Decreased Anti-Inflammatory Responses to Vitamin D in Neonatal Neutrophils

Neutrophil activity is prolonged in newborns, suggesting decreased exposure and/or responses to immunosuppressive modulators, such as 1,25-hydroxyvitamin D3 (1,25-vit D3). We hypothesized that 1,25-vit D3 suppresses neutrophil activation and that this response is impaired in newborns. Consistent wit...

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Main Authors: Daniel Hirsch, Faith E. Archer, Meera Joshi-Kale, Anna M. Vetrano, Barry Weinberger
Format: Article
Language:English
Published: Wiley 2011-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2011/598345
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author Daniel Hirsch
Faith E. Archer
Meera Joshi-Kale
Anna M. Vetrano
Barry Weinberger
author_facet Daniel Hirsch
Faith E. Archer
Meera Joshi-Kale
Anna M. Vetrano
Barry Weinberger
author_sort Daniel Hirsch
collection DOAJ
description Neutrophil activity is prolonged in newborns, suggesting decreased exposure and/or responses to immunosuppressive modulators, such as 1,25-hydroxyvitamin D3 (1,25-vit D3). We hypothesized that 1,25-vit D3 suppresses neutrophil activation and that this response is impaired in newborns. Consistent with this, 1,25-vit D3 decreased LPS-induced expression of macrophage inflammatory protein-1β and VEGF in adult, but not neonatal, neutrophils. Expression of vitamin D receptor (VDR) and 25-hydroxyvitamin D3-1α-hydroxylase was reduced in neonatal, relative to adult neutrophils. Moreover, 1,25-vit D3 induced VDR gene expression in activated adult, but not neonatal, neutrophils. 1,25-vit D3 also suppressed expression of cyclooxygenase-2 and induced expression of 5-lipoxygenase in LPS-exposed adult neutrophils, while neonatal cells were not affected. 1,25-vit D3 had no effect on respiratory burst in either adult or neonatal cells. Anti-inflammatory activity of vitamin D is impaired in neonatal neutrophils, and this may be due to decreased expression of VDR and 1α-hydroxylase. Insensitivity to 1,25-vit D3 may contribute to chronic inflammation in neonates.
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spelling doaj-art-673b51e6c3684a719f7fda535aeb29342025-08-20T02:23:53ZengWileyMediators of Inflammation0962-93511466-18612011-01-01201110.1155/2011/598345598345Decreased Anti-Inflammatory Responses to Vitamin D in Neonatal NeutrophilsDaniel Hirsch0Faith E. Archer1Meera Joshi-Kale2Anna M. Vetrano3Barry Weinberger4Division of Neonatology, Department of Pediatrics, Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, New Brunswick, NJ 08901, USADivision of Neonatology, Department of Pediatrics, Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, New Brunswick, NJ 08901, USADivision of Neonatology, Department of Pediatrics, Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, New Brunswick, NJ 08901, USADivision of Neonatology, Department of Pediatrics, Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, New Brunswick, NJ 08901, USADivision of Neonatology, Department of Pediatrics, Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, New Brunswick, NJ 08901, USANeutrophil activity is prolonged in newborns, suggesting decreased exposure and/or responses to immunosuppressive modulators, such as 1,25-hydroxyvitamin D3 (1,25-vit D3). We hypothesized that 1,25-vit D3 suppresses neutrophil activation and that this response is impaired in newborns. Consistent with this, 1,25-vit D3 decreased LPS-induced expression of macrophage inflammatory protein-1β and VEGF in adult, but not neonatal, neutrophils. Expression of vitamin D receptor (VDR) and 25-hydroxyvitamin D3-1α-hydroxylase was reduced in neonatal, relative to adult neutrophils. Moreover, 1,25-vit D3 induced VDR gene expression in activated adult, but not neonatal, neutrophils. 1,25-vit D3 also suppressed expression of cyclooxygenase-2 and induced expression of 5-lipoxygenase in LPS-exposed adult neutrophils, while neonatal cells were not affected. 1,25-vit D3 had no effect on respiratory burst in either adult or neonatal cells. Anti-inflammatory activity of vitamin D is impaired in neonatal neutrophils, and this may be due to decreased expression of VDR and 1α-hydroxylase. Insensitivity to 1,25-vit D3 may contribute to chronic inflammation in neonates.http://dx.doi.org/10.1155/2011/598345
spellingShingle Daniel Hirsch
Faith E. Archer
Meera Joshi-Kale
Anna M. Vetrano
Barry Weinberger
Decreased Anti-Inflammatory Responses to Vitamin D in Neonatal Neutrophils
Mediators of Inflammation
title Decreased Anti-Inflammatory Responses to Vitamin D in Neonatal Neutrophils
title_full Decreased Anti-Inflammatory Responses to Vitamin D in Neonatal Neutrophils
title_fullStr Decreased Anti-Inflammatory Responses to Vitamin D in Neonatal Neutrophils
title_full_unstemmed Decreased Anti-Inflammatory Responses to Vitamin D in Neonatal Neutrophils
title_short Decreased Anti-Inflammatory Responses to Vitamin D in Neonatal Neutrophils
title_sort decreased anti inflammatory responses to vitamin d in neonatal neutrophils
url http://dx.doi.org/10.1155/2011/598345
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AT meerajoshikale decreasedantiinflammatoryresponsestovitamindinneonatalneutrophils
AT annamvetrano decreasedantiinflammatoryresponsestovitamindinneonatalneutrophils
AT barryweinberger decreasedantiinflammatoryresponsestovitamindinneonatalneutrophils