Omega-3 Fatty Acids: Possible Neuroprotective Mechanisms in the Model of Global Ischemia in Rats

Background. Omega-3 (ω3) administration was shown to protect against hypoxic-ischemic injury. The objectives were to study the neuroprotective effects of ω3, in a model of global ischemia. Methods. Male Wistar rats were subjected to carotid occlusion (30 min), followed by reperfusion. The groups wer...

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Main Authors: Maria Elizabeth Pereira Nobre, Alyne Oliveira Correia, Francisco Nilson Maciel Mendonça, Luiz Ricardo Araújo Uchoa, Jessica Tamara Nunes Vasconcelos, Carlos Ney Alencar de Araújo, Gerly Anne de Castro Brito, Rafaelly Maria Pinheiro Siqueira, Gilberto dos Santos Cerqueira, Kelly Rose Tavares Neves, Ricardo Mário Arida, Glauce Socorro de Barros Viana
Format: Article
Language:English
Published: Wiley 2016-01-01
Series:Journal of Nutrition and Metabolism
Online Access:http://dx.doi.org/10.1155/2016/6462120
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author Maria Elizabeth Pereira Nobre
Alyne Oliveira Correia
Francisco Nilson Maciel Mendonça
Luiz Ricardo Araújo Uchoa
Jessica Tamara Nunes Vasconcelos
Carlos Ney Alencar de Araújo
Gerly Anne de Castro Brito
Rafaelly Maria Pinheiro Siqueira
Gilberto dos Santos Cerqueira
Kelly Rose Tavares Neves
Ricardo Mário Arida
Glauce Socorro de Barros Viana
author_facet Maria Elizabeth Pereira Nobre
Alyne Oliveira Correia
Francisco Nilson Maciel Mendonça
Luiz Ricardo Araújo Uchoa
Jessica Tamara Nunes Vasconcelos
Carlos Ney Alencar de Araújo
Gerly Anne de Castro Brito
Rafaelly Maria Pinheiro Siqueira
Gilberto dos Santos Cerqueira
Kelly Rose Tavares Neves
Ricardo Mário Arida
Glauce Socorro de Barros Viana
author_sort Maria Elizabeth Pereira Nobre
collection DOAJ
description Background. Omega-3 (ω3) administration was shown to protect against hypoxic-ischemic injury. The objectives were to study the neuroprotective effects of ω3, in a model of global ischemia. Methods. Male Wistar rats were subjected to carotid occlusion (30 min), followed by reperfusion. The groups were SO, untreated ischemic and ischemic treated rats with ω3 (5 and 10 mg/kg, 7 days). The SO and untreated ischemic animals were orally treated with 1% cremophor and, 1 h after the last administration, they were behaviorally tested and euthanized for neurochemical (DA, DOPAC, and NE determinations), histological (Fluoro jade staining), and immunohistochemical (TNF-alpha, COX-2 and iNOS) evaluations. The data were analyzed by ANOVA and Newman-Keuls as the post hoc test. Results. Ischemia increased the locomotor activity and rearing behavior that were partly reversed by ω3. Ischemia decreased striatal DA and DOPAC contents and increased NE contents, effects reversed by ω3. This drug protected hippocampal neuron degeneration, as observed by Fluoro-Jade staining, and the increased immunostainings for TNF-alpha, COX-2, and iNOS were partly or totally blocked by ω3. Conclusion. This study showed a neuroprotective effect of ω3, in great part due to its anti-inflammatory properties, stimulating translational studies focusing on its use in clinic for stroke managing.
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spelling doaj-art-64598935c3ac4d2a9835d5bb015dfcf52025-08-20T03:34:24ZengWileyJournal of Nutrition and Metabolism2090-07242090-07322016-01-01201610.1155/2016/64621206462120Omega-3 Fatty Acids: Possible Neuroprotective Mechanisms in the Model of Global Ischemia in RatsMaria Elizabeth Pereira Nobre0Alyne Oliveira Correia1Francisco Nilson Maciel Mendonça2Luiz Ricardo Araújo Uchoa3Jessica Tamara Nunes Vasconcelos4Carlos Ney Alencar de Araújo5Gerly Anne de Castro Brito6Rafaelly Maria Pinheiro Siqueira7Gilberto dos Santos Cerqueira8Kelly Rose Tavares Neves9Ricardo Mário Arida10Glauce Socorro de Barros Viana11Faculty of Medicine, Estácio of Juazeiro do Norte (FMJ), Rua Tenente Raimundo Rocha 515, 63040-360 Juazeiro do Norte, CE, BrazilFaculty of Medicine, Estácio of Juazeiro do Norte (FMJ), Rua Tenente Raimundo Rocha 515, 63040-360 Juazeiro do Norte, CE, BrazilFaculty of Medicine, Estácio of Juazeiro do Norte (FMJ), Rua Tenente Raimundo Rocha 515, 63040-360 Juazeiro do Norte, CE, BrazilFaculty of Medicine, Estácio of Juazeiro do Norte (FMJ), Rua Tenente Raimundo Rocha 515, 63040-360 Juazeiro do Norte, CE, BrazilFaculty of Medicine, Estácio of Juazeiro do Norte (FMJ), Rua Tenente Raimundo Rocha 515, 63040-360 Juazeiro do Norte, CE, BrazilFaculty of Medicine, Estácio of Juazeiro do Norte (FMJ), Rua Tenente Raimundo Rocha 515, 63040-360 Juazeiro do Norte, CE, BrazilFederal University of Ceará (UFC), Rua Coronel Nunes de Melo 1127, 60430-270 Fortaleza, CE, BrazilFederal University of Ceará (UFC), Rua Coronel Nunes de Melo 1127, 60430-270 Fortaleza, CE, BrazilFederal University of Ceará (UFC), Rua Coronel Nunes de Melo 1127, 60430-270 Fortaleza, CE, BrazilFederal University of Ceará (UFC), Rua Coronel Nunes de Melo 1127, 60430-270 Fortaleza, CE, BrazilFederal University of São Paulo (UNIFESP), Rua Pedro de Toledo 669, 04039-032 São Paulo, SP, BrazilFaculty of Medicine, Estácio of Juazeiro do Norte (FMJ), Rua Tenente Raimundo Rocha 515, 63040-360 Juazeiro do Norte, CE, BrazilBackground. Omega-3 (ω3) administration was shown to protect against hypoxic-ischemic injury. The objectives were to study the neuroprotective effects of ω3, in a model of global ischemia. Methods. Male Wistar rats were subjected to carotid occlusion (30 min), followed by reperfusion. The groups were SO, untreated ischemic and ischemic treated rats with ω3 (5 and 10 mg/kg, 7 days). The SO and untreated ischemic animals were orally treated with 1% cremophor and, 1 h after the last administration, they were behaviorally tested and euthanized for neurochemical (DA, DOPAC, and NE determinations), histological (Fluoro jade staining), and immunohistochemical (TNF-alpha, COX-2 and iNOS) evaluations. The data were analyzed by ANOVA and Newman-Keuls as the post hoc test. Results. Ischemia increased the locomotor activity and rearing behavior that were partly reversed by ω3. Ischemia decreased striatal DA and DOPAC contents and increased NE contents, effects reversed by ω3. This drug protected hippocampal neuron degeneration, as observed by Fluoro-Jade staining, and the increased immunostainings for TNF-alpha, COX-2, and iNOS were partly or totally blocked by ω3. Conclusion. This study showed a neuroprotective effect of ω3, in great part due to its anti-inflammatory properties, stimulating translational studies focusing on its use in clinic for stroke managing.http://dx.doi.org/10.1155/2016/6462120
spellingShingle Maria Elizabeth Pereira Nobre
Alyne Oliveira Correia
Francisco Nilson Maciel Mendonça
Luiz Ricardo Araújo Uchoa
Jessica Tamara Nunes Vasconcelos
Carlos Ney Alencar de Araújo
Gerly Anne de Castro Brito
Rafaelly Maria Pinheiro Siqueira
Gilberto dos Santos Cerqueira
Kelly Rose Tavares Neves
Ricardo Mário Arida
Glauce Socorro de Barros Viana
Omega-3 Fatty Acids: Possible Neuroprotective Mechanisms in the Model of Global Ischemia in Rats
Journal of Nutrition and Metabolism
title Omega-3 Fatty Acids: Possible Neuroprotective Mechanisms in the Model of Global Ischemia in Rats
title_full Omega-3 Fatty Acids: Possible Neuroprotective Mechanisms in the Model of Global Ischemia in Rats
title_fullStr Omega-3 Fatty Acids: Possible Neuroprotective Mechanisms in the Model of Global Ischemia in Rats
title_full_unstemmed Omega-3 Fatty Acids: Possible Neuroprotective Mechanisms in the Model of Global Ischemia in Rats
title_short Omega-3 Fatty Acids: Possible Neuroprotective Mechanisms in the Model of Global Ischemia in Rats
title_sort omega 3 fatty acids possible neuroprotective mechanisms in the model of global ischemia in rats
url http://dx.doi.org/10.1155/2016/6462120
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