SOD1 Transcriptional and Posttranscriptional Regulation and Its Potential Implications in ALS
Copper-zinc superoxide dismutase (SOD1) is a detoxifying enzyme localized in the cytosol, nucleus, peroxisomes, and mitochondria. The discovery that mutations in SOD1 gene cause a subset of familial amyotrophic lateral sclerosis (FALS) has attracted great attention, and studies to date have been mai...
Saved in:
Main Authors: | Pamela Milani, Stella Gagliardi, Emanuela Cova, Cristina Cereda |
---|---|
Format: | Article |
Language: | English |
Published: |
Wiley
2011-01-01
|
Series: | Neurology Research International |
Online Access: | http://dx.doi.org/10.1155/2011/458427 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
An Examination of the Role of Transcriptional and Posttranscriptional Regulation in Rhabdomyosarcoma
by: Alexander J. Hron, et al.
Published: (2017-01-01) -
Transcriptional and Posttranscriptional Regulations of the HLA-G Gene
by: Erick C. Castelli, et al.
Published: (2014-01-01) -
Competing Endogenous RNA: The Key to Posttranscriptional Regulation
by: Rituparno Sen, et al.
Published: (2014-01-01) -
Regulation of Posttranscriptional Modification as a Possible Therapeutic Approach for Retinal Neuroprotection
by: Yoko Ozawa, et al.
Published: (2011-01-01) -
SNP Analysis of the SOD1 gene involved in Amyotrophic Lateral Sclerosis (ALS)
by: Hetal Patel, et al.
Published: (2025-12-01)