Rates of lobar atrophy in asymptomatic MAPT mutation carriers

Abstract Introduction The aim of this study was to investigate the rates of lobar atrophy in the asymptomatic microtubule‐associated protein tau (MAPT) mutation carriers. Methods MAPT mutation carriers (n = 14; 10 asymptomatic, 4 converters from asymptomatic to symptomatic) and noncarriers (n = 13)...

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Main Authors: Qin Chen, Bradley F. Boeve, Matthew Senjem, Nirubol Tosakulwong, Timothy G. Lesnick, Danielle Brushaber, Christina Dheel, Julie Fields, Leah Forsberg, Ralitza Gavrilova, Debra Gearhart, Jonathan Graff‐Radford, Neill R. Graff‐Radford, Clifford R. Jack Jr., David T. Jones, David S. Knopman, Walter K. Kremers, Maria Lapid, Rosa Rademakers, Jeremy Syrjanen, Adam L. Boxer, Howie Rosen, Zbigniew K. Wszolek, Kejal Kantarci, LEFFTDS Consortium
Format: Article
Language:English
Published: Wiley 2019-01-01
Series:Alzheimer’s & Dementia: Translational Research & Clinical Interventions
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Online Access:https://doi.org/10.1016/j.trci.2019.05.010
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author Qin Chen
Bradley F. Boeve
Matthew Senjem
Nirubol Tosakulwong
Timothy G. Lesnick
Danielle Brushaber
Christina Dheel
Julie Fields
Leah Forsberg
Ralitza Gavrilova
Debra Gearhart
Jonathan Graff‐Radford
Neill R. Graff‐Radford
Clifford R. Jack Jr.
David T. Jones
David S. Knopman
Walter K. Kremers
Maria Lapid
Rosa Rademakers
Jeremy Syrjanen
Adam L. Boxer
Howie Rosen
Zbigniew K. Wszolek
Kejal Kantarci
LEFFTDS Consortium
author_facet Qin Chen
Bradley F. Boeve
Matthew Senjem
Nirubol Tosakulwong
Timothy G. Lesnick
Danielle Brushaber
Christina Dheel
Julie Fields
Leah Forsberg
Ralitza Gavrilova
Debra Gearhart
Jonathan Graff‐Radford
Neill R. Graff‐Radford
Clifford R. Jack Jr.
David T. Jones
David S. Knopman
Walter K. Kremers
Maria Lapid
Rosa Rademakers
Jeremy Syrjanen
Adam L. Boxer
Howie Rosen
Zbigniew K. Wszolek
Kejal Kantarci
LEFFTDS Consortium
author_sort Qin Chen
collection DOAJ
description Abstract Introduction The aim of this study was to investigate the rates of lobar atrophy in the asymptomatic microtubule‐associated protein tau (MAPT) mutation carriers. Methods MAPT mutation carriers (n = 14; 10 asymptomatic, 4 converters from asymptomatic to symptomatic) and noncarriers (n = 13) underwent structural magnetic resonance imaging and were followed annually with a median of 9.2 years. Longitudinal changes in lobar atrophy were analyzed using the tensor‐based morphometry with symmetric normalization algorithm. Results The rate of temporal lobe atrophy in asymptomatic MAPT mutation carriers was faster than that in noncarriers. Although the greatest rate of atrophy was observed in the temporal lobe in converters, they also had increased atrophy rates in the frontal and parietal lobes compared to noncarriers. Discussion Accelerated decline in temporal lobe volume occurs in asymptomatic MAPT mutation carriers followed by the frontal and parietal lobe in those who have become symptomatic. The findings have implications for monitoring the progression of neurodegeneration during clinical trials in asymptomatic MAPT mutation carriers.
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spelling doaj-art-449ee00a80f4445289d9ebe809ee80b52025-08-20T03:30:39ZengWileyAlzheimer’s & Dementia: Translational Research & Clinical Interventions2352-87372019-01-015133834610.1016/j.trci.2019.05.010Rates of lobar atrophy in asymptomatic MAPT mutation carriersQin Chen0Bradley F. Boeve1Matthew Senjem2Nirubol Tosakulwong3Timothy G. Lesnick4Danielle Brushaber5Christina Dheel6Julie Fields7Leah Forsberg8Ralitza Gavrilova9Debra Gearhart10Jonathan Graff‐Radford11Neill R. Graff‐Radford12Clifford R. Jack Jr.13David T. Jones14David S. Knopman15Walter K. Kremers16Maria Lapid17Rosa Rademakers18Jeremy Syrjanen19Adam L. Boxer20Howie Rosen21Zbigniew K. Wszolek22Kejal Kantarci23LEFFTDS Consortium24Department of RadiologyMayo ClinicRochesterMNUSADepartment of NeurologyMayo ClinicRochesterMNUSADepartment of RadiologyMayo ClinicRochesterMNUSADepartment of Health Sciences ResearchMayo ClinicRochesterMNUSADepartment of Health Sciences ResearchMayo ClinicRochesterMNUSAAlzheimer's Disease Research CenterMayo ClinicRochesterMNUSADepartment of NeurologyMayo ClinicRochesterMNUSADepartment of Psychology and PsychiatryMayo ClinicRochesterMNUSADepartment of NeurologyMayo ClinicRochesterMNUSADepartment of Clinical Genomic and NeurologyMayo ClinicRochesterMNUSADepartment of NeurologyMayo ClinicRochesterMNUSADepartment of NeurologyMayo ClinicRochesterMNUSADepartment of NeurologyMayo ClinicJacksonvilleFLUSADepartment of RadiologyMayo ClinicRochesterMNUSADepartment of NeurologyMayo ClinicRochesterMNUSADepartment of NeurologyMayo ClinicRochesterMNUSADepartment of Health Sciences ResearchMayo ClinicRochesterMNUSADepartment of Psychology and PsychiatryMayo ClinicRochesterMNUSAAlzheimer's Disease Research CenterMayo ClinicRochesterMNUSADepartment of Health Sciences ResearchMayo ClinicRochesterMNUSAMemory and Aging Center, University of California San FranciscoSan FranciscoCAUSAMemory and Aging Center, University of California San FranciscoSan FranciscoCAUSADepartment of NeurologyMayo ClinicJacksonvilleFLUSADepartment of RadiologyMayo ClinicRochesterMNUSADepartment of RadiologyMayo ClinicRochesterMNUSAAbstract Introduction The aim of this study was to investigate the rates of lobar atrophy in the asymptomatic microtubule‐associated protein tau (MAPT) mutation carriers. Methods MAPT mutation carriers (n = 14; 10 asymptomatic, 4 converters from asymptomatic to symptomatic) and noncarriers (n = 13) underwent structural magnetic resonance imaging and were followed annually with a median of 9.2 years. Longitudinal changes in lobar atrophy were analyzed using the tensor‐based morphometry with symmetric normalization algorithm. Results The rate of temporal lobe atrophy in asymptomatic MAPT mutation carriers was faster than that in noncarriers. Although the greatest rate of atrophy was observed in the temporal lobe in converters, they also had increased atrophy rates in the frontal and parietal lobes compared to noncarriers. Discussion Accelerated decline in temporal lobe volume occurs in asymptomatic MAPT mutation carriers followed by the frontal and parietal lobe in those who have become symptomatic. The findings have implications for monitoring the progression of neurodegeneration during clinical trials in asymptomatic MAPT mutation carriers.https://doi.org/10.1016/j.trci.2019.05.010Frontotemporal dementiaMagnetic resonance imageMAPTAsymptomaticLongitudinal
spellingShingle Qin Chen
Bradley F. Boeve
Matthew Senjem
Nirubol Tosakulwong
Timothy G. Lesnick
Danielle Brushaber
Christina Dheel
Julie Fields
Leah Forsberg
Ralitza Gavrilova
Debra Gearhart
Jonathan Graff‐Radford
Neill R. Graff‐Radford
Clifford R. Jack Jr.
David T. Jones
David S. Knopman
Walter K. Kremers
Maria Lapid
Rosa Rademakers
Jeremy Syrjanen
Adam L. Boxer
Howie Rosen
Zbigniew K. Wszolek
Kejal Kantarci
LEFFTDS Consortium
Rates of lobar atrophy in asymptomatic MAPT mutation carriers
Alzheimer’s & Dementia: Translational Research & Clinical Interventions
Frontotemporal dementia
Magnetic resonance image
MAPT
Asymptomatic
Longitudinal
title Rates of lobar atrophy in asymptomatic MAPT mutation carriers
title_full Rates of lobar atrophy in asymptomatic MAPT mutation carriers
title_fullStr Rates of lobar atrophy in asymptomatic MAPT mutation carriers
title_full_unstemmed Rates of lobar atrophy in asymptomatic MAPT mutation carriers
title_short Rates of lobar atrophy in asymptomatic MAPT mutation carriers
title_sort rates of lobar atrophy in asymptomatic mapt mutation carriers
topic Frontotemporal dementia
Magnetic resonance image
MAPT
Asymptomatic
Longitudinal
url https://doi.org/10.1016/j.trci.2019.05.010
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