HO-1 Upregulation Attenuates Adipocyte Dysfunction, Obesity, and Isoprostane Levels in Mice Fed High Fructose Diets
Background. Fructose metabolism is an unregulated metabolic pathway and excessive fructose consumption is known to activate ROS. HO-1 is a potent antioxidant gene that plays a key role in decreasing ROS and isoprostanes. We examined whether the fructose-mediated increase in adipocyte dysfunction inv...
Saved in:
| Main Authors: | Zeid Khitan, Mohit Harsh, Komal Sodhi, Joseph I. Shapiro, Nader G. Abraham |
|---|---|
| Format: | Article |
| Language: | English |
| Published: |
Wiley
2014-01-01
|
| Series: | Journal of Nutrition and Metabolism |
| Online Access: | http://dx.doi.org/10.1155/2014/980547 |
| Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Apo A1 Mimetic Rescues the Diabetic Phenotype of HO-2 Knockout Mice via an Increase in HO-1 Adiponectin and LKBI Signaling Pathway
by: Jian Cao, et al.
Published: (2012-01-01) -
8-isoprostane as the main marker of oxidative stress
by: N. M. Herasymchuk
Published: (2018-12-01) -
Effects of Azanza garckeana Fruit Pulp on Metabolic Syndrome in Wistar Rats Fed on High Fructose Diet
by: Godian C Iloabuchi, et al.
Published: (2024-05-01) -
Cardiac Hypertrophy in Pregnant Rats, Descendants of Fructose-Fed Mothers, an Effect That Worsens with Fructose Supplementation
by: Cristina Donis, et al.
Published: (2024-09-01) -
Regulatory effects of Xihu Longjing tea on the intestinal flora of mice fed with high-fat and high-fructose diet
by: HU Liquan, et al.
Published: (2024-06-01)