Genomic virulence markers are associated with severe outcomes in patients with Pseudomonas aeruginosa bloodstream infection

Abstract Background Pseudomonas aeruginosa (PA) bloodstream infection (BSI) is a common healthcare-associated complication linked to antimicrobial resistance and high mortality. Ongoing clinical trials are exploring novel anti-virulence agents, yet studies on how bacterial virulence affects PA infec...

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Main Authors: John Karlsson Valik, Christian G. Giske, Badrul Hasan, Mónica Gozalo-Margüello, Luis Martínez-Martínez, Manica Mueller Premru, Žiga Martinčič, Bojana Beović, Sofia Maraki, Maria Zacharioudaki, Diamantis Kofteridis, Kate McCarthy, David Paterson, Marina de Cueto, Isabel Morales, Leonard Leibovici, Tanya Babich, Fredrik Granath, Jesús Rodríguez-Baño, Antonio Oliver, Dafna Yahav, Pontus Nauclér
Format: Article
Language:English
Published: Nature Portfolio 2024-12-01
Series:Communications Medicine
Online Access:https://doi.org/10.1038/s43856-024-00696-4
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author John Karlsson Valik
Christian G. Giske
Badrul Hasan
Mónica Gozalo-Margüello
Luis Martínez-Martínez
Manica Mueller Premru
Žiga Martinčič
Bojana Beović
Sofia Maraki
Maria Zacharioudaki
Diamantis Kofteridis
Kate McCarthy
David Paterson
Marina de Cueto
Isabel Morales
Leonard Leibovici
Tanya Babich
Fredrik Granath
Jesús Rodríguez-Baño
Antonio Oliver
Dafna Yahav
Pontus Nauclér
author_facet John Karlsson Valik
Christian G. Giske
Badrul Hasan
Mónica Gozalo-Margüello
Luis Martínez-Martínez
Manica Mueller Premru
Žiga Martinčič
Bojana Beović
Sofia Maraki
Maria Zacharioudaki
Diamantis Kofteridis
Kate McCarthy
David Paterson
Marina de Cueto
Isabel Morales
Leonard Leibovici
Tanya Babich
Fredrik Granath
Jesús Rodríguez-Baño
Antonio Oliver
Dafna Yahav
Pontus Nauclér
author_sort John Karlsson Valik
collection DOAJ
description Abstract Background Pseudomonas aeruginosa (PA) bloodstream infection (BSI) is a common healthcare-associated complication linked to antimicrobial resistance and high mortality. Ongoing clinical trials are exploring novel anti-virulence agents, yet studies on how bacterial virulence affects PA infection outcomes is conflicting and data from real-world clinical populations is limited. Methods We studied a multicentre cohort of 773 adult patients with PA BSI consecutively collected during 7-years from sites in Europe and Australia. Comprehensive clinical data and whole-genome sequencing of all bacterial strains were obtained. Results Based on the virulence genotype, we identify several virulence clusters, each showing varying proportions of multidrug-resistant phenotypes. Genes tied to biofilm synthesis and epidemic clones ST175 and ST235 are associated with mortality, while the type III secretion system is associated with septic shock. Adding genomic biomarkers to machine learning models based on clinical data indicates improved prediction of severe outcomes in PA BSI patients. Conclusions These findings suggest that virulence markers provide prognostic information with potential applications in guiding adjuvant sepsis treatments.
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spelling doaj-art-60f9a45c77c340d79f1ba94f12d0e8c52025-08-20T01:56:42ZengNature PortfolioCommunications Medicine2730-664X2024-12-014111410.1038/s43856-024-00696-4Genomic virulence markers are associated with severe outcomes in patients with Pseudomonas aeruginosa bloodstream infectionJohn Karlsson Valik0Christian G. Giske1Badrul Hasan2Mónica Gozalo-Margüello3Luis Martínez-Martínez4Manica Mueller Premru5Žiga Martinčič6Bojana Beović7Sofia Maraki8Maria Zacharioudaki9Diamantis Kofteridis10Kate McCarthy11David Paterson12Marina de Cueto13Isabel Morales14Leonard Leibovici15Tanya Babich16Fredrik Granath17Jesús Rodríguez-Baño18Antonio Oliver19Dafna Yahav20Pontus Nauclér21Department of Medicine, Solna, Division of Infectious Diseases, Karolinska InstitutetDepartment of Laboratory Medicine, Karolinska InstitutetDepartment of Laboratory Medicine, Karolinska InstitutetService of Microbiology. Hospital Universitario Marqués de Valdecilla. Instituto de Investigación Marqués de Valdecilla (IDIVAL)Unit of Microbiology, University Hospital Reina SofíaFaculty of Medicine, University of LjubljanaDepartment of Infectious Diseases, University Medical Centre LjubljanaFaculty of Medicine, University of LjubljanaDepartment of Internal Medicine, University Hospital of HeraklionDepartment of Internal Medicine, University Hospital of HeraklionDepartment of Internal Medicine, University Hospital of HeraklionPathology Queensland, Royal Brisbane and Woman’s HospitalUniversity of Queensland Centre for Clinical ResearchUnidad Clínica de Enfermedades Infecciosas y Microbiología, Instituto de Biomedicina de Sevilla (IBiS)/CSIC, Hospital Universitario Virgen Macarena / Departamentos de Medicina y Microbiología, Universidad de SevillaServicio de Urgencias, Hospital Universitario Virgen MacarenaResearch Authority, Rabin Medical Center, Beilinson hospitalResearch Authority, Rabin Medical Center, Beilinson hospitalDepartment of Medicine, Solna, Division of Clinical Epidemiology, Karolinska InstitutetUnidad Clínica de Enfermedades Infecciosas y Microbiología, Instituto de Biomedicina de Sevilla (IBiS)/CSIC, Hospital Universitario Virgen Macarena / Departamentos de Medicina y Microbiología, Universidad de SevillaServicio de Microbiología, Hospital Universitario Son Espases, Instituto de Investigación Sanitaria Illes Balears (IdISBa), CIBERINFECInfectious Diseases Unit, Sheba Medical CenterDepartment of Medicine, Solna, Division of Infectious Diseases, Karolinska InstitutetAbstract Background Pseudomonas aeruginosa (PA) bloodstream infection (BSI) is a common healthcare-associated complication linked to antimicrobial resistance and high mortality. Ongoing clinical trials are exploring novel anti-virulence agents, yet studies on how bacterial virulence affects PA infection outcomes is conflicting and data from real-world clinical populations is limited. Methods We studied a multicentre cohort of 773 adult patients with PA BSI consecutively collected during 7-years from sites in Europe and Australia. Comprehensive clinical data and whole-genome sequencing of all bacterial strains were obtained. Results Based on the virulence genotype, we identify several virulence clusters, each showing varying proportions of multidrug-resistant phenotypes. Genes tied to biofilm synthesis and epidemic clones ST175 and ST235 are associated with mortality, while the type III secretion system is associated with septic shock. Adding genomic biomarkers to machine learning models based on clinical data indicates improved prediction of severe outcomes in PA BSI patients. Conclusions These findings suggest that virulence markers provide prognostic information with potential applications in guiding adjuvant sepsis treatments.https://doi.org/10.1038/s43856-024-00696-4
spellingShingle John Karlsson Valik
Christian G. Giske
Badrul Hasan
Mónica Gozalo-Margüello
Luis Martínez-Martínez
Manica Mueller Premru
Žiga Martinčič
Bojana Beović
Sofia Maraki
Maria Zacharioudaki
Diamantis Kofteridis
Kate McCarthy
David Paterson
Marina de Cueto
Isabel Morales
Leonard Leibovici
Tanya Babich
Fredrik Granath
Jesús Rodríguez-Baño
Antonio Oliver
Dafna Yahav
Pontus Nauclér
Genomic virulence markers are associated with severe outcomes in patients with Pseudomonas aeruginosa bloodstream infection
Communications Medicine
title Genomic virulence markers are associated with severe outcomes in patients with Pseudomonas aeruginosa bloodstream infection
title_full Genomic virulence markers are associated with severe outcomes in patients with Pseudomonas aeruginosa bloodstream infection
title_fullStr Genomic virulence markers are associated with severe outcomes in patients with Pseudomonas aeruginosa bloodstream infection
title_full_unstemmed Genomic virulence markers are associated with severe outcomes in patients with Pseudomonas aeruginosa bloodstream infection
title_short Genomic virulence markers are associated with severe outcomes in patients with Pseudomonas aeruginosa bloodstream infection
title_sort genomic virulence markers are associated with severe outcomes in patients with pseudomonas aeruginosa bloodstream infection
url https://doi.org/10.1038/s43856-024-00696-4
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