The role and mechanism of NAT10‐mediated ac4C modification in tumor development and progression

Abstract RNA modification has emerged as a crucial area of research in epigenetics, significantly influencing tumor biology by regulating RNA metabolism. N‐acetyltransferase 10 (NAT10)‐mediated N4‐acetylcytidine (ac4C) modification, the sole known acetylation in eukaryotic RNA, influences cancer pat...

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Main Authors: Zhuoran Gu, Libin Zou, Xinjian Pan, Yang Yu, Yongqiang Liu, Zhijin Zhang, Ji Liu, Shiyu Mao, Junfeng Zhang, Changcheng Guo, Wei Li, Jiang Geng, Wentao Zhang, Xudong Yao, Bing Shen
Format: Article
Language:English
Published: Wiley 2024-12-01
Series:MedComm
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Online Access:https://doi.org/10.1002/mco2.70026
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author Zhuoran Gu
Libin Zou
Xinjian Pan
Yang Yu
Yongqiang Liu
Zhijin Zhang
Ji Liu
Shiyu Mao
Junfeng Zhang
Changcheng Guo
Wei Li
Jiang Geng
Wentao Zhang
Xudong Yao
Bing Shen
author_facet Zhuoran Gu
Libin Zou
Xinjian Pan
Yang Yu
Yongqiang Liu
Zhijin Zhang
Ji Liu
Shiyu Mao
Junfeng Zhang
Changcheng Guo
Wei Li
Jiang Geng
Wentao Zhang
Xudong Yao
Bing Shen
author_sort Zhuoran Gu
collection DOAJ
description Abstract RNA modification has emerged as a crucial area of research in epigenetics, significantly influencing tumor biology by regulating RNA metabolism. N‐acetyltransferase 10 (NAT10)‐mediated N4‐acetylcytidine (ac4C) modification, the sole known acetylation in eukaryotic RNA, influences cancer pathogenesis and progression. NAT10 is the only writer of ac4C and catalyzes acetyl transfer on targeted RNA, and ac4C helps to improve the stability and translational efficiency of ac4C‐modified RNA. NAT10 is highly expressed and associated with poor prognosis in pan‐cancers. Based on its molecular mechanism and biological functions, ac4C is a central factor in tumorigenesis, tumor progression, drug resistance, and tumor immune escape. Despite the increasing focus on ac4C, the specific regulatory mechanisms of ac4C in cancer remain elusive. The present review thoroughly analyzes the current knowledge on NAT10‐mediated ac4C modification in cancer, highlighting its broad regulatory influence on targeted gene expression and tumor biology. This review also summarizes the limitations and perspectives of current research on NAT10 and ac4C in cancer, to identify new therapeutic targets and advance cancer treatment strategies.
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spelling doaj-art-54c4a92227d242d4a14739bc3ff7c7732025-08-20T02:58:44ZengWileyMedComm2688-26632024-12-01512n/an/a10.1002/mco2.70026The role and mechanism of NAT10‐mediated ac4C modification in tumor development and progressionZhuoran Gu0Libin Zou1Xinjian Pan2Yang Yu3Yongqiang Liu4Zhijin Zhang5Ji Liu6Shiyu Mao7Junfeng Zhang8Changcheng Guo9Wei Li10Jiang Geng11Wentao Zhang12Xudong Yao13Bing Shen14Department of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaDepartment of Urology Shanghai Tenth People's Hospital School of Medicine Tongji University Shanghai ChinaAbstract RNA modification has emerged as a crucial area of research in epigenetics, significantly influencing tumor biology by regulating RNA metabolism. N‐acetyltransferase 10 (NAT10)‐mediated N4‐acetylcytidine (ac4C) modification, the sole known acetylation in eukaryotic RNA, influences cancer pathogenesis and progression. NAT10 is the only writer of ac4C and catalyzes acetyl transfer on targeted RNA, and ac4C helps to improve the stability and translational efficiency of ac4C‐modified RNA. NAT10 is highly expressed and associated with poor prognosis in pan‐cancers. Based on its molecular mechanism and biological functions, ac4C is a central factor in tumorigenesis, tumor progression, drug resistance, and tumor immune escape. Despite the increasing focus on ac4C, the specific regulatory mechanisms of ac4C in cancer remain elusive. The present review thoroughly analyzes the current knowledge on NAT10‐mediated ac4C modification in cancer, highlighting its broad regulatory influence on targeted gene expression and tumor biology. This review also summarizes the limitations and perspectives of current research on NAT10 and ac4C in cancer, to identify new therapeutic targets and advance cancer treatment strategies.https://doi.org/10.1002/mco2.70026cancerepitranscriptomeN4‐acetylation (ac4C)N‐acetyltransferase 10 (NAT10)RNA modification
spellingShingle Zhuoran Gu
Libin Zou
Xinjian Pan
Yang Yu
Yongqiang Liu
Zhijin Zhang
Ji Liu
Shiyu Mao
Junfeng Zhang
Changcheng Guo
Wei Li
Jiang Geng
Wentao Zhang
Xudong Yao
Bing Shen
The role and mechanism of NAT10‐mediated ac4C modification in tumor development and progression
MedComm
cancer
epitranscriptome
N4‐acetylation (ac4C)
N‐acetyltransferase 10 (NAT10)
RNA modification
title The role and mechanism of NAT10‐mediated ac4C modification in tumor development and progression
title_full The role and mechanism of NAT10‐mediated ac4C modification in tumor development and progression
title_fullStr The role and mechanism of NAT10‐mediated ac4C modification in tumor development and progression
title_full_unstemmed The role and mechanism of NAT10‐mediated ac4C modification in tumor development and progression
title_short The role and mechanism of NAT10‐mediated ac4C modification in tumor development and progression
title_sort role and mechanism of nat10 mediated ac4c modification in tumor development and progression
topic cancer
epitranscriptome
N4‐acetylation (ac4C)
N‐acetyltransferase 10 (NAT10)
RNA modification
url https://doi.org/10.1002/mco2.70026
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