The Prostaglandin E2-EP3 Receptor Axis Regulates Anaplasma phagocytophilum-Mediated NLRC4 Inflammasome Activation.

Rickettsial agents are sensed by pattern recognition receptors but lack pathogen-associated molecular patterns commonly observed in facultative intracellular bacteria. Due to these molecular features, the order Rickettsiales can be used to uncover broader principles of bacterial immunity. Here, we u...

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Main Authors: Xiaowei Wang, Dana K Shaw, Holly L Hammond, Fayyaz S Sutterwala, Manira Rayamajhi, Kari Ann Shirey, Darren J Perkins, Joseph V Bonventre, Thangam S Velayutham, Sean M Evans, Kyle G Rodino, Lauren VieBrock, Karen M Scanlon, Nicholas H Carbonetti, Jason A Carlyon, Edward A Miao, Jere W McBride, Michail Kotsyfakis, Joao H F Pedra
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2016-08-01
Series:PLoS Pathogens
Online Access:https://journals.plos.org/plospathogens/article/file?id=10.1371/journal.ppat.1005803&type=printable
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author Xiaowei Wang
Dana K Shaw
Holly L Hammond
Fayyaz S Sutterwala
Manira Rayamajhi
Kari Ann Shirey
Darren J Perkins
Joseph V Bonventre
Thangam S Velayutham
Sean M Evans
Kyle G Rodino
Lauren VieBrock
Karen M Scanlon
Nicholas H Carbonetti
Jason A Carlyon
Edward A Miao
Jere W McBride
Michail Kotsyfakis
Joao H F Pedra
author_facet Xiaowei Wang
Dana K Shaw
Holly L Hammond
Fayyaz S Sutterwala
Manira Rayamajhi
Kari Ann Shirey
Darren J Perkins
Joseph V Bonventre
Thangam S Velayutham
Sean M Evans
Kyle G Rodino
Lauren VieBrock
Karen M Scanlon
Nicholas H Carbonetti
Jason A Carlyon
Edward A Miao
Jere W McBride
Michail Kotsyfakis
Joao H F Pedra
author_sort Xiaowei Wang
collection DOAJ
description Rickettsial agents are sensed by pattern recognition receptors but lack pathogen-associated molecular patterns commonly observed in facultative intracellular bacteria. Due to these molecular features, the order Rickettsiales can be used to uncover broader principles of bacterial immunity. Here, we used the bacterium Anaplasma phagocytophilum, the agent of human granulocytic anaplasmosis, to reveal a novel microbial surveillance system. Mechanistically, we discovered that upon A. phagocytophilum infection, cytosolic phospholipase A2 cleaves arachidonic acid from phospholipids, which is converted to the eicosanoid prostaglandin E2 (PGE2) via cyclooxygenase 2 (COX2) and the membrane associated prostaglandin E synthase-1 (mPGES-1). PGE2-EP3 receptor signaling leads to activation of the NLRC4 inflammasome and secretion of interleukin (IL)-1β and IL-18. Importantly, the receptor-interacting serine/threonine-protein kinase 2 (RIPK2) was identified as a major regulator of the immune response against A. phagocytophilum. Accordingly, mice lacking COX2 were more susceptible to A. phagocytophilum, had a defect in IL-18 secretion and exhibited splenomegaly and damage to the splenic architecture. Remarkably, Salmonella-induced NLRC4 inflammasome activation was not affected by either chemical inhibition or genetic ablation of genes associated with PGE2 biosynthesis and signaling. This divergence in immune circuitry was due to reduced levels of the PGE2-EP3 receptor during Salmonella infection when compared to A. phagocytophilum. Collectively, we reveal the existence of a functionally distinct NLRC4 inflammasome illustrated by the rickettsial agent A. phagocytophilum.
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spelling doaj-art-130f8b62410e47cdafb35a596bcc86c12025-08-20T03:12:47ZengPublic Library of Science (PLoS)PLoS Pathogens1553-73661553-73742016-08-01128e100580310.1371/journal.ppat.1005803The Prostaglandin E2-EP3 Receptor Axis Regulates Anaplasma phagocytophilum-Mediated NLRC4 Inflammasome Activation.Xiaowei WangDana K ShawHolly L HammondFayyaz S SutterwalaManira RayamajhiKari Ann ShireyDarren J PerkinsJoseph V BonventreThangam S VelayuthamSean M EvansKyle G RodinoLauren VieBrockKaren M ScanlonNicholas H CarbonettiJason A CarlyonEdward A MiaoJere W McBrideMichail KotsyfakisJoao H F PedraRickettsial agents are sensed by pattern recognition receptors but lack pathogen-associated molecular patterns commonly observed in facultative intracellular bacteria. Due to these molecular features, the order Rickettsiales can be used to uncover broader principles of bacterial immunity. Here, we used the bacterium Anaplasma phagocytophilum, the agent of human granulocytic anaplasmosis, to reveal a novel microbial surveillance system. Mechanistically, we discovered that upon A. phagocytophilum infection, cytosolic phospholipase A2 cleaves arachidonic acid from phospholipids, which is converted to the eicosanoid prostaglandin E2 (PGE2) via cyclooxygenase 2 (COX2) and the membrane associated prostaglandin E synthase-1 (mPGES-1). PGE2-EP3 receptor signaling leads to activation of the NLRC4 inflammasome and secretion of interleukin (IL)-1β and IL-18. Importantly, the receptor-interacting serine/threonine-protein kinase 2 (RIPK2) was identified as a major regulator of the immune response against A. phagocytophilum. Accordingly, mice lacking COX2 were more susceptible to A. phagocytophilum, had a defect in IL-18 secretion and exhibited splenomegaly and damage to the splenic architecture. Remarkably, Salmonella-induced NLRC4 inflammasome activation was not affected by either chemical inhibition or genetic ablation of genes associated with PGE2 biosynthesis and signaling. This divergence in immune circuitry was due to reduced levels of the PGE2-EP3 receptor during Salmonella infection when compared to A. phagocytophilum. Collectively, we reveal the existence of a functionally distinct NLRC4 inflammasome illustrated by the rickettsial agent A. phagocytophilum.https://journals.plos.org/plospathogens/article/file?id=10.1371/journal.ppat.1005803&type=printable
spellingShingle Xiaowei Wang
Dana K Shaw
Holly L Hammond
Fayyaz S Sutterwala
Manira Rayamajhi
Kari Ann Shirey
Darren J Perkins
Joseph V Bonventre
Thangam S Velayutham
Sean M Evans
Kyle G Rodino
Lauren VieBrock
Karen M Scanlon
Nicholas H Carbonetti
Jason A Carlyon
Edward A Miao
Jere W McBride
Michail Kotsyfakis
Joao H F Pedra
The Prostaglandin E2-EP3 Receptor Axis Regulates Anaplasma phagocytophilum-Mediated NLRC4 Inflammasome Activation.
PLoS Pathogens
title The Prostaglandin E2-EP3 Receptor Axis Regulates Anaplasma phagocytophilum-Mediated NLRC4 Inflammasome Activation.
title_full The Prostaglandin E2-EP3 Receptor Axis Regulates Anaplasma phagocytophilum-Mediated NLRC4 Inflammasome Activation.
title_fullStr The Prostaglandin E2-EP3 Receptor Axis Regulates Anaplasma phagocytophilum-Mediated NLRC4 Inflammasome Activation.
title_full_unstemmed The Prostaglandin E2-EP3 Receptor Axis Regulates Anaplasma phagocytophilum-Mediated NLRC4 Inflammasome Activation.
title_short The Prostaglandin E2-EP3 Receptor Axis Regulates Anaplasma phagocytophilum-Mediated NLRC4 Inflammasome Activation.
title_sort prostaglandin e2 ep3 receptor axis regulates anaplasma phagocytophilum mediated nlrc4 inflammasome activation
url https://journals.plos.org/plospathogens/article/file?id=10.1371/journal.ppat.1005803&type=printable
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