Endocrine Dysfunction in Traumatic Subarachnoid Hemorrhage: A Prospective Study

Background This study has prospectively investigated pituitary function and their correlation with severity, pressure effect, and Glasgow outcome scale in the acute phase of traumatic subarachnoid hemorrhage (SAH). Most of the retrospective studies have demonstrated that traumatic SAH-mediated hypop...

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Main Authors: Pankaj Gupta, Radheyshyam S. Mittal, Achal Sharma, Varsha Kumar
Format: Article
Language:English
Published: Thieme Medical and Scientific Publishers Pvt. Ltd. 2021-01-01
Series:Indian Journal of Neurosurgery
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Online Access:http://www.thieme-connect.de/DOI/DOI?10.1055/s-0041-1722835
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author Pankaj Gupta
Radheyshyam S. Mittal
Achal Sharma
Varsha Kumar
author_facet Pankaj Gupta
Radheyshyam S. Mittal
Achal Sharma
Varsha Kumar
author_sort Pankaj Gupta
collection DOAJ
description Background This study has prospectively investigated pituitary function and their correlation with severity, pressure effect, and Glasgow outcome scale in the acute phase of traumatic subarachnoid hemorrhage (SAH). Most of the retrospective studies have demonstrated that traumatic SAH-mediated hypopituitarism could be more frequent than previously known. Objectives The aim of the study is to find the prevalence of endocrine dysfunction in traumatic SAH and its correlation with severity of injury and final outcome. Materials and Methods Eighty-four consecutive patients of traumatic SAH formed the study group. Apart from clinical assessment, noncontrast computed tomography of the head was performed on all patients on admission. The hormonal analysis (FT3, FT4, thyroid-stimulating hormone, growth hormone [GH], cortisol, prolactin, testosterone) was performed within 24 hours of traumatic brain injury and was repeated on the seventh day amongst the patients who survived. Results Most common hormone to increase on day one was cortisol (48.78%), while on day seven follicle-stimulating hormones and cortisol (15.38%) showed increment in levels. Most common hormone to decrease on day one was FT3 (36.84%) and GH (36.26%), while on day seven testosterone (66.67%) and FT4 (30.76%) showed decreasing levels. Hormone most resistant to change was prolactin. Conclusion Hormonal dysfunction is common in moderate to severe traumatic brain injury. There is a direct association between radiological grading (Fischer) of SAH and hormonal profile changes. Performance of hormonal analysis should be considered in patients with moderate to severe traumatic brain injury, preferably with high-grade SAH, so that appropriate hormonal replacement can be done to optimize the clinical outcome.
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spelling doaj-art-77464083517c4f1fb0407bdaf9c0d60d2025-08-20T01:57:55ZengThieme Medical and Scientific Publishers Pvt. Ltd.Indian Journal of Neurosurgery2277-954X2277-91672021-01-01100322022910.1055/s-0041-1722835Endocrine Dysfunction in Traumatic Subarachnoid Hemorrhage: A Prospective StudyPankaj Gupta0Radheyshyam S. Mittal1Achal Sharma2Varsha Kumar3Department of Neurosurgery, Sawai Man Singh Medical College, Jaipur, Rajasthan, IndiaDepartment of Neurosurgery, Sawai Man Singh Medical College, Jaipur, Rajasthan, IndiaDepartment of Neurosurgery, Sawai Man Singh Medical College, Jaipur, Rajasthan, IndiaDepartment of Pathology, Sawai Man Singh Medical College, Jaipur, Rajasthan, IndiaBackground This study has prospectively investigated pituitary function and their correlation with severity, pressure effect, and Glasgow outcome scale in the acute phase of traumatic subarachnoid hemorrhage (SAH). Most of the retrospective studies have demonstrated that traumatic SAH-mediated hypopituitarism could be more frequent than previously known. Objectives The aim of the study is to find the prevalence of endocrine dysfunction in traumatic SAH and its correlation with severity of injury and final outcome. Materials and Methods Eighty-four consecutive patients of traumatic SAH formed the study group. Apart from clinical assessment, noncontrast computed tomography of the head was performed on all patients on admission. The hormonal analysis (FT3, FT4, thyroid-stimulating hormone, growth hormone [GH], cortisol, prolactin, testosterone) was performed within 24 hours of traumatic brain injury and was repeated on the seventh day amongst the patients who survived. Results Most common hormone to increase on day one was cortisol (48.78%), while on day seven follicle-stimulating hormones and cortisol (15.38%) showed increment in levels. Most common hormone to decrease on day one was FT3 (36.84%) and GH (36.26%), while on day seven testosterone (66.67%) and FT4 (30.76%) showed decreasing levels. Hormone most resistant to change was prolactin. Conclusion Hormonal dysfunction is common in moderate to severe traumatic brain injury. There is a direct association between radiological grading (Fischer) of SAH and hormonal profile changes. Performance of hormonal analysis should be considered in patients with moderate to severe traumatic brain injury, preferably with high-grade SAH, so that appropriate hormonal replacement can be done to optimize the clinical outcome.http://www.thieme-connect.de/DOI/DOI?10.1055/s-0041-1722835pituitarysubarachnoid hemorrhageendocrine dysfunction
spellingShingle Pankaj Gupta
Radheyshyam S. Mittal
Achal Sharma
Varsha Kumar
Endocrine Dysfunction in Traumatic Subarachnoid Hemorrhage: A Prospective Study
Indian Journal of Neurosurgery
pituitary
subarachnoid hemorrhage
endocrine dysfunction
title Endocrine Dysfunction in Traumatic Subarachnoid Hemorrhage: A Prospective Study
title_full Endocrine Dysfunction in Traumatic Subarachnoid Hemorrhage: A Prospective Study
title_fullStr Endocrine Dysfunction in Traumatic Subarachnoid Hemorrhage: A Prospective Study
title_full_unstemmed Endocrine Dysfunction in Traumatic Subarachnoid Hemorrhage: A Prospective Study
title_short Endocrine Dysfunction in Traumatic Subarachnoid Hemorrhage: A Prospective Study
title_sort endocrine dysfunction in traumatic subarachnoid hemorrhage a prospective study
topic pituitary
subarachnoid hemorrhage
endocrine dysfunction
url http://www.thieme-connect.de/DOI/DOI?10.1055/s-0041-1722835
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AT achalsharma endocrinedysfunctionintraumaticsubarachnoidhemorrhageaprospectivestudy
AT varshakumar endocrinedysfunctionintraumaticsubarachnoidhemorrhageaprospectivestudy