT yingchen reducedmir3383pcontributestopolycysticovariansyndromebyinhibitingproliferationandenhancingapoptosis
AT huafangwei reducedmir3383pcontributestopolycysticovariansyndromebyinhibitingproliferationandenhancingapoptosis