Causal Relationships Between Immune Cells and Risk of Gastric Cancer: A Mendelian Randomization Study

ObjectiveTo analyze the causal relationship between immune cell phenotype and gastric cancer. MethodsBidirectional two-sample Mendelian randomization (MR) analysis was used to select 731 genetic variants involving immune cell phenotypes from the GWAS dataset as instrumental variables. Inverse-varian...

Full description

Saved in:
Bibliographic Details
Main Authors: Jiawei HE, Longnyu CAO, Mengyuan TANG, Hongquan CUI
Format: Article
Language:zho
Published: Magazine House of Cancer Research on Prevention and Treatment 2025-02-01
Series:Zhongliu Fangzhi Yanjiu
Subjects:
Online Access:http://www.zlfzyj.com/cn/article/doi/10.3971/j.issn.1000-8578.2025.24.0438
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849717151973244928
author Jiawei HE
Longnyu CAO
Mengyuan TANG
Hongquan CUI
author_facet Jiawei HE
Longnyu CAO
Mengyuan TANG
Hongquan CUI
author_sort Jiawei HE
collection DOAJ
description ObjectiveTo analyze the causal relationship between immune cell phenotype and gastric cancer. MethodsBidirectional two-sample Mendelian randomization (MR) analysis was used to select 731 genetic variants involving immune cell phenotypes from the GWAS dataset as instrumental variables. Inverse-variance weighting method (IVW), weighted median method (WM), and MR-Egger regression were used for sensitivity analysis. Cochran Q test, MR-Egger regression, MR-PRESSO method, and remain-one method were also conducted. ResultsChanges in the absolute count of IgD+ B cells and CD14-CD16- cells were significantly associated with the risk of gastric cancer. A lower proportion of IgD+ B cells was associated with a lower risk of gastric cancer (OR=0.86, 95%CI: 0.79-0.94), while an increased number of CD4-CD8-T cells was associated with an increased risk of gastric cancer (OR=1.2, 95%CI: 1.1-1.3). ConclusionA causal relationship exists between immune cell phenotype and the risk of gastric cancer. Changes in specific immune markers may regulate the development of gastric cancer by affecting the tumor microenvironment.
format Article
id doaj-art-4e8d35eddbc54ea2b08753b4dbbccab0
institution DOAJ
issn 1000-8578
language zho
publishDate 2025-02-01
publisher Magazine House of Cancer Research on Prevention and Treatment
record_format Article
series Zhongliu Fangzhi Yanjiu
spelling doaj-art-4e8d35eddbc54ea2b08753b4dbbccab02025-08-20T03:12:46ZzhoMagazine House of Cancer Research on Prevention and TreatmentZhongliu Fangzhi Yanjiu1000-85782025-02-0152217217610.3971/j.issn.1000-8578.2025.24.043820240438Causal Relationships Between Immune Cells and Risk of Gastric Cancer: A Mendelian Randomization StudyJiawei HE0Longnyu CAO1Mengyuan TANG2Hongquan CUI3College of Traditional Chinese Medicine Clinical Medicine, Hubei University of Chinese Medicine, Wuhan 431061, ChinaDepartment of Oncology, Suzhou Jiulong Hospital, School of Medicine, Shanghai Jiao Tong University, Suzhou 215018, ChinaCollege of Traditional Chinese Medicine Clinical Medicine, Hubei University of Chinese Medicine, Wuhan 431061, ChinaDepartment of Oncology, Suzhou Jiulong Hospital, School of Medicine, Shanghai Jiao Tong University, Suzhou 215018, ChinaObjectiveTo analyze the causal relationship between immune cell phenotype and gastric cancer. MethodsBidirectional two-sample Mendelian randomization (MR) analysis was used to select 731 genetic variants involving immune cell phenotypes from the GWAS dataset as instrumental variables. Inverse-variance weighting method (IVW), weighted median method (WM), and MR-Egger regression were used for sensitivity analysis. Cochran Q test, MR-Egger regression, MR-PRESSO method, and remain-one method were also conducted. ResultsChanges in the absolute count of IgD+ B cells and CD14-CD16- cells were significantly associated with the risk of gastric cancer. A lower proportion of IgD+ B cells was associated with a lower risk of gastric cancer (OR=0.86, 95%CI: 0.79-0.94), while an increased number of CD4-CD8-T cells was associated with an increased risk of gastric cancer (OR=1.2, 95%CI: 1.1-1.3). ConclusionA causal relationship exists between immune cell phenotype and the risk of gastric cancer. Changes in specific immune markers may regulate the development of gastric cancer by affecting the tumor microenvironment.http://www.zlfzyj.com/cn/article/doi/10.3971/j.issn.1000-8578.2025.24.0438immune cellsgastric cancermendelian randomizationcausal effects
spellingShingle Jiawei HE
Longnyu CAO
Mengyuan TANG
Hongquan CUI
Causal Relationships Between Immune Cells and Risk of Gastric Cancer: A Mendelian Randomization Study
Zhongliu Fangzhi Yanjiu
immune cells
gastric cancer
mendelian randomization
causal effects
title Causal Relationships Between Immune Cells and Risk of Gastric Cancer: A Mendelian Randomization Study
title_full Causal Relationships Between Immune Cells and Risk of Gastric Cancer: A Mendelian Randomization Study
title_fullStr Causal Relationships Between Immune Cells and Risk of Gastric Cancer: A Mendelian Randomization Study
title_full_unstemmed Causal Relationships Between Immune Cells and Risk of Gastric Cancer: A Mendelian Randomization Study
title_short Causal Relationships Between Immune Cells and Risk of Gastric Cancer: A Mendelian Randomization Study
title_sort causal relationships between immune cells and risk of gastric cancer a mendelian randomization study
topic immune cells
gastric cancer
mendelian randomization
causal effects
url http://www.zlfzyj.com/cn/article/doi/10.3971/j.issn.1000-8578.2025.24.0438
work_keys_str_mv AT jiaweihe causalrelationshipsbetweenimmunecellsandriskofgastriccanceramendelianrandomizationstudy
AT longnyucao causalrelationshipsbetweenimmunecellsandriskofgastriccanceramendelianrandomizationstudy
AT mengyuantang causalrelationshipsbetweenimmunecellsandriskofgastriccanceramendelianrandomizationstudy
AT hongquancui causalrelationshipsbetweenimmunecellsandriskofgastriccanceramendelianrandomizationstudy