Participation of gut microbiota and bacterial translocation in chronic systemic inflammation in recently diagnosed rheumatoid arthritis patients

The objective of this study was to investigate the link between gut microbiota (GM) dysbiosis, gut inflammation, and bacterial translocation (BT) in recently diagnosed rheumatoid arthritis (RA). This case-control, observational study prospectively recruited recently diagnosed (<12 months) RA pati...

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Main Authors: Catherine Dunyach-Remy, Cassandra Pouget, Yves-Marie Pers, Cécile Gaujoux-Viala, Christophe Demattei, Florian Salipante, Lucia Grenga, Jean Armengaud, Jean-Philippe Lavigne, Christian Jorgensen
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Language:English
Published: Elsevier 2025-01-01
Series:Current Research in Microbial Sciences
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Online Access:http://www.sciencedirect.com/science/article/pii/S2666517425000288
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author Catherine Dunyach-Remy
Cassandra Pouget
Yves-Marie Pers
Cécile Gaujoux-Viala
Christophe Demattei
Florian Salipante
Lucia Grenga
Jean Armengaud
Jean-Philippe Lavigne
Christian Jorgensen
author_facet Catherine Dunyach-Remy
Cassandra Pouget
Yves-Marie Pers
Cécile Gaujoux-Viala
Christophe Demattei
Florian Salipante
Lucia Grenga
Jean Armengaud
Jean-Philippe Lavigne
Christian Jorgensen
author_sort Catherine Dunyach-Remy
collection DOAJ
description The objective of this study was to investigate the link between gut microbiota (GM) dysbiosis, gut inflammation, and bacterial translocation (BT) in recently diagnosed rheumatoid arthritis (RA). This case-control, observational study prospectively recruited recently diagnosed (<12 months) RA patients and age-matched healthy controls (HC) from two French hospitals between July 2014 to March 2018. The primary objective was to investigate GM composition in each group using 16S rRNA sequencing and metaproteomics approaches. Three plasmatic BT markers (sCD14, LPS-binding protein, and number of 16S rRNA gene copies) and one intestinal permeability marker (I-FABP) were quantified in blood samples.Twenty-five were included in each group, and 50 stools and blood samples were analyzed. 16S rRNA gene analysis showed an decrease in Coprococcus in RA patients after Body Mass Index and HLA status. Circulating bacterial DNA (number of copies of the 16S rRNA gene) and plasmatic I-FABP were higher in RA patients compared to HCs (p < 0.01), indicating increased BT and intestinal permeability in these patients. Metaproteomics from stool samples highlighted an increased host humoral immune response in RA, with elevated levels of inflammatory proteins (azurocidin, cathepsin G, neutrophil defensing 1). Gut inflammation may contribute to increased intestinal permeability, leading to BT into the systemic circulation and thus chronic inflammation.
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spelling doaj-art-1a7fbaa4d2964116a7ca3f663ed439f52025-08-20T03:46:49ZengElsevierCurrent Research in Microbial Sciences2666-51742025-01-01810036610.1016/j.crmicr.2025.100366Participation of gut microbiota and bacterial translocation in chronic systemic inflammation in recently diagnosed rheumatoid arthritis patientsCatherine Dunyach-Remy0Cassandra Pouget1Yves-Marie Pers2Cécile Gaujoux-Viala3Christophe Demattei4Florian Salipante5Lucia Grenga6Jean Armengaud7Jean-Philippe Lavigne8Christian Jorgensen9Bacterial Virulence and Chronic Infections, INSERM U1047, Univ Montpellier, Department of Microbiology and Hospital Hygiene, CHU Nîmes, Nîmes, France; Correspondence author: Univeristy Hospital of Nîmes, place Robert Debré, 30 908 Nîmes, FranceBacterial Virulence and Chronic Infections, INSERM U1047, Univ Montpellier, Department of Microbiology and Hospital Hygiene, CHU Nîmes, Nîmes, FranceDepartment of Rheumatology, Stem cells, Cellular plasticity, Regenerative medicine and Immunotherapies, IRMB, INSERM UMR1183, University of Montpellier &amp; University Hospital of Montpellier, Montpellier, FranceDesbrest Institute of Epidemiology and Public Health, University of Montpellier, INSERM, Department of Rheumatology, CHU Nîmes, Montpellier, FranceDepartment of Biostatistics, Epidemiology, Public Health and Innovation in Methodology (BESPIM), CHU Nîmes, Univ Montpellier, Nîmes, FranceDepartment of Biostatistics, Epidemiology, Public Health and Innovation in Methodology (BESPIM), CHU Nîmes, Univ Montpellier, Nîmes, FranceDepartment of Medicines and Technologies for Health, Atomic Energy and Alternative Energies Commission (CEA), Paris-Saclay University, Bagnols-sur-Cèze, FranceDepartment of Medicines and Technologies for Health, Atomic Energy and Alternative Energies Commission (CEA), Paris-Saclay University, Bagnols-sur-Cèze, FranceBacterial Virulence and Chronic Infections, INSERM U1047, Univ Montpellier, Department of Microbiology and Hospital Hygiene, CHU Nîmes, Nîmes, FranceDepartment of Rheumatology, Stem cells, Cellular plasticity, Regenerative medicine and Immunotherapies, IRMB, INSERM UMR1183, University of Montpellier &amp; University Hospital of Montpellier, Montpellier, FranceThe objective of this study was to investigate the link between gut microbiota (GM) dysbiosis, gut inflammation, and bacterial translocation (BT) in recently diagnosed rheumatoid arthritis (RA). This case-control, observational study prospectively recruited recently diagnosed (<12 months) RA patients and age-matched healthy controls (HC) from two French hospitals between July 2014 to March 2018. The primary objective was to investigate GM composition in each group using 16S rRNA sequencing and metaproteomics approaches. Three plasmatic BT markers (sCD14, LPS-binding protein, and number of 16S rRNA gene copies) and one intestinal permeability marker (I-FABP) were quantified in blood samples.Twenty-five were included in each group, and 50 stools and blood samples were analyzed. 16S rRNA gene analysis showed an decrease in Coprococcus in RA patients after Body Mass Index and HLA status. Circulating bacterial DNA (number of copies of the 16S rRNA gene) and plasmatic I-FABP were higher in RA patients compared to HCs (p < 0.01), indicating increased BT and intestinal permeability in these patients. Metaproteomics from stool samples highlighted an increased host humoral immune response in RA, with elevated levels of inflammatory proteins (azurocidin, cathepsin G, neutrophil defensing 1). Gut inflammation may contribute to increased intestinal permeability, leading to BT into the systemic circulation and thus chronic inflammation.http://www.sciencedirect.com/science/article/pii/S2666517425000288Rheumatoid arthritisGut microbiotaBacterial translocationIntestinal permeability
spellingShingle Catherine Dunyach-Remy
Cassandra Pouget
Yves-Marie Pers
Cécile Gaujoux-Viala
Christophe Demattei
Florian Salipante
Lucia Grenga
Jean Armengaud
Jean-Philippe Lavigne
Christian Jorgensen
Participation of gut microbiota and bacterial translocation in chronic systemic inflammation in recently diagnosed rheumatoid arthritis patients
Current Research in Microbial Sciences
Rheumatoid arthritis
Gut microbiota
Bacterial translocation
Intestinal permeability
title Participation of gut microbiota and bacterial translocation in chronic systemic inflammation in recently diagnosed rheumatoid arthritis patients
title_full Participation of gut microbiota and bacterial translocation in chronic systemic inflammation in recently diagnosed rheumatoid arthritis patients
title_fullStr Participation of gut microbiota and bacterial translocation in chronic systemic inflammation in recently diagnosed rheumatoid arthritis patients
title_full_unstemmed Participation of gut microbiota and bacterial translocation in chronic systemic inflammation in recently diagnosed rheumatoid arthritis patients
title_short Participation of gut microbiota and bacterial translocation in chronic systemic inflammation in recently diagnosed rheumatoid arthritis patients
title_sort participation of gut microbiota and bacterial translocation in chronic systemic inflammation in recently diagnosed rheumatoid arthritis patients
topic Rheumatoid arthritis
Gut microbiota
Bacterial translocation
Intestinal permeability
url http://www.sciencedirect.com/science/article/pii/S2666517425000288
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