Transcriptional landscape of pleural mesothelioma patients in relation to NF2 gene mutational status

Abstract Background Pleural mesothelioma (PM) is an aggressive cancer with poor prognosis, often driven by asbestos exposure. Mutations in the NF2 gene, a key regulator of the Hippo signaling pathway, are frequently observed in PM. However, their impact on tumor biology, immune infiltration, cytokin...

Full description

Saved in:
Bibliographic Details
Main Authors: Carlos Orozco-Castaño, Alejandro Mejía-Garcia, Hsuan Megan Tsao, Diego A. Bonilla, Carlos Carvajal-Fierro, Ricardo Bruges-Maya, Alba Combita, Rafael Parra-Medina
Format: Article
Language:English
Published: SpringerOpen 2025-06-01
Series:Journal of the Egyptian National Cancer Institute
Subjects:
Online Access:https://doi.org/10.1186/s43046-025-00284-0
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849238536493989888
author Carlos Orozco-Castaño
Alejandro Mejía-Garcia
Hsuan Megan Tsao
Diego A. Bonilla
Carlos Carvajal-Fierro
Ricardo Bruges-Maya
Alba Combita
Rafael Parra-Medina
author_facet Carlos Orozco-Castaño
Alejandro Mejía-Garcia
Hsuan Megan Tsao
Diego A. Bonilla
Carlos Carvajal-Fierro
Ricardo Bruges-Maya
Alba Combita
Rafael Parra-Medina
author_sort Carlos Orozco-Castaño
collection DOAJ
description Abstract Background Pleural mesothelioma (PM) is an aggressive cancer with poor prognosis, often driven by asbestos exposure. Mutations in the NF2 gene, a key regulator of the Hippo signaling pathway, are frequently observed in PM. However, their impact on tumor biology, immune infiltration, cytokine signaling, and therapeutic response remains poorly understood. Methods Using data from The Cancer Genome Atlas, we analyzed 82 PM cases to assess the prevalence and consequences of NF2 mutations. Logistic regression was used to evaluate associations with clinical variables, while transcriptomic differences were examined through differential expression and functional enrichment analyses. Immune and stromal infiltration were inferred via the xCell algorithm, cytokine signaling analyzed with Cytosig, and chemotherapeutic sensitivity predicted using the pRRophetic R package. Single-cell RNA sequencing data provided further insights into transcriptional patterns in NF2-mutated tumors. Results NF2 mutations were present in 22% of cases, with no significant correlations to histological subtype, stage, or age. NF2-mutated tumors exhibited increased infiltration of basophils, naïve B cells, and pericytes, along with altered cytokine profiles, including NRG1, TGFB3, and reduced FGF2. Differentially expressed genes, such as MYL7 and HOXA11, were linked to poorer survival. Chemotherapy modeling indicated higher sensitivity to camptothecin and vinblastine in NF2-mutated tumors. Conclusions NF2 mutations influence the tumor microenvironment, transcriptional landscape, and predicted therapeutic response in PM, underscoring their potential as prognostic biomarkers. These findings support tailored therapeutic strategies targeting NF2-related pathways, including Hippo signaling and cytokine modulation.
format Article
id doaj-art-4e6a1941e0a941908dc2df4ade6dc717
institution Kabale University
issn 2589-0409
language English
publishDate 2025-06-01
publisher SpringerOpen
record_format Article
series Journal of the Egyptian National Cancer Institute
spelling doaj-art-4e6a1941e0a941908dc2df4ade6dc7172025-08-20T04:01:35ZengSpringerOpenJournal of the Egyptian National Cancer Institute2589-04092025-06-0137111610.1186/s43046-025-00284-0Transcriptional landscape of pleural mesothelioma patients in relation to NF2 gene mutational statusCarlos Orozco-Castaño0Alejandro Mejía-Garcia1Hsuan Megan Tsao2Diego A. Bonilla3Carlos Carvajal-Fierro4Ricardo Bruges-Maya5Alba Combita6Rafael Parra-Medina7Instituto Nacional de CancerologíaMcGill UniversityMcGill UniversityUniversity of the Basque CountryInstituto Nacional de CancerologíaInstituto Nacional de CancerologíaInstituto Nacional de CancerologíaInstituto Nacional de CancerologíaAbstract Background Pleural mesothelioma (PM) is an aggressive cancer with poor prognosis, often driven by asbestos exposure. Mutations in the NF2 gene, a key regulator of the Hippo signaling pathway, are frequently observed in PM. However, their impact on tumor biology, immune infiltration, cytokine signaling, and therapeutic response remains poorly understood. Methods Using data from The Cancer Genome Atlas, we analyzed 82 PM cases to assess the prevalence and consequences of NF2 mutations. Logistic regression was used to evaluate associations with clinical variables, while transcriptomic differences were examined through differential expression and functional enrichment analyses. Immune and stromal infiltration were inferred via the xCell algorithm, cytokine signaling analyzed with Cytosig, and chemotherapeutic sensitivity predicted using the pRRophetic R package. Single-cell RNA sequencing data provided further insights into transcriptional patterns in NF2-mutated tumors. Results NF2 mutations were present in 22% of cases, with no significant correlations to histological subtype, stage, or age. NF2-mutated tumors exhibited increased infiltration of basophils, naïve B cells, and pericytes, along with altered cytokine profiles, including NRG1, TGFB3, and reduced FGF2. Differentially expressed genes, such as MYL7 and HOXA11, were linked to poorer survival. Chemotherapy modeling indicated higher sensitivity to camptothecin and vinblastine in NF2-mutated tumors. Conclusions NF2 mutations influence the tumor microenvironment, transcriptional landscape, and predicted therapeutic response in PM, underscoring their potential as prognostic biomarkers. These findings support tailored therapeutic strategies targeting NF2-related pathways, including Hippo signaling and cytokine modulation.https://doi.org/10.1186/s43046-025-00284-0Pleural mesotheliomaNF2 mutationsTranscriptome analysisChemotherapeutic responseImmune infiltrationPersonalized therapy
spellingShingle Carlos Orozco-Castaño
Alejandro Mejía-Garcia
Hsuan Megan Tsao
Diego A. Bonilla
Carlos Carvajal-Fierro
Ricardo Bruges-Maya
Alba Combita
Rafael Parra-Medina
Transcriptional landscape of pleural mesothelioma patients in relation to NF2 gene mutational status
Journal of the Egyptian National Cancer Institute
Pleural mesothelioma
NF2 mutations
Transcriptome analysis
Chemotherapeutic response
Immune infiltration
Personalized therapy
title Transcriptional landscape of pleural mesothelioma patients in relation to NF2 gene mutational status
title_full Transcriptional landscape of pleural mesothelioma patients in relation to NF2 gene mutational status
title_fullStr Transcriptional landscape of pleural mesothelioma patients in relation to NF2 gene mutational status
title_full_unstemmed Transcriptional landscape of pleural mesothelioma patients in relation to NF2 gene mutational status
title_short Transcriptional landscape of pleural mesothelioma patients in relation to NF2 gene mutational status
title_sort transcriptional landscape of pleural mesothelioma patients in relation to nf2 gene mutational status
topic Pleural mesothelioma
NF2 mutations
Transcriptome analysis
Chemotherapeutic response
Immune infiltration
Personalized therapy
url https://doi.org/10.1186/s43046-025-00284-0
work_keys_str_mv AT carlosorozcocastano transcriptionallandscapeofpleuralmesotheliomapatientsinrelationtonf2genemutationalstatus
AT alejandromejiagarcia transcriptionallandscapeofpleuralmesotheliomapatientsinrelationtonf2genemutationalstatus
AT hsuanmegantsao transcriptionallandscapeofpleuralmesotheliomapatientsinrelationtonf2genemutationalstatus
AT diegoabonilla transcriptionallandscapeofpleuralmesotheliomapatientsinrelationtonf2genemutationalstatus
AT carloscarvajalfierro transcriptionallandscapeofpleuralmesotheliomapatientsinrelationtonf2genemutationalstatus
AT ricardobrugesmaya transcriptionallandscapeofpleuralmesotheliomapatientsinrelationtonf2genemutationalstatus
AT albacombita transcriptionallandscapeofpleuralmesotheliomapatientsinrelationtonf2genemutationalstatus
AT rafaelparramedina transcriptionallandscapeofpleuralmesotheliomapatientsinrelationtonf2genemutationalstatus