Unilateral Optic Neuropathy and Acute Angle-Closure Glaucoma following Snake Envenomation
Purpose. We aimed to describe a unique case in which a patient developed unilateral optic neuritis and angle-closure glaucoma as a result of snake envenomation. Case Report. Approximately 18 hours after envenomation, a 67-year-old female patient described visual impairment and severe pain in her lef...
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| Format: | Article |
| Language: | English |
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Wiley
2015-01-01
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| Series: | Case Reports in Ophthalmological Medicine |
| Online Access: | http://dx.doi.org/10.1155/2015/687829 |
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| author | Osman Okan Olcaysu Kenan Cadirci Ahmet Altun Afak Durur Karakaya Huseyin Bayramlar |
| author_facet | Osman Okan Olcaysu Kenan Cadirci Ahmet Altun Afak Durur Karakaya Huseyin Bayramlar |
| author_sort | Osman Okan Olcaysu |
| collection | DOAJ |
| description | Purpose. We aimed to describe a unique case in which a patient developed unilateral optic neuritis and angle-closure glaucoma as a result of snake envenomation. Case Report. Approximately 18 hours after envenomation, a 67-year-old female patient described visual impairment and severe pain in her left eye (LE). The patient’s best corrected visual acuity was 10/10 in the RE and hand motion in the LE. Cranial magnetic resonance imaging showed signs of neuropathy in the left optic nerve. In the LE, corneal haziness, closure of the iridocorneal angle, and mild mydriasis were observed and pupillary light reflex was absent. Intraocular pressure was 25 mmHg and 57 mmHg in the RE and LE, respectively. The patient was diagnosed with acute angle-closure glaucoma in the LE. Optic neuropathy was treated with intravenous pulse methylprednisolone. Left intraocular pressure was within normal range starting on the fourth day. One month after the incident, there was no sign of optic neuropathy; relative afferent pupillary defect and optic nerve swelling disappeared. Conclusions. Patients with severe headache and visual loss after snake envenomation must be carefully examined for possible optic neuropathy and angle-closure glaucoma. Early diagnosis and treatment of these cases are necessary to prevent permanent damage to optic nerves. |
| format | Article |
| id | doaj-art-148eb39e9f1247cb9548145ce21f97f7 |
| institution | Kabale University |
| issn | 2090-6722 2090-6730 |
| language | English |
| publishDate | 2015-01-01 |
| publisher | Wiley |
| record_format | Article |
| series | Case Reports in Ophthalmological Medicine |
| spelling | doaj-art-148eb39e9f1247cb9548145ce21f97f72025-02-03T06:01:35ZengWileyCase Reports in Ophthalmological Medicine2090-67222090-67302015-01-01201510.1155/2015/687829687829Unilateral Optic Neuropathy and Acute Angle-Closure Glaucoma following Snake EnvenomationOsman Okan Olcaysu0Kenan Cadirci1Ahmet Altun2Afak Durur Karakaya3Huseyin Bayramlar4Clinic of Ophthalmology, Erzurum Education and Research Hospital, 25240 Erzurum, TurkeyClinic of Internal Medicine, Erzurum Education and Research Hospital, 25240 Erzurum, TurkeyClinic of Ophthalmology, Fatih Sultan Mehmet Education and Research Hospital, 34752 Istanbul, TurkeyClinic of Radiology, Erzurum Education and Research Hospital, 25240 Erzurum, TurkeyDepartment of Ophthalmology, Medical Faculty, Medeniyet University, 34730 Istanbul, TurkeyPurpose. We aimed to describe a unique case in which a patient developed unilateral optic neuritis and angle-closure glaucoma as a result of snake envenomation. Case Report. Approximately 18 hours after envenomation, a 67-year-old female patient described visual impairment and severe pain in her left eye (LE). The patient’s best corrected visual acuity was 10/10 in the RE and hand motion in the LE. Cranial magnetic resonance imaging showed signs of neuropathy in the left optic nerve. In the LE, corneal haziness, closure of the iridocorneal angle, and mild mydriasis were observed and pupillary light reflex was absent. Intraocular pressure was 25 mmHg and 57 mmHg in the RE and LE, respectively. The patient was diagnosed with acute angle-closure glaucoma in the LE. Optic neuropathy was treated with intravenous pulse methylprednisolone. Left intraocular pressure was within normal range starting on the fourth day. One month after the incident, there was no sign of optic neuropathy; relative afferent pupillary defect and optic nerve swelling disappeared. Conclusions. Patients with severe headache and visual loss after snake envenomation must be carefully examined for possible optic neuropathy and angle-closure glaucoma. Early diagnosis and treatment of these cases are necessary to prevent permanent damage to optic nerves.http://dx.doi.org/10.1155/2015/687829 |
| spellingShingle | Osman Okan Olcaysu Kenan Cadirci Ahmet Altun Afak Durur Karakaya Huseyin Bayramlar Unilateral Optic Neuropathy and Acute Angle-Closure Glaucoma following Snake Envenomation Case Reports in Ophthalmological Medicine |
| title | Unilateral Optic Neuropathy and Acute Angle-Closure Glaucoma following Snake Envenomation |
| title_full | Unilateral Optic Neuropathy and Acute Angle-Closure Glaucoma following Snake Envenomation |
| title_fullStr | Unilateral Optic Neuropathy and Acute Angle-Closure Glaucoma following Snake Envenomation |
| title_full_unstemmed | Unilateral Optic Neuropathy and Acute Angle-Closure Glaucoma following Snake Envenomation |
| title_short | Unilateral Optic Neuropathy and Acute Angle-Closure Glaucoma following Snake Envenomation |
| title_sort | unilateral optic neuropathy and acute angle closure glaucoma following snake envenomation |
| url | http://dx.doi.org/10.1155/2015/687829 |
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